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Endothelial activation by platelets from sickle cell anemia patients.

Endothelial activation by platelets from sickle cell anemia patients.

http://www.wikidata.org/entity/Q35097680

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Inhibitory Effect of Serotonin Antagonist on Leukocyte-Endothelial Interactions In Vivo and In Vitro Neutrophils, platelets, and inflammatory pathways at the nexus of sickle cell disease pathophysiology.Integrin β3/Akt signaling contributes to platelet-induced hemangioendothelioma growth.Emerging point-of-care technologies for sickle cell disease screening and monitoring.Microfluidics for investigating vaso-occlusions in sickle cell disease.Prasugrel hydrochloride for the treatment of sickle cell disease.Prominent role of platelets in the formation of circulating neutrophil-red cell heterocellular aggregates in sickle cell anemia.Activated Platelets Induce Endothelial Cell Activation via an Interleukin-1β Pathway in Systemic Lupus Erythematosus.

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Endothelial activation by platelets from sickle cell anemia patients.

http://www.wikidata.org/entity/Q35097680

description

2014 nî lūn-bûn

@nan

2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2014年の論文

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2014年論文

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2014年論文

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2014年論文

@zh-hk

2014年論文

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2014年論文

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2014年论文

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Endothelial activation by platelets from sickle cell anemia patients.

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Endothelial activation by platelets from sickle cell anemia patients.

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Endothelial activation by platelets from sickle cell anemia patients.

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Endothelial activation by platelets from sickle cell anemia patients.

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Endothelial activation by platelets from sickle cell anemia patients.

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Endothelial activation by platelets from sickle cell anemia patients.

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Ana Flávia Brugnerotto

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Daiana Morelli Vital

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Fernando Ferreira Costa

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Marilene de Fátima Reis Ribeiro

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Melissa Ercolin dos Santos

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Nicola Conran

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Renata Proença-Ferreira

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Sara T Olalla-Saad

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Vanessa Tonin Garrido

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Venina Marcela Dominical

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P2860

Platelet hyperactivity in sickle-cell disease: a consequence of hyposplenism.

Sickle-cell disease

Nuclear factor-kappaB: a pivotal transcription factor in chronic inflammatory diseases

P-selectin-mediated platelet-neutrophil aggregate formation activates neutrophils in mouse and human sickle cell disease.

Heterotypic interactions enabled by polarized neutrophil microdomains mediate thromboinflammatory injury

A pilot study of eptifibatide for treatment of acute pain episodes in sickle cell disease.

Preliminary trial of minidose heparin prophylaxis for painful sickle cell crises.

Diverse agents act at multiple levels to inhibit the Rel/NF-kappaB signal transduction pathway.

Big strokes in small persons.

Newer aspects of the pathophysiology of sickle cell disease vaso-occlusion.

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sickle-cell disease

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