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Evaluating historical candidate genes for schizophrenia

Evaluating historical candidate genes for schizophrenia

http://www.wikidata.org/entity/Q28082318

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ehavioral Genetics DNA Damage and Repair in Schizophrenia and Autism: Implications for Cancer Comorbidity and Beyond Neuronal networks in mental diseases and neuropathic pain: Beyond brain derived neurotrophic factor and collapsin response mediator proteins Schizophrenia and reelin: a model based on prenatal stress to study epigenetics, brain development and behavior Can we observe epigenetic effects on human brain function?Unraveling the genetic architecture of copy number variants associated with schizophrenia and other neuropsychiatric disorders.From Shortage to Surge: A Developmental Switch in Hippocampal-Prefrontal Coupling in a Gene-Environment Model of Neuropsychiatric Disorders ENIGMA and the individual: Predicting factors that affect the brain in 35 countries worldwide.Genetic variation in the 3'-untranslated region of PAK1 influences schizophrenia susceptibility Uniting the neuro developmental and immunological hypotheses: Neuregulin 1 receptor ErbB and Toll-like receptor activation in first-episode schizophrenia.Understanding the pathophysiology of postpartum psychosis: Challenges and new approaches.The NMDA Receptor and Schizophrenia: From Pathophysiology to Treatment.Schizophrenia interactome with 504 novel protein-protein interactions A Role for the Transcription Factor Nk2 Homeobox 1 in Schizophrenia: Convergent Evidence from Animal and Human Studies COMT Val(158) Met genotype is associated with reward learning: a replication study and meta-analysis Bridging the Gap between Genes and Language Deficits in Schizophrenia: An Oscillopathic Approach.Balanced translocation linked to psychiatric disorder, glutamate, and cortical structure/function.Modeling psychiatric disorders: from genomic findings to cellular phenotypes Genotype-Dependent Effects of COMT Inhibition on Cognitive Function in a Highly Specific, Novel Mouse Model of Altered COMT Activity.The Proteome of BLOC-1 Genetic Defects Identifies the Arp2/3 Actin Polymerization Complex to Function Downstream of the Schizophrenia Susceptibility Factor Dysbindin at the Synapse The Major Histocompatibility Complex (MHC) in Schizophrenia: A Review.Dysbindin Deficiency Modifies the Expression of GABA Neuron and Ion Permeation Transcripts in the Developing Hippocampus.Commentary for Special Issue of Prevention Science "Using Genetics in Prevention: Science Fiction or Science Fact?"Translating advances in the molecular basis of schizophrenia into novel cognitive treatment strategies.Chlorpromazine Increases the Expression of Polysialic Acid (PolySia) in Human Neuroblastoma Cells and Mouse Prefrontal Cortex.Association between COMT Val158Met and psychiatric disorders: A comprehensive meta-analysis.Critical Issues in the Inclusion of Genetic and Epigenetic Information in Prevention and Intervention Trials.Connecting Brain Proteomics with Behavioural Neuroscience in Translational Animal Models of Neuropsychiatric Disorders.Endophenotype best practices.Improving Current Treatments for Schizophrenia.The Role of Genes, Stress, and Dopamine in the Development of Schizophrenia.Gene-environment interactions in cortical interneuron development and dysfunction: A review of preclinical studies.More than 25 years of genetic studies of clozapine-induced agranulocytosis.The highly pleiotropic gene SLC39A8 as an opportunity to gain insight into the molecular pathogenesis of schizophrenia.Role of DISC1 in Neuronal Trafficking and its Implication in Neuropsychiatric Manifestation and Neurotherapeutics.Comparative genomic evidence for the involvement of schizophrenia risk genes in antipsychotic effects.BDNF rescues prefrontal dysfunction elicited by pyramidal neuron-specific DTNBP1 deletion in vivo.The dopamine beta-hydroxylase gene polymorphism rs1611114 is associated with schizophrenia in the Chinese Zhuang but not Chinese Han population.The potential of DISC1 protein as a therapeutic target for mental illness.Evaluating the dopamine hypothesis of schizophrenia in a large-scale genome-wide association study.

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P2860

Evaluating historical candidate genes for schizophrenia

http://www.wikidata.org/entity/Q28082318

description

2015 nî lūn-bûn

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2015 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2015 թվականի մայիսին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Evaluating historical candidate genes for schizophrenia

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Evaluating historical candidate genes for schizophrenia

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Evaluating historical candidate genes for schizophrenia

@nl

type

label

Evaluating historical candidate genes for schizophrenia

@ast

Evaluating historical candidate genes for schizophrenia

@en

Evaluating historical candidate genes for schizophrenia

@nl

prefLabel

Evaluating historical candidate genes for schizophrenia

@ast

Evaluating historical candidate genes for schizophrenia

@en

Evaluating historical candidate genes for schizophrenia

@nl

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Molecular Psychiatry

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Evaluating historical candidate genes for schizophrenia

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M C O'Donovan

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M J Owen

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M S Farrell

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P Sklar

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R A Ophoff

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S Cichon

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Initial sequencing and analysis of the human genome

Guidelines for genome-wide association studies

Potential etiologic and functional implications of genome-wide association loci for human diseases and traits

Meta-analysis of genetic association studies supports a contribution of common variants to susceptibility to common disease

Replication in Genome-Wide Association Studies

Genetic and physiological data implicating the new human gene G72 and the gene for D-amino acid oxidase in schizophrenia

Genetic variation at the 22q11 PRODH2/DGCR6 locus presents an unusual pattern and increases susceptibility to schizophrenia

Genetic studies of alcoholism and substance dependence.

Genetic variation in the 22q11 locus and susceptibility to schizophrenia

Genome-wide association studies for complex traits: consensus, uncertainty and challenges

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