Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole. - Wikidata - wikimedia - TriplyDB

Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole.

Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole.

http://www.wikidata.org/entity/Q33655969

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The Role of Cardiolipin in Cardiovascular Health Therapeutic targeting of autophagy: potential and concerns in treating cardiovascular disease Cardiovascular autophagy: crossroads of pathology, pharmacology and toxicology Autophagy in cardiovascular biology Myocardial autophagic energy stress responses--macroautophagy, mitophagy, and glycophagy The divergent roles of autophagy in ischemia and preconditioning Antioxidant Treatment and Induction of Autophagy Cooperate to Reduce Desmin Aggregation in a Cellular Model of Desminopathy Berbamine postconditioning protects the heart from ischemia/reperfusion injury through modulation of autophagy Preconditioning involves selective mitophagy mediated by Parkin and p62/SQSTM1.Autophagy induced by ischemic preconditioning is essential for cardioprotection.Molecular mechanisms of autophagy in the cardiovascular system.Lipid-induced NOX2 activation inhibits autophagic flux by impairing lysosomal enzyme activity Autophagy as a therapeutic target in cardiovascular disease.Cardioprotective Effect of Licochalcone D against Myocardial Ischemia/Reperfusion Injury in Langendorff-Perfused Rat Hearts.Cell death pathways in acute ischemia/reperfusion injury.Profound cardioprotection with chloramphenicol succinate in the swine model of myocardial ischemia-reperfusion injury Autophagy and protein aggregation after brain ischemia Tuning flux: autophagy as a target of heart disease therapy.Polyamine depletion inhibits the autophagic response modulating Trypanosoma cruzi infectivity Activation of the homeostatic intracellular repair response during cardiac surgery Autophagy: an affair of the heart Therapeutic targeting of autophagy in disease: biology and pharmacology.Autophagy: definition, molecular machinery, and potential role in myocardial ischemia-reperfusion injury.Oxidative stress and autophagy in cardiac disease, neurological disorders, aging and cancer.Mitochondrial turnover in the heart.Autophagy as a therapeutic target for ischaemia /reperfusion injury? Concepts, controversies, and challenges.Autophagy, myocardial protection, and the metabolic syndrome.Pharmacological modulation of autophagy during cardiac stress.Let-7i-Induced Atg4B Suppression Is Essential for Autophagy of Placental Trophoblast in Preeclampsia.PKCδ Inhibition Impairs Mammary Cancer Proliferative Capacity But Selects Cancer Stem Cells, Involving Autophagy.Cardiac ubiquitin ligases: Their role in cardiac metabolism, autophagy, cardioprotection and therapeutic potential.Autophagy-from molecular mechanisms to clinical relevance.Ischemia/Reperfusion.New and revisited approaches to preserving the reperfused myocardium.Regulation of BECN1-Mediated Autophagy by HSPB6: Insights from A Human HSPB6(S10F) Mutant.

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P2860

Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole.

http://www.wikidata.org/entity/Q33655969

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

@zh-hk

2009年論文

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2009年論文

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2009年论文

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name

Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole.

@ast

Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole.

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type

label

Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole.

@ast

Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole.

@en

prefLabel

Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole.

@ast

Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole.

@en

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AJPHEART.00716.2009

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American Journal of Physiology - Heart and Circulatory Physiology

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Autophagy and protein kinase C are required for cardioprotection by sulfaphenazole.

@en

P2093

Anne Hamacher-Brady

http://www.w3.org/2001/XMLSchema#string

Chengqun Huang

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Cynthia N Perry

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Hua Yuan

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Robert M Mentzer

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Roberta A Gottlieb

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Smadar Yitzhaki

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Wayne Liu

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Yayoi Tetsuo Tsukada

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Youngil Lee

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P2860

Mechanism of activation of protein kinase B by insulin and IGF-1

Essential roles of Atg5 and FADD in autophagic cell death: dissection of autophagic cell death into vacuole formation and cell death

Response to myocardial ischemia/reperfusion injury involves Bnip3 and autophagy

Reduction of ischemia and reperfusion-induced myocardial damage by cytochrome P450 inhibitors.

Arachidonic acid protects neonatal rat cardiac myocytes from ischaemic injury through epsilon protein kinase C.

Essential activation of PKC-delta in opioid-initiated cardioprotection.

Sulfaphenazole protects heart against ischemia-reperfusion injury and cardiac dysfunction by overexpression of iNOS, leading to enhancement of nitric oxide bioavailability and tissue oxygenation.

Opposing cardioprotective actions and parallel hypertrophic effects of delta PKC and epsilon PKC.

TAT-mediated protein transduction into mammalian cells.

Cardioprotective mechanisms of PKC isozyme-selective activators and inhibitors in the treatment of ischemia-reperfusion injury.

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