Mycobacterium tuberculosis uses host triacylglycerol to accumulate lipid droplets and acquires a dormancy-like phenotype in lipid-loaded macrophages
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Nocardia brasiliensis induces formation of foamy macrophages and dendritic cells in vitro and in vivoLipid body-phagosome interaction in macrophages during infectious diseases: host defense or pathogen survival strategy?Mycobacterium tuberculosis: Manipulator of Protective ImmunityThe lipid droplet-a well-connected organelleHeterogeneity in tuberculosis pathology, microenvironments and therapeutic responsesTranslating basic science insight into public health action for multidrug- and extensively drug-resistant tuberculosisMycobacterium tuberculosis: success through dormancyFeeding uninvited guests: mTOR and AMPK set the table for intracellular pathogensLipids at the interface of virus-host interactionsHypoxia-mediated regulation of macrophage functions in pathophysiologyLipid Droplet Formation, Their Localization and Dynamics during Leishmania major Macrophage InfectionLipid Body Organelles within the Parasite Trypanosoma cruzi: A Role for Intracellular Arachidonic Acid MetabolismExperimental Models of Foamy Macrophages and Approaches for Dissecting the Mechanisms of Lipid Accumulation and Consumption during Dormancy and Reactivation of TuberculosisReprogramming neutral lipid metabolism in mouse dendritic leucocytes hosting live Leishmania amazonensis amastigotesAssessment of treatment response in tuberculosisMmPPOX inhibits Mycobacterium tuberculosis lipolytic enzymes belonging to the hormone-sensitive lipase family and alters mycobacterial growthAn acyl-CoA synthetase in Mycobacterium tuberculosis involved in triacylglycerol accumulation during dormancyNovel inhibitors of cholesterol degradation in Mycobacterium tuberculosis reveal how the bacterium's metabolism is constrained by the intracellular environmentReplication rates of Mycobacterium tuberculosis in human macrophages do not correlate with mycobacterial antibiotic susceptibilityMycobacterial Metabolic Syndrome: LprG and Rv1410 Regulate Triacylglyceride Levels, Growth Rate and Virulence in Mycobacterium tuberculosisThe Mycobacterium tuberculosis regulatory network and hypoxiaThree-dimensional in vitro models of granuloma to study bacteria-host interactions, drug-susceptibility, and resuscitation of dormant mycobacteriaMetabolic Perspectives on PersistenceComparative analyses of transport proteins encoded within the genomes of Mycobacterium tuberculosis and Mycobacterium lepraeDelayed bactericidal response of Mycobacterium tuberculosis to bedaquiline involves remodelling of bacterial metabolism.Lipid accumulation during the establishment of kleptoplasty in Elysia chlorotica.Bovis Bacillus Calmette-Guerin (BCG) infection induces exosomal miRNA release by human macrophagesRv3723/LucA coordinates fatty acid and cholesterol uptake in Mycobacterium tuberculosis.Pathogenicity of Mycobacterium tuberculosis is expressed by regulating metabolic thresholds of the host macrophage.Oleoyl coenzyme A regulates interaction of transcriptional regulator RaaS (Rv1219c) with DNA in mycobacteria.Simvastatin increases the in vivo activity of the first-line tuberculosis regimen.Manipulation of the mononuclear phagocyte system by Mycobacterium tuberculosis.Wax ester synthesis is required for Mycobacterium tuberculosis to enter in vitro dormancy.Human granuloma in vitro model, for TB dormancy and resuscitation.Characterizing virulence-specific perturbations in the mitochondrial function of macrophages infected with Mycobacterium tuberculosis.Identification of residues involved in substrate specificity and cytotoxicity of two closely related cutinases from Mycobacterium tuberculosisThe Sculpting of the Mycobacterium tuberculosis Genome by Host Cell-Derived PressuresMacrophage immunoregulatory pathways in tuberculosis.Cyclic AMP-dependent resuscitation of dormant Mycobacteria by exogenous free fatty acids.Non-replicating Mycobacterium tuberculosis elicits a reduced infectivity profile with corresponding modifications to the cell wall and extracellular matrix.
P2860
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P2860
Mycobacterium tuberculosis uses host triacylglycerol to accumulate lipid droplets and acquires a dormancy-like phenotype in lipid-loaded macrophages
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages
@ast
Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages
@en
Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages
@nl
type
label
Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages
@ast
Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages
@en
Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages
@nl
prefLabel
Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages
@ast
Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages
@en
Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages
@nl
P2093
P2860
P3181
P1433
P1476
Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages
@en
P2093
Chirajyoti Deb
Hédia Maamar
Jaiyanth Daniel
Pappachan E Kolattukudy
Tatiana D Sirakova
P2860
P304
P3181
P356
10.1371/JOURNAL.PPAT.1002093
P407
P577
2011-06-01T00:00:00Z