Protein stickiness, rather than number of functional protein-protein interactions, predicts expression noise and plasticity in yeastComplex adaptations can drive the evolution of the capacitor [PSI], even with realistic rates of yeast sexThe spontaneous appearance rate of the yeast prion [PSI+] and its implications for the evolution of the evolvability properties of the [PSI+] systemThe roles of mutation accumulation and selection in loss of sporulation in experimental populations of Bacillus subtilisTHE EVOLUTION OF THE EVOLVABILITY PROPERTIES OF THE YEAST PRION [PSI + ]The loss of adaptive plasticity during long periods of environmental stasisEvolution of molecular error rates and the consequences for evolvability.Cryptic genetic variation is enriched for potential adaptationsCompensatory evolution and the origins of innovationsQuantitative prediction of molecular clock and ka/ks at short timescalesQ&A;: Evolutionary capacitanceRobustness and evolvabilityThe strength of selection against the yeast prion [PSI+]The evolution of reversible switches in the presence of irreversible mimicsRobustness: mechanisms and consequencesThe evolution of bet-hedging adaptations to rare scenariosThe conversion of 3' UTRs into coding regionsMutations leading to loss of sporulation ability in Bacillus subtilis are sufficiently frequent to favor genetic canalizationThe evolution of the evolvability properties of the yeast prion [PSI+]Genetic driftCryptic genetic variation can make “irreducible complexity” a common mode of adaptation in sexual populationsAnswering evolutionary questions: A guide for mechanistic biologists.Quantifying the kinetic parameters of prion replication.Designing drugs to stop the formation of prion aggregates and other amyloids.Rethinking Hardy-Weinberg and genetic drift in undergraduate biology.Putatively noncoding transcripts show extensive association with ribosomesWhat Fraction of Duplicates Observed in Recently Sequenced Genomes Is Segregating and Destined to Fail to Fix?Constraints on the evolution of phenotypic plasticity: limits and costs of phenotype and plasticity.Chance, purpose, and progress in evolution and christianity.Prion kinetics.Intercellular competition and the inevitability of multicellular aging.Drift Barriers to Quality Control When Genes Are Expressed at Different Levels.The population genetics of phenotypic deterioration in experimental populations of Bacillus subtilis.Efficient inhibition of prion replication by PrP-Fc(2) suggests that the prion is a PrP(Sc) oligomer.Young Genes are Highly Disordered as Predicted by the Preadaptation Hypothesis of De Novo Gene Birth.A shift in aggregation avoidance strategy marks a long-term direction to protein evolutionReply to Cheong et al.: Unicellular survival precludes Parrondo's paradoxThe measured level of prion infectivity varies in a predictable way according to the aggregation state of the infectious agentGene Birth Contributes to Structural Disorder Encoded by Overlapping GenesFeed-forward regulation adaptively evolves via dynamics rather than topology when there is intrinsic noise.
P50
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P50
description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Joanna Masel
@ast
Joanna Masel
@en
Joanna Masel
@es
Joanna Masel
@nl
Joanna Masel
@sl
type
label
Joanna Masel
@ast
Joanna Masel
@en
Joanna Masel
@es
Joanna Masel
@nl
Joanna Masel
@sl
prefLabel
Joanna Masel
@ast
Joanna Masel
@en
Joanna Masel
@es
Joanna Masel
@nl
Joanna Masel
@sl
P21
P31
P3829
P496
0000-0002-7398-2127
P569
2000-01-01T00:00:00Z