Overexpression of SK3 channels dampens uterine contractility to prevent preterm labor in mice.
about
Transforming growth factor β signaling in uterine development and functionOverexpression of the SK3 channel alters vascular remodeling during pregnancy, leading to fetal demise.A computational model of the ionic currents, Ca2+ dynamics and action potentials underlying contraction of isolated uterine smooth muscle.Uterine dysfunction in biglycan and decorin deficient mice leads to dystocia during parturition.Kv7 and Kv11 channels in myometrial regulation.SK3 channel expression during pregnancy is regulated through estrogen and Sp factor-mediated transcriptional control of the KCNN3 gene.The inwardly rectifying K+ channel KIR7.1 controls uterine excitability throughout pregnancyIn vivo measurement of intrauterine pressure by telemetry: a new approach for studying parturition in mouse modelsSingle-nucleotide polymorphisms in the KCNN3 gene associate with preterm birth.Expression of the small conductance Ca²⁺-activated potassium channel subtype 3 (SK3) in rat uterus after stimulation with 17β-estradiol.Immunolocalization and expression of small-conductance calcium-activated potassium channels in human myometriumReconstruction of Cell Surface Densities of Ion Pumps, Exchangers, and Channels from mRNA Expression, Conductance Kinetics, Whole-Cell Calcium, and Current-Clamp Voltage Recordings, with an Application to Human Uterine Smooth Muscle Cells.Polymorphisms in the fetal progesterone receptor and a calcium-activated potassium channel isoform are associated with preterm birth in an Argentinian population.Insights into parturition biology from genetically altered mice.The contribution of Kv7 channels to pregnant mouse and human myometrial contractility.Association of candidate gene polymorphisms with clinical subtypes of preterm birth in a Latin American population.CyPPA, a positive modulator of small-conductance Ca(2+)-activated K(+) channels, inhibits phasic uterine contractions and delays preterm birth in mice.Identification of uterine pacemaker regions at the myometrial-placental interface in the rat.The Small-Conductance Ca2+-Activated Potassium Channel, Subtype SK3, in the Human Myometrium Is Downregulated in Early Stages of Pregnancy
P2860
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P2860
Overexpression of SK3 channels dampens uterine contractility to prevent preterm labor in mice.
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2008 nî lūn-bûn
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2008 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի փետրվարին հրատարակված գիտական հոդված
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2008年の論文
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2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Overexpression of SK3 channels ...... prevent preterm labor in mice.
@ast
Overexpression of SK3 channels ...... prevent preterm labor in mice.
@en
Overexpression of SK3 channels ...... prevent preterm labor in mice.
@nl
type
label
Overexpression of SK3 channels ...... prevent preterm labor in mice.
@ast
Overexpression of SK3 channels ...... prevent preterm labor in mice.
@en
Overexpression of SK3 channels ...... prevent preterm labor in mice.
@nl
prefLabel
Overexpression of SK3 channels ...... prevent preterm labor in mice.
@ast
Overexpression of SK3 channels ...... prevent preterm labor in mice.
@en
Overexpression of SK3 channels ...... prevent preterm labor in mice.
@nl
P2093
P2860
P1476
Overexpression of SK3 channels ...... prevent preterm labor in mice.
@en
P2093
Jessica D K Kresowik
Kathryn G Lamping
Sarah K England
Stephanie L Pierce
P2860
P304
P356
10.1095/BIOLREPROD.107.066423
P577
2008-02-27T00:00:00Z