Neuroanatomical abnormalities as risk factors for bipolar disorder.
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The Intense World Theory – A Unifying Theory of the Neurobiology of AutismDiffusion tensor imaging in first degree relatives of schizophrenia and bipolar disorder patientsCellular plasticity cascades in the pathophysiology and treatment of bipolar disorderA neural model of voluntary and automatic emotion regulation: implications for understanding the pathophysiology and neurodevelopment of bipolar disorder.Identification and individualized prediction of clinical phenotypes in bipolar disorders using neurocognitive data, neuroimaging scans and machine learning.Hyperintense MRI lesions in bipolar disorder: A meta-analysis and review.White-matter hyperintensities in first-episode psychosisA voxel-based diffusion tensor imaging study of white matter in bipolar disorder.White matter development in adolescence: the influence of puberty and implications for affective disordersPharmacological treatment of bipolar disorder among children and adolescents.Insulin resistance, diabetes mellitus, and brain structure in bipolar disordersAltered development of white matter in youth at high familial risk for bipolar disorder: a diffusion tensor imaging studyAmygdala enlargement in unaffected offspring of bipolar parents.Analysis of the presence or absence of atrophy of the subgenual and subcallosal cingulate cortices using voxel-based morphometry on MRI is useful to select prescriptions for patients with depressive symptomsHippocampal volume is reduced in schizophrenia and schizoaffective disorder but not in psychotic bipolar I disorder demonstrated by both manual tracing and automated parcellation (FreeSurfer).The intense world syndrome--an alternative hypothesis for autismCurrent research in child and adolescent bipolar disorder.Hippocampal morphology in lithium and non-lithium-treated bipolar I disorder patients, non-bipolar co-twins, and control twins.The transcription factor SP4 is reduced in postmortem cerebellum of bipolar disorder subjects: control by depolarization and lithium.Loss of erbB signaling in oligodendrocytes alters myelin and dopaminergic function, a potential mechanism for neuropsychiatric disorders.Using structural MRI to identify individuals at genetic risk for bipolar disorders: a 2-cohort, machine learning study.Transcription factor SP4 phosphorylation is altered in the postmortem cerebellum of bipolar disorder and schizophrenia subjectsDistinct and Shared Endophenotypes of Neural Substrates in Bipolar and Major Depressive Disorders.Rostral anterior cingulate cortex volume correlates with depressed mood in normal healthy children.Subgenual cingulate volumes in affected and unaffected offspring of bipolar parents.Brain structural signature of familial predisposition for bipolar disorder: replicable evidence for involvement of the right inferior frontal gyrus.Reduced hippocampal volumes in healthy carriers of brain-derived neurotrophic factor Val66Met polymorphism: meta-analysisEarly identification and high-risk strategies for bipolar disorder.nArgBP2 regulates excitatory synapse formation by controlling dendritic spine morphology.Subcortical gray matter volume abnormalities in healthy bipolar offspring: potential neuroanatomical risk marker for bipolar disorder?Alteration of cyclic-AMP response element binding protein in the postmortem brain of subjects with bipolar disorder and schizophrenia.Effects of ANK3 variation on gray and white matter in bipolar disorder.Translating neurotrophic and cellular plasticity: from pathophysiology to improved therapeutics for bipolar disorder.A role for white matter abnormalities in the pathophysiology of bipolar disorder.Signaling mechanisms regulating myelination in the central nervous system.Recognition of bipolar disorder type I before the first manic episode: challenges and developments.Glial pathology in bipolar disorder: potential therapeutic implications.Interhemispheric white matter integrity in young people with bipolar disorder and at high genetic risk.The burden of mood-disorder/cerebrovascular disease comorbidity: essential neurobiology, psychopharmacology, and physical activity interventions.Increased bcl-2 Protein Levels in Rat Primary Astrocyte Culture Following Chronic Lithium Treatment.
P2860
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P2860
Neuroanatomical abnormalities as risk factors for bipolar disorder.
description
2005 nî lūn-bûn
@nan
2005 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Neuroanatomical abnormalities as risk factors for bipolar disorder.
@ast
Neuroanatomical abnormalities as risk factors for bipolar disorder.
@en
type
label
Neuroanatomical abnormalities as risk factors for bipolar disorder.
@ast
Neuroanatomical abnormalities as risk factors for bipolar disorder.
@en
prefLabel
Neuroanatomical abnormalities as risk factors for bipolar disorder.
@ast
Neuroanatomical abnormalities as risk factors for bipolar disorder.
@en
P2860
P1433
P1476
Neuroanatomical abnormalities as risk factors for bipolar disorder.
@en
P2093
Martin Alda
Normand Carrey
P2860
P304
P356
10.1111/J.1399-5618.2005.00238.X
P50
P577
2005-10-01T00:00:00Z