Reactive hyperemia is not responsible for stimulating muscle protein synthesis following blood flow restriction exercise. - Wikidata - wikimedia - TriplyDB

Reactive hyperemia is not responsible for stimulating muscle protein synthesis following blood flow restriction exercise.

Reactive hyperemia is not responsible for stimulating muscle protein synthesis following blood flow restriction exercise.

http://www.wikidata.org/entity/Q35994715

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Effects of blood flow restricted low-intensity concentric or eccentric training on muscle size and strength.Activation of mTORC1 signaling and protein synthesis in human muscle following blood flow restriction exercise is inhibited by rapamycin.Effect of age on basal muscle protein synthesis and mTORC1 signaling in a large cohort of young and older men and women Blood flow restricted exercise and vascular function.Role of Ingested Amino Acids and Protein in the Promotion of Resistance Exercise-Induced Muscle Protein Anabolism.Potential mechanisms for a role of metabolic stress in hypertrophic adaptations to resistance training.Characterization and regulation of mechanical loading-induced compensatory muscle hypertrophy.Hypoxia and resistance exercise: a comparison of localized and systemic methods.A review on the mechanisms of blood-flow restriction resistance training-induced muscle hypertrophy.A single 60-min bout of peristaltic pulse external pneumatic compression transiently upregulates phosphorylated ribosomal protein s6.Effects of low-load resistance exercise with blood flow restriction on high-energy phosphate metabolism and oxygenation level in skeletal muscle.Effects of a pre-workout supplement on hyperemia following leg extension resistance exercise to failure with different resistance loads.Blood flow restriction: how does it work?Blood flow restriction pressure recommendations: a tale of two cuffs.Practical blood flow restriction training increases muscle hypertrophy during a periodized resistance training programme.Acute low-load resistance exercise with and without blood flow restriction increased protein signalling and number of satellite cells in human skeletal muscle.Increased FXYD1 and PGC-1α mRNA after blood flow-restricted running is related to fibre type-specific AMPK signalling and oxidative stress in human muscle.Blood flow restriction late in recovery after heavy resistance exercise hampers muscle recuperation.Blood flow in humans following low-load exercise with and without blood flow restriction.Post-exercise blood flow restriction attenuates muscle hypertrophy.Acute low-intensity cycling with blood-flow restriction has no effect on metabolic signaling in human skeletal muscle compared to traditional exercise.Effects of walking combined with restricted leg blood flow on mTOR and MAPK signalling in young men.Pharmacological enhancement of leg and muscle microvascular blood flow does not augment anabolic responses in skeletal muscle of young men under fed conditions.Effects of exercise with and without different degrees of blood flow restriction on torque and muscle activation Commentaries on Viewpoint: What is the relationship between acute measure of muscle protein synthesis and changes in muscle mass?

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P2860

Reactive hyperemia is not responsible for stimulating muscle protein synthesis following blood flow restriction exercise.

http://www.wikidata.org/entity/Q35994715

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Reactive hyperemia is not resp ...... ood flow restriction exercise.

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Reactive hyperemia is not resp ...... ood flow restriction exercise.

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Reactive hyperemia is not resp ...... ood flow restriction exercise.

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Reactive hyperemia is not resp ...... ood flow restriction exercise.

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Reactive hyperemia is not resp ...... ood flow restriction exercise.

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Reactive hyperemia is not resp ...... ood flow restriction exercise.

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JAPPLPHYSIOL.01267.2011

P1433

Journal of Applied Physiology

P1476

Reactive hyperemia is not resp ...... ood flow restriction exercise.

@en

P2093

Blake B Rasmussen

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Christopher S Fry

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David M Gundermann

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Dillon K Walker

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Elena Volpi

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Jared M Dickinson

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Kyle L Timmerman

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Micah J Drummond

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Nutritional and contractile regulation of human skeletal muscle protein synthesis and mTORC1 signaling

Pharmacological vasodilation improves insulin-stimulated muscle protein anabolism but not glucose utilization in older adults.

Insulin resistance of muscle protein metabolism in aging.

Low-load high volume resistance exercise stimulates muscle protein synthesis more than high-load low volume resistance exercise in young men.

Supraphysiological hyperinsulinaemia is necessary to stimulate skeletal muscle protein anabolism in older adults: evidence of a true age-related insulin resistance of muscle protein metabolism.

Blood flow restriction exercise stimulates mTORC1 signaling and muscle protein synthesis in older men

Insulin stimulates human skeletal muscle protein synthesis via an indirect mechanism involving endothelial-dependent vasodilation and mammalian target of rapamycin complex 1 signaling

Nutrient signalling in the regulation of human muscle protein synthesis.

Time course of proteolytic, cytokine, and myostatin gene expression after acute exercise in human skeletal muscle.

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1520-1528

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scholarly article

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10.1152/JAPPLPHYSIOL.01267.2011

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9

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112

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2012-02-23T00:00:00Z

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221858702

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22362401

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