NADPH oxidases regulate cell growth and migration in myeloid cells transformed by oncogenic tyrosine kinases.
about
Targeting JAK2 in the therapy of myeloproliferative neoplasmsUbiquitin C-terminal hydrolase-L1 increases cancer cell invasion by modulating hydrogen peroxide generated via NADPH oxidase 4Signaling and stress: The redox landscape in NOS2 biologyH2O2 production downstream of FLT3 is mediated by p22phox in the endoplasmic reticulum and is required for STAT5 signalling.Role of c-Met/phosphatidylinositol 3-kinase (PI3k)/Akt signaling in hepatocyte growth factor (HGF)-mediated lamellipodia formation, reactive oxygen species (ROS) generation, and motility of lung endothelial cells.Overexpression of pyruvate dehydrogenase kinase 1 in retinoblastoma: A potential therapeutic opportunity for targeting vitreous seeds and hypoxic regions.Novel roles of reactive oxygen species in the pathogenesis of acute myeloid leukemia.Kinase domain mutations confer resistance to novel inhibitors targeting JAK2V617F in myeloproliferative neoplasms.Targeting myristoylated alanine-rich C kinase substrate phosphorylation site domain in lung cancer. Mechanisms and therapeutic implications.A key role for NOX4 in epithelial cell death during development of lung fibrosis.The JAK2V617F oncogene requires expression of inducible phosphofructokinase/fructose-bisphosphatase 3 for cell growth and increased metabolic activityRedox signaling at invasive microdomains in cancer cells.A ROS-Activatable Agent Elicits Homologous Recombination DNA Repair and Synergizes with Pathway CompoundsPathological glycogenesis through glycogen synthase 1 and suppression of excessive AMP kinase activity in myeloid leukemia cells.Regulation of Reactive Oxygen Species and the Antioxidant Protein DJ-1 in Mastocytosis.A peptide that inhibits function of Myristoylated Alanine-Rich C Kinase Substrate (MARCKS) reduces lung cancer metastasis.A NOX2/Egr-1/Fyn pathway delineates new targets for TKI-resistant malignanciesRedox control of leukemia: from molecular mechanisms to therapeutic opportunities.SAP domain-dependent Mkl1 signaling stimulates proliferation and cell migration by induction of a distinct gene set indicative of poor prognosis in breast cancer patients.Proteome alterations associated with transformation of multiple myeloma to secondary plasma cell leukemia.Never-ageing cellular senescenceProtein tyrosine phosphatase structure-function relationships in regulation and pathogenesis.Aiding and abetting roles of NOX oxidases in cellular transformation.Oxidative metabolism in cancer: A STAT affair?ROS signalling, NADPH oxidases and cancer.Cyclin D1 unbalances the redox status controlling cell adhesion, migration, and drug resistance in myeloma cells.NADPH oxidase-generated hydrogen peroxide induces DNA damage in mutant FLT3-expressing leukemia cells.Jab1/Csn5-Thioredoxin Signaling in Relapsed Acute Monocytic Leukemia under Oxidative Stress.FLT3-driven redox-modulation of Ezrin regulates leukaemic cell migration.Dietary phenolic acids act as effective antioxidants in membrane models and in cultured cells, exhibiting proapoptotic effects in leukaemia cellsParadoxical roles of dual oxidases in cancer biology.Inhibition of the NADPH oxidase regulates heme oxygenase 1 expression in chronic myeloid leukemia.Acute myeloid leukemia cells require 6-phosphogluconate dehydrogenase for cell growth and NADPH-dependent metabolic reprogramming.NCF2/p67phox: A novel player in the anti-apoptotic functions of p53.Alterations in the Reactive Oxygen Species in Peripheral Blood of Chronic Myeloid Leukaemia Patients from Northern India.MARCKS promotes invasion and is associated with biochemical recurrence in prostate cancer.A novel predictor of cancer malignancy: up-regulation of myristoylated alanine-rich C kinase substrate phosphorylation in lung cancer.Role of NOX2 for leukaemic expansion in a murine model of BCR-ABL1(+) leukaemia.NOX4-driven ROS formation mediates PTP inactivation and cell transformation in FLT3ITD-positive AML cells.Nuclear membrane-localised NOX4D generates pro-survival ROS in FLT3-ITD-expressing AML.
P2860
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P2860
NADPH oxidases regulate cell growth and migration in myeloid cells transformed by oncogenic tyrosine kinases.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
name
NADPH oxidases regulate cell g ...... by oncogenic tyrosine kinases.
@ast
NADPH oxidases regulate cell g ...... by oncogenic tyrosine kinases.
@en
type
label
NADPH oxidases regulate cell g ...... by oncogenic tyrosine kinases.
@ast
NADPH oxidases regulate cell g ...... by oncogenic tyrosine kinases.
@en
prefLabel
NADPH oxidases regulate cell g ...... by oncogenic tyrosine kinases.
@ast
NADPH oxidases regulate cell g ...... by oncogenic tyrosine kinases.
@en
P2093
P2860
P356
P1433
P1476
NADPH oxidases regulate cell g ...... by oncogenic tyrosine kinases.
@en
P2093
J D Griffin
M S Fernandes
R L Levine
P2860
P2888
P304
P356
10.1038/LEU.2010.263
P577
2010-11-12T00:00:00Z
P5875
P6179
1000863387