Prenatal exposure to bisphenol a at environmentally relevant doses adversely affects the murine female reproductive tract later in life - Wikidata - wikimedia - TriplyDB

Prenatal exposure to bisphenol a at environmentally relevant doses adversely affects the murine female reproductive tract later in life

Prenatal exposure to bisphenol a at environmentally relevant doses adversely affects the murine female reproductive tract later in life

http://www.wikidata.org/entity/Q24657419

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LINE-1 hypomethylation in spermatozoa is associated with Bisphenol A exposure Disruption of adult expression of sexually selected traits by developmental exposure to bisphenol A.The Impact of External Factors on the Epigenome: In Utero and over Lifetime Bisphenol-A and Female Infertility: A Possible Role of Gene-Environment Interactions Polycystic ovary syndrome: do endocrine-disrupting chemicals play a role?Bisphenol A and the female reproductive tract: an overview of recent laboratory evidence and epidemiological studies Peroxisome Proliferator-Activated Receptor γ Is a Target for Halogenated Analogs of Bisphenol A Characterization of novel ligands of ERα, Erβ, and PPARγ: the case of halogenated bisphenol A and their conjugated metabolites Structural and mechanistic insights into bisphenols action provide guidelines for risk assessment and discovery of bisphenol A substitutes EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals Effects of High-Butterfat Diet on Embryo Implantation in Female Rats Exposed to Bisphenol A Epigenetic effects of endocrine-disrupting chemicals on female reproduction: an ovarian perspective Evidence for bisphenol A-induced female infertility: a review (2007-2016)Scientific Statement on the Diagnostic Criteria, Epidemiology, Pathophysiology, and Molecular Genetics of Polycystic Ovary Syndrome.Bisphenol a and reproductive health: update of experimental and human evidence, 2007-2013 The bisphenol A experience: a primer for the analysis of environmental effects on mammalian reproduction Prenatal environmental exposures, epigenetics, and disease Female Reproductive Disorders, Diseases, and Costs of Exposure to Endocrine Disrupting Chemicals in the European Union DNA methylation: a mechanism linking environmental chemical exposures to risk of autism spectrum disorders?Strain-Specific Induction of Endometrial Periglandular Fibrosis in Mice Exposed During Adulthood to the Endocrine Disrupting Chemical Bisphenol A Risk, Uncertainty and Precaution in Science: The Threshold of the Toxicological Concern Approach in Food Toxicology.Critical evaluation of key evidence on the human health hazards of exposure to bisphenol A.A transgenic mouse model expressing an ERα folding biosensor reveals the effects of Bisphenol A on estrogen receptor signaling.Perinatal exposure to bisphenol-a and the development of metabolic syndrome in CD-1 mice.Effects of in utero exposure to Bisphenol A or diethylstilbestrol on the adult male reproductive system.Perinatal bisphenol A exposure in C57B6/129svj male mice: potential altered cytokine/chemokine production in adulthood.Human excretion of bisphenol A: blood, urine, and sweat (BUS) study.Endocrine-disrupting chemicals and public health protection: a statement of principles from The Endocrine Society Nonproliferative and proliferative lesions of the rat and mouse female reproductive system.Structural and functional profiling of environmental ligands for estrogen receptors.Endocrine disrupting chemicals promote the growth of ovarian cancer cells via the ER-CXCL12-CXCR4 signaling axis.Bisphenol A, benzophenone-type ultraviolet filters, and phthalates in relation to uterine leiomyoma.Bisphenol A increases mammary cancer risk in two distinct mouse models of breast cancer Familiar and novel reproductive endocrine disruptors: xenoestrogens, dioxins and nanoparticles Normal and abnormal epithelial differentiation in the female reproductive tract.Preimplantation exposure to bisphenol A (BPA) affects embryo transport, preimplantation embryo development, and uterine receptivity in mice.In Vitro Screening of Environmental Chemicals Identifies Zearalenone as a Novel Substrate of the Placental BCRP/ABCG2 Transporter.Effects of early prepubertal exposure to bisphenol A on the onset of puberty, ovarian weights, and estrous cycle in female mice.Early programing of uterine tissue by bisphenol A: Critical evaluation of evidence from animal exposure studies.Synergistic activation of human pregnane X receptor by binary cocktails of pharmaceutical and environmental compounds.

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P2860

Prenatal exposure to bisphenol a at environmentally relevant doses adversely affects the murine female reproductive tract later in life

http://www.wikidata.org/entity/Q24657419

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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Prenatal exposure to bisphenol ...... productive tract later in life

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Prenatal exposure to bisphenol ...... productive tract later in life

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Prenatal exposure to bisphenol ...... productive tract later in life

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Environmental Health Perspectives

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Prenatal exposure to bisphenol ...... productive tract later in life

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Elizabeth Padilla-Banks

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Retha R Newbold

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Wendy N Jefferson

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P2860

The estrogenic effect of bisphenol A disrupts pancreatic beta-cell function in vivo and induces insulin resistance

No effect of route of exposure (oral; subcutaneous injection) on plasma bisphenol A throughout 24h after administration in neonatal female mice

Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4

Exposure of the U.S. population to bisphenol A and 4-tertiary-octylphenol: 2003-2004

Induction of mammary gland ductal hyperplasias and carcinoma in situ following fetal bisphenol A exposure

Developmental exposure to endocrine disruptors and the obesity epidemic

Parent bisphenol A accumulation in the human maternal-fetal-placental unit

Perinatal exposure to low doses of bisphenol A affects body weight, patterns of estrous cyclicity, and plasma LH levels

Low doses of bisphenol A and diethylstilbestrol impair Ca2+ signals in pancreatic alpha-cells through a nonclassical membrane estrogen receptor within intact islets of Langerhans.

Summary of the National Toxicology Program's report of the endocrine disruptors low-dose peer review

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scholarly article

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10.1289/EHP.0800045

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6

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117

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26659684

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19590677

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2702400

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