Desmoglein 2 mutant mice develop cardiac fibrosis and dilation. - Wikidata - wikimedia - TriplyDB

Desmoglein 2 mutant mice develop cardiac fibrosis and dilation.

Desmoglein 2 mutant mice develop cardiac fibrosis and dilation.

http://www.wikidata.org/entity/Q35017328

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Mutations with pathogenic potential in proteins located in or at the composite junctions of the intercalated disk connecting mammalian cardiomyocytes: a reference thesaurus for arrhythmogenic cardiomyopathies and for Naxos and Carvajal diseases Circulating NOS3 modulates left ventricular remodeling following reperfused myocardial infarction Histological and ultrastructural abnormalities in murine desmoglein 2-mutant hearts Plakophilin-2 and the migration, differentiation and transformation of cells derived from the epicardium of neonatal rat hearts Dissection of keratin network formation, turnover and reorganization in living murine embryos.A novel role for the fifth component of complement (C5) in cardiac physiology.Mechanotransduction in cardiac hypertrophy and failure.Left ventricular remodeling in swine after myocardial infarction: a transcriptional genomics approach.Transgenic mice overexpressing desmocollin-2 (DSC2) develop cardiomyopathy associated with myocardial inflammation and fibrotic remodeling.Central role for GSK3β in the pathogenesis of arrhythmogenic cardiomyopathy.A non-canonical role for desmoglein-2 in endothelial cells: implications for neoangiogenesis.Pathophysiology of arrhythmogenic cardiomyopathy.Mouse models in arrhythmogenic right ventricular cardiomyopathy.Growth differentiation factor 15 in heart failure: an update.Desmosomes in the heart: a review of clinical and mechanistic analyses.Vinexin-β protects against cardiac hypertrophy by blocking the Akt-dependent signalling pathway.Cell models of arrhythmogenic cardiomyopathy: advances and opportunities.Whole Genome Sequence Identified a Rare Homozygous Pathogenic Mutation of the DSG2 Gene in a Familial Arrhythmogenic Cardiomyopathy Involving Both Ventricles.Murine Electrophysiological Models of Cardiac Arrhythmogenesis.Depletion of circulating blood NOS3 increases severity of myocardial infarction and left ventricular dysfunction Complement system modulation as a target for treatment of arrhythmogenic cardiomyopathy.New Insight into the Variable Expression of Arrhythmogenic Right Ventricular Cardiomyopathy Provided by the Analysis of a Plakophilin-2 Splice Mutation

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Desmoglein 2 mutant mice develop cardiac fibrosis and dilation.

http://www.wikidata.org/entity/Q35017328

description

2011 nî lūn-bûn

@nan

2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

Desmoglein 2 mutant mice develop cardiac fibrosis and dilation.

@ast

Desmoglein 2 mutant mice develop cardiac fibrosis and dilation.

@en

type

label

Desmoglein 2 mutant mice develop cardiac fibrosis and dilation.

@ast

Desmoglein 2 mutant mice develop cardiac fibrosis and dilation.

@en

prefLabel

Desmoglein 2 mutant mice develop cardiac fibrosis and dilation.

@ast

Desmoglein 2 mutant mice develop cardiac fibrosis and dilation.

@en

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S00395-011-0175-Y

P1433

Basic Research in Cardiology

P1476

Desmoglein 2 mutant mice develop cardiac fibrosis and dilation

@en

P2093

Annette M van de Sandt

http://www.w3.org/2001/XMLSchema#string

Bastian Holthöfer

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Claudia A Krusche

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Ernesto Bockamp

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Leonid Eshkind

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Marc W Merx

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Sebastian Kant

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Valérie Hofe

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P2860

Arrhythmogenic right ventricular cardiomyopathy/dysplasia

Identification of mutations in the cardiac ryanodine receptor gene in families affected with arrhythmogenic right ventricular cardiomyopathy type 2 (ARVD2)

Role of genetic analysis in the management of patients with arrhythmogenic right ventricular dysplasia/cardiomyopathy

Desmosomal dysfunction due to mutations in desmoplakin causes arrhythmogenic right ventricular dysplasia/cardiomyopathy

A friend within the heart: natriuretic peptide receptor signaling

Molecular interactions between desmosomal cadherins

Suppression of canonical Wnt/beta-catenin signaling by nuclear plakoglobin recapitulates phenotype of arrhythmogenic right ventricular cardiomyopathy

Arrhythmogenic right ventricular cardiomyopathy type 5 is a fully penetrant, lethal arrhythmic disorder caused by a missense mutation in the TMEM43 gene

Direct Ca2+-dependent heterophilic interaction between desmosomal cadherins, desmoglein and desmocollin, contributes to cell-cell adhesion

C-cadherin ectodomain structure and implications for cell adhesion mechanisms

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s00395-011-0175-y

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617-633

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scholarly article

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10.1007/S00395-011-0175-Y

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4

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106

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Reinhard Windoffer

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2011-04-01T00:00:00Z

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50940896

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