IpaB, a Shigella flexneri invasin, colocalizes with interleukin-1 beta-converting enzyme in the cytoplasm of macrophages.
about
Type III protein secretion systems in bacterial pathogens of animals and plantsReactive nitrogen species contribute to innate host defense against Campylobacter jejuniThe virulence plasmid of Yersinia, an antihost genomeThe Salmonella invasin SipB induces macrophage apoptosis by binding to caspase-1Spontaneous formation of IpaB ion channels in host cell membranes reveals how Shigella induces pyroptosis in macrophagesMolecular basis of the interaction of Salmonella with the intestinal mucosa.Tripeptidyl peptidase II promotes maturation of caspase-1 in Shigella flexneri-induced macrophage apoptosisErwinia amylovora secretes DspE, a pathogenicity factor and functional AvrE homolog, through the Hrp (type III secretion) pathway.Structure-function analysis of the Shigella virulence factor IpaB.Analysis of putative Chlamydia trachomatis chaperones Scc2 and Scc3 and their use in the identification of type III secretion substrates.Identification and characterization of a novel bacterial virulence factor that shares homology with mammalian Toll/interleukin-1 receptor family proteinsThe interleukin 1beta-converting enzyme, caspase 1, is activated during Shigella flexneri-induced apoptosis in human monocyte-derived macrophagesMacrophage cell death upon intracellular bacterial infection.Next-Generation mRNA Sequencing Reveals Pyroptosis-Induced CD4+ T Cell Death in Early Simian Immunodeficiency Virus-Infected Lymphoid Tissues.Structure and biophysics of type III secretion in bacteriaRole for the chlamydial type III secretion apparatus in host cytokine expression.Apoptosis of human intestinal epithelial cells after bacterial invasionHow Do the Virulence Factors of Shigella Work Together to Cause Disease?Bacterial perturbation of cytokine networks.Lactoferrin protects rabbits from Shigella flexneri-induced inflammatory enteritis.The mechanism of cell death in Listeria monocytogenes-infected murine macrophages is distinct from apoptosis.ShiA abrogates the innate T-cell response to Shigella flexneri infection
P2860
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P2860
IpaB, a Shigella flexneri invasin, colocalizes with interleukin-1 beta-converting enzyme in the cytoplasm of macrophages.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
IpaB, a Shigella flexneri inva ...... the cytoplasm of macrophages.
@ast
IpaB, a Shigella flexneri inva ...... the cytoplasm of macrophages.
@en
type
label
IpaB, a Shigella flexneri inva ...... the cytoplasm of macrophages.
@ast
IpaB, a Shigella flexneri inva ...... the cytoplasm of macrophages.
@en
prefLabel
IpaB, a Shigella flexneri inva ...... the cytoplasm of macrophages.
@ast
IpaB, a Shigella flexneri inva ...... the cytoplasm of macrophages.
@en
P2093
P2860
P1476
IpaB, a Shigella flexneri inva ...... the cytoplasm of macrophages.
@en
P2093
A Zychlinsky
K Thirumalai
P2860
P304
P407
P577
1997-02-01T00:00:00Z