Severe osmotic compression triggers a slowdown of intracellular signaling, which can be explained by molecular crowding - Wikidata - wikimedia - TriplyDB

Severe osmotic compression triggers a slowdown of intracellular signaling, which can be explained by molecular crowding

Severe osmotic compression triggers a slowdown of intracellular signaling, which can be explained by molecular crowding

http://www.wikidata.org/entity/Q30538482

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Mechanical slowing-down of cytoplasmic diffusion allows in vivo counting of proteins in individual cells.Real-time quantification of protein expression at the single-cell level via dynamic protein synthesis translocation reporters.Ionic imbalance, in addition to molecular crowding, abates cytoskeletal dynamics and vesicle motility during hypertonic stress.Understanding FRET as a research tool for cellular studies Osmostress-induced cell volume loss delays yeast Hog1 signaling by limiting diffusion processes and by Hog1-specific effects Decreased Uptake and Enhanced Mitochondrial Protection Underlie Reduced Toxicity of Nanoceria in Human Monocyte-Derived Macrophages Wide-ranging and unexpected consequences of altered Pol II catalytic activity in vivo.Response of Escherichia coli growth rate to osmotic shock The dynamics of signal amplification by macromolecular assemblies for the control of chromosome segregation Connecting the dots: the effects of macromolecular crowding on cell physiology.Calcineurin regulates the yeast synaptojanin Inp53/Sjl3 during membrane stress.ATG18 and FAB1 are involved in dehydration stress tolerance in Saccharomyces cerevisiae.Perception and regulatory principles of microbial growth control MAPK feedback encodes a switch and timer for tunable stress adaptation in yeast.Determination of Intracellular Vitrification Temperatures for Unicellular Micro Organisms under Conditions Relevant for Cryopreservation.Aspergillus penicillioides differentiation and cell division at 0.585 water activity.Characterizing Cellular Biophysical Responses to Stress by Relating Density, Deformability, and Size Amino acid metabolites that regulate G protein signaling during osmotic stress.A glucose-starvation response regulates the diffusion of macromolecules A pH-driven transition of the cytoplasm from a fluid- to a solid-like state promotes entry into dormancy.Regulating the Membrane Transport Activity and Death of Cells via Electroosmotic Manipulation Dramatic influence of patchy attractions on short-time protein diffusion under crowded conditions Single-Cell and Single-Molecule Analysis of Gene Expression Regulation.Mechanosensitive physiology of Chlamydomonas reinhardtii under direct membrane distortion.Survival kit of Saccharomyces cerevisiae for anhydrobiosis.An integrated view on a eukaryotic osmoregulation system.Chromatin decondensation is accompanied by a transient increase in transcriptional output.Role of Proteome Physical Chemistry in Cell Behavior.Yeast AMP-activated protein kinase monitors glucose concentration changes and absolute glucose levels.Analysis of osmoadaptation system in budding yeast suggests that regulated degradation of glycerol synthesis enzyme is key to near-perfect adaptation.Coordinated gene regulation in the initial phase of salt stress adaptation.Additive effects on the energy barrier for synaptic vesicle fusion cause supralinear effects on the vesicle fusion rate.Concomitant osmotic and chaotropicity-induced stresses in Aspergillus wentii: compatible solutes determine the biotic window.Different Mechanisms Confer Gradual Control and Memory at Nutrient- and Stress-Regulated Genes in Yeast.Transcription factor clusters regulate genes in eukaryotic cells.Measurement of Rapid Protein Diffusion in the Cytoplasm by Photo-Converted Intensity Profile Expansion.The yeast osmostress response is carbon source dependent Hyperosmotic Agents and Antibiotics Affect Dissolved Oxygen and pH Concentration Gradients in Staphylococcus aureus Biofilms.Cell volume change through water efflux impacts cell stiffness and stem cell fate.Applications of Microfluidics in Quantitative Biology.

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P2860

Severe osmotic compression triggers a slowdown of intracellular signaling, which can be explained by molecular crowding

http://www.wikidata.org/entity/Q30538482

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2013 nî lūn-bûn

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2013 թուականի Մարտին հրատարակուած գիտական յօդուած

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2013 թվականի մարտին հրատարակված գիտական հոդված

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Severe osmotic compression tri ...... xplained by molecular crowding

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Severe osmotic compression tri ...... xplained by molecular crowding

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Proceedings of the National Academy of Sciences of the United States of America

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Severe osmotic compression tri ...... xplained by molecular crowding

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P2093

Agnès Miermont

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François Waharte

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Megan Nicole McClean

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Pascal Hersen

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Samuel Bottani

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Shiqiong Hu

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Sébastien Léon

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P2860

The frequency dependence of osmo-adaptation in Saccharomyces cerevisiae

Macromolecular crowding and confinement: biochemical, biophysical, and potential physiological consequences

The TOR signalling pathway controls nuclear localization of nutrient-regulated transcription factors.

Phosphorylation of Hsl1 by Hog1 leads to a G2 arrest essential for cell survival at high osmolarity

Nuclear localization of the C2H2 zinc finger protein Msn2p is regulated by stress and protein kinase A activity

A pathway for association of receptors, adaptors, and actin during endocytic internalization.

Hog1 mediates cell-cycle arrest in G1 phase by the dual targeting of Sic1.

Localization of components involved in protein transport and processing through the yeast Golgi apparatus

A new vital stain for visualizing vacuolar membrane dynamics and endocytosis in yeast

The influence of macromolecular crowding and macromolecular confinement on biochemical reactions in physiological media

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5725-5730

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14

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110

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François Waharte

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2013-03-14T00:00:00Z

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236048012

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23493557

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3619343

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