Increased response to morphine in mice lacking protein kinase C epsilon
about
A blocker of N- and T-type voltage-gated calcium channels attenuates ethanol-induced intoxication, place preference, self-administration, and reinstatementDo initial responses to drugs predict future use or abuse?Striatal signal transduction and drug addictionAgonist-dependent mu-opioid receptor signaling can lead to heterologous desensitization.Shared mechanisms for opioid tolerance and a transition to chronic pain.Activation of protein kinase C (PKC)α or PKCε as an approach to increase morphine tolerance in respiratory depression and lethal overdoseMice lacking rhes show altered morphine analgesia, tolerance, and dependence.Protein kinases and addiction.Protein kinase C isozymes as regulators of sensitivity to and self-administration of drugs of abuse-studies with genetically modified mice.Effect of KEPI (Ppp1r14c) deletion on morphine analgesia and tolerance in mice of different genetic backgrounds: when a knockout is near a relevant quantitative trait locus.Involvement of protein kinase C in morphine tolerance at spinal levels of rats.Rhes: a GTP-binding protein integral to striatal physiology and pathology.Synaptic Plasticity and Signal Transduction Gene Polymorphisms and Vulnerability to Drug Addictions in Populations of European or African Ancestry.Regulation of μ-opioid receptors: desensitization, phosphorylation, internalization, and tolerance.Morphine-induced mu-opioid receptor rapid desensitization is independent of receptor phosphorylation and beta-arrestinsInvolvement of PKC alpha and G-protein-coupled receptor kinase 2 in agonist-selective desensitization of mu-opioid receptors in mature brain neurons.Heterologous regulation of agonist-independent μ-opioid receptor phosphorylation by protein kinase CMolecular mechanism for opioid dichotomy: bidirectional effect of μ-opioid receptors on P2X₃ receptor currents in rat sensory neurones.Role of protein kinase C and mu-opioid receptor (MOPr) desensitization in tolerance to morphine in rat locus coeruleus neurons.PKCepsilon regulates behavioral sensitivity, binding and tolerance to the CB1 receptor agonist WIN55,212-2.The activation of μ-opioid receptor potentiates LPS-induced NF-kB promoting an inflammatory phenotype in microglia.Cocaine craving during protracted withdrawal requires PKCε priming within vmPFC.Molecular Regulation of Sprouting Angiogenesis.
P2860
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P2860
Increased response to morphine in mice lacking protein kinase C epsilon
description
2007 nî lūn-bûn
@nan
2007 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Increased response to morphine in mice lacking protein kinase C epsilon
@ast
Increased response to morphine in mice lacking protein kinase C epsilon
@en
Increased response to morphine in mice lacking protein kinase C epsilon
@nl
type
label
Increased response to morphine in mice lacking protein kinase C epsilon
@ast
Increased response to morphine in mice lacking protein kinase C epsilon
@en
Increased response to morphine in mice lacking protein kinase C epsilon
@nl
prefLabel
Increased response to morphine in mice lacking protein kinase C epsilon
@ast
Increased response to morphine in mice lacking protein kinase C epsilon
@en
Increased response to morphine in mice lacking protein kinase C epsilon
@nl
P2093
P2860
P50
P1476
Increased response to morphine in mice lacking protein kinase C epsilon
@en
P2093
P2860
P304
P356
10.1111/J.1601-183X.2006.00261.X
P407
P577
2006-08-07T00:00:00Z