Type-I IFN signaling suppresses an excessive IFN-gamma response and thus prevents lung damage and chronic inflammation during Pneumocystis (PC) clearance in CD4 T cell-competent mice.
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Overlapping, additive and counterregulatory effects of type II and I interferons on myeloid dendritic cell functionsChemokine Receptor Ccr7 Restricts Fatal West Nile Virus EncephalitisHigher-Order Chromatin Regulation of Inflammatory Gene ExpressionRegulation of IFN-γ by IL-13 dictates susceptibility to secondary postinfluenza MRSA pneumonia.IL-33 and M2a alveolar macrophages promote lung defense against the atypical fungal pathogen Pneumocystis murina.Pneumocystis infection in an immunocompetent host can promote collateral sensitization to respiratory antigens.The role of macrophage IL-10/innate IFN interplay during virus-induced asthma.Dynamic roles of type I and type II IFNs in early infection with Mycobacterium tuberculosis.Increased toll-like receptors and p53 levels regulate apoptosis and angiogenesis in non-muscle invasive bladder cancer: mechanism of action of P-MAPA biological response modifier.Differential Type I Interferon Signaling Is a Master Regulator of Susceptibility to Postinfluenza Bacterial Superinfection.OM-85 is an immunomodulator of interferon-β production and inflammasome activity.Non-hematopoietic cells contribute to protective tolerance to Aspergillus fumigatus via a TRIF pathway converging on IDO.Cytokines and the regulation of fungus-specific CD4 T cell differentiation.Vascular Dysfunction in Pneumocystis-Associated Pulmonary Hypertension Is Related to Endothelin Response and Adrenomedullin Concentration.Biomimetic antigenic nanoparticles elicit controlled protective immune response to influenza.Modulation of inflammasome-mediated pulmonary immune activation by type I IFNs protects bone marrow homeostasis during systemic responses to Pneumocystis lung infection.Endothelial cells and fibroblasts amplify the arthritogenic type I IFN response in murine Lyme disease and are major sources of chemokines in Borrelia burgdorferi-infected joint tissue.STAT4-dependent and -independent Th2 responses correlate with protective immunity against lung infection with Pneumocystis murina.Type I Interferon Signaling Is Required for CpG-Oligodesoxynucleotide-Induced Control of Leishmania major, but Not for Spontaneous Cure of Subcutaneous Primary or Secondary L. major Infection.
P2860
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P2860
Type-I IFN signaling suppresses an excessive IFN-gamma response and thus prevents lung damage and chronic inflammation during Pneumocystis (PC) clearance in CD4 T cell-competent mice.
description
2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Type-I IFN signaling suppresse ...... in CD4 T cell-competent mice.
@ast
Type-I IFN signaling suppresse ...... in CD4 T cell-competent mice.
@en
type
label
Type-I IFN signaling suppresse ...... in CD4 T cell-competent mice.
@ast
Type-I IFN signaling suppresse ...... in CD4 T cell-competent mice.
@en
prefLabel
Type-I IFN signaling suppresse ...... in CD4 T cell-competent mice.
@ast
Type-I IFN signaling suppresse ...... in CD4 T cell-competent mice.
@en
P2093
P2860
P1476
Type-I IFN signaling suppresse ...... in CD4 T cell-competent mice.
@en
P2093
Allen G Harmsen
Kate McInnerney
Nicole Meissner
Steve Swain
P2860
P304
P356
10.2353/AJPATH.2010.091158
P407
P577
2010-04-15T00:00:00Z