A role for G-CSF receptor signaling in the regulation of hematopoietic cell function but not lineage commitment or differentiation
about
Protective and pathogenic functions of macrophage subsetsNeutrophil mobilization via plerixafor-mediated CXCR4 inhibition arises from lung demargination and blockade of neutrophil homing to the bone marrowPhosphoprotein profiling of erythropoietin receptor- dependent pathways using different proteomic strategies.Hematopoiesis and its disorders: a systems biology approachGranulocyte colony-stimulating factor: molecular mechanisms of action during steady state and 'emergency' hematopoiesis.Innate immune regulation by STAT-mediated transcriptional mechanisms.Lymphoid and myeloid lineage commitment in multipotent hematopoietic progenitorsThe signaling domain of the erythropoietin receptor rescues prolactin receptor-mutant mammary epithelium.Early systemic granulocyte-colony stimulating factor treatment attenuates neuropathic pain after peripheral nerve injury.G-CSF preferentially supports the generation of gut-homing Gr-1high macrophages in M-CSF-treated bone marrow cells.Granulocyte colony-stimulating factor reprograms bone marrow stromal cells to actively suppress B lymphopoiesis in mice.STAT3 governs distinct pathways in emergency granulopoiesis and mature neutrophils.G-CSF down-regulation of CXCR4 expression identified as a mechanism for mobilization of myeloid cells.Activation of mitogen-activated protein kinase kinase (MEK)/extracellular signal regulated kinase (ERK) signaling pathway is involved in myeloid lineage commitment.Enforced granulocyte/macrophage colony-stimulating factor signals do not support lymphopoiesis, but instruct lymphoid to myelomonocytic lineage conversion.G-CSF is a key modulator of MDSC and could be a potential therapeutic target in colitis-associated colorectal cancers.LRG-accelerated differentiation defines unique G-CSFR signaling pathways downstream of PU.1 and C/EBPepsilon that modulate neutrophil activationCytokine signals through STAT3 promote expression of granulocyte secondary granule proteins in 32D cellsControl of myeloid-specific integrin alpha Mbeta 2 (CD11b/CD18) expression by cytokines is regulated by Stat3-dependent activation of PU.1.Cytokine signaling through Stat3 activates integrins, promotes adhesion, and induces growth arrest in the myeloid cell line 32D.G-CSF activation of AKT is not sufficient to prolong neutrophil survivalThe coordinated action of G-CSF and ELR + CXC chemokines in neutrophil mobilization during acute inflammation.Cytokine receptors and hematopoietic differentiation.Cytokine signaling modules in inflammatory responses.Pertussis toxin-sensitive G proteins regulate lymphoid lineage specification in multipotent hematopoietic progenitors.A late-lineage murine neutrophil precursor population exhibits dynamic changes during demand-adapted granulopoiesisHyperglycemia promotes myelopoiesis and impairs the resolution of atherosclerosis.Immunosurveillance of senescent cells: the bright side of the senescence program.STAT3 signaling in immunity.G-CSF potently inhibits osteoblast activity and CXCL12 mRNA expression in the bone marrow.Distinctive and indispensable roles of PU.1 in maintenance of hematopoietic stem cells and their differentiation.Neutrophil elastase downmodulates native G-CSFR expression and granulocyte-macrophage colony formation.FIP1L1-PDGFRalpha imposes eosinophil lineage commitment on hematopoietic stem/progenitor cells.Retroviral transduction of IL-7Ralpha into IL-7Ralpha-/- bone marrow progenitors: correction of lymphoid deficiency and induction of neutrophilia.Epidermal Growth Factor and Granulocyte Colony Stimulating Factor Signaling Are Synergistic for Hematopoietic Regeneration.C/EBPα Is Required for Proteolytic Cleavage of Cyclin A by Calpain 3 in Myeloid Precursor Cells
P2860
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P2860
A role for G-CSF receptor signaling in the regulation of hematopoietic cell function but not lineage commitment or differentiation
description
1999 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 1999
@ast
im August 1999 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 1999/08/01)
@sk
vědecký článek publikovaný v roce 1999
@cs
wetenschappelijk artikel (gepubliceerd op 1999/08/01)
@nl
наукова стаття, опублікована в серпні 1999
@uk
مقالة علمية (نشرت في أغسطس 1999)
@ar
name
A role for G-CSF receptor sign ...... commitment or differentiation
@ast
A role for G-CSF receptor sign ...... commitment or differentiation
@en
A role for G-CSF receptor sign ...... commitment or differentiation
@nl
type
label
A role for G-CSF receptor sign ...... commitment or differentiation
@ast
A role for G-CSF receptor sign ...... commitment or differentiation
@en
A role for G-CSF receptor sign ...... commitment or differentiation
@nl
prefLabel
A role for G-CSF receptor sign ...... commitment or differentiation
@ast
A role for G-CSF receptor sign ...... commitment or differentiation
@en
A role for G-CSF receptor sign ...... commitment or differentiation
@nl
P2093
P1433
P1476
A role for G-CSF receptor sign ...... commitment or differentiation
@en
P2093
C. L. Semerad
D. C. Link
J. Poursine-Laurent
P304
P356
10.1016/S1074-7613(00)80090-4
P407
P577
1999-08-01T00:00:00Z