Breast cancer progression: controversies and consensus in the molecular mechanisms of metastasis and EMT
about
Estrogen stimulation of cell migration involves multiple signaling pathway interactionsDown-regulation of epithelial cadherin is required to initiate metastatic outgrowth of breast cancer.An in vitro correlation of mechanical forces and metastatic capacity.GATA1 induces epithelial-mesenchymal transition in breast cancer cells through PAK5 oncogenic signaling.Tenascin C induces epithelial-mesenchymal transition-like change accompanied by SRC activation and focal adhesion kinase phosphorylation in human breast cancer cellsTransforming growth factor-β-induced epithelial-mesenchymal transition facilitates epidermal growth factor-dependent breast cancer progressionTGFbeta/TNF(alpha)-mediated epithelial-mesenchymal transition generates breast cancer stem cells with a claudin-low phenotype.Stanniocalicin 2 suppresses breast cancer cell migration and invasion via the PKC/claudin-1-mediated signaling.PAC down-regulates estrogen receptor alpha and suppresses epithelial-to-mesenchymal transition in breast cancer cells.Peritumoral lymphangiogenesis induced by vascular endothelial growth factor C and D promotes lymph node metastasis in breast cancer patientsCurcumin inhibits hypoxia-induced migration in K1 papillary thyroid cancer cells.Epithelial-mesenchymal transition and cell cooperativity in metastasis.STC2 promotes head and neck squamous cell carcinoma metastasis through modulating the PI3K/AKT/Snail signaling.AXL induces epithelial-to-mesenchymal transition and regulates the function of breast cancer stem cells.Histone deacetylase inhibitor entinostat reverses epithelial to mesenchymal transition of breast cancer cells by reversing the repression of E-cadherin.Comparative proteome profiling of breast tumor cell lines by gel electrophoresis and mass spectrometry reveals an epithelial mesenchymal transition associated protein signature.STAT3, a Poor Survival Predicator, Is Associated with Lymph Node Metastasis from Breast Cancer.Overexpression of ILK1 in breast cancer associates with poor prognosis.
P2860
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P2860
Breast cancer progression: controversies and consensus in the molecular mechanisms of metastasis and EMT
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Breast cancer progression: con ...... chanisms of metastasis and EMT
@ast
Breast cancer progression: con ...... chanisms of metastasis and EMT
@en
type
label
Breast cancer progression: con ...... chanisms of metastasis and EMT
@ast
Breast cancer progression: con ...... chanisms of metastasis and EMT
@en
prefLabel
Breast cancer progression: con ...... chanisms of metastasis and EMT
@ast
Breast cancer progression: con ...... chanisms of metastasis and EMT
@en
P2860
P1476
Breast cancer progression: con ...... chanisms of metastasis and EMT
@en
P2093
Danny R Welch
Pamela Cowin
P2860
P2888
P304
P356
10.1007/S10911-007-9041-9
P577
2007-09-01T00:00:00Z