MicroRNA214 Is Associated With Progression of Ulcerative Colitis, and Inhibition Reduces Development of Colitis and Colitis-Associated Cancer in Mice - Wikidata - wikimedia - TriplyDB

MicroRNA214 Is Associated With Progression of Ulcerative Colitis, and Inhibition Reduces Development of Colitis and Colitis-Associated Cancer in Mice

MicroRNA214 Is Associated With Progression of Ulcerative Colitis, and Inhibition Reduces Development of Colitis and Colitis-Associated Cancer in Mice

http://www.wikidata.org/entity/Q36096047

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MicroRNAs: Clinical Relevance in Colorectal Cancer GFI1 downregulation promotes inflammation-linked metastasis of colorectal cancer.Colorectal Cancer: From the Genetic Model to Posttranscriptional Regulation by Noncoding RNAs.Expression Profiles of miRNA Subsets Distinguish Human Colorectal Carcinoma and Normal Colonic Mucosa.Downregulated expression of microRNA-124 in pediatric intestinal failure patients modulates macrophages activation by inhibiting STAT3 and AChE.Effects of Herb-Partitioned Moxibustion on the miRNA Expression Profiles in Colon from Rats with DSS-Induced Ulcerative Colitis rs35301225 polymorphism in miR-34a promotes development of human colon cancer by deregulation of 3'UTR in E2F1 in Chinese population.Upregulation of miR-665 promotes apoptosis and colitis in inflammatory bowel disease by repressing the endoplasmic reticulum stress components XBP1 and ORMDL3.Chemopreventive Strategies for Inflammation-Related Carcinogenesis: Current Status and Future Direction.MicroRNAs: how many in inflammatory bowel disease?Function analysis of rs9589207 polymorphism in miR-92a in gastric cancer.MicroRNAs and Inflammation in Colorectal Cancer.MicroRNAs at the epicenter of intestinal homeostasis.Challenges for epigenetic research in inflammatory bowel diseases.Role of MiRNAs in Inflammatory Bowel Disease.MicroRNAs in mucosal inflammation.Unique microRNA expression in the colonic mucosa during chronic HIV-1 infection.Maintenance of cancer stemness by miR-196b-5p contributes to chemoresistance of colorectal cancer cells via activating STAT3 signaling pathway A long noncoding RNA signature for ulcerative colitis identifies IFNG-AS1 as an enhancer of inflammation.High throughput RNA sequencing utility for diagnosis and prognosis in colon diseases.MicroRNAs as Therapeutic Targets in Colitis and Colitis-Associated Cancer: Tiny Players With a Giant Impact Role of G protein-coupled receptors-microRNA interactions in gastrointestinal pathophysiology.Environmental triggers in IBD: a review of progress and evidence.Identification of Circulating MicroRNA Signatures in Crohn's Disease Using the Nanostring nCounter Technology.MicroRNAs in intestinal barrier function, inflammatory bowel disease and related cancers-their effects and therapeutic potentials.CRHR2/Ucn2 signaling is a novel regulator of miR-7/YY1/Fas circuitry contributing to reversal of colorectal cancer cell resistance to Fas-mediated apoptosis.Novel therapeutic concepts for inflammatory bowel disease-from bench to bedside.MicroRNA-31-3p Is Involved in Substance P (SP)-Associated Inflammation in Human Colonic Epithelial Cells and Experimental Colitis.Functional role and therapeutic targeting of microRNAs in inflammatory bowel disease.A step-by-step microRNA guide to cancer development and metastasis.MicroRNA-30a attenuates mutant KRAS-driven colorectal tumorigenesis via direct suppression of ME1.PIAS3-mediated feedback loops promote chronic colitis-associated malignant transformation.Early detection of ulcerative colitis-associated colorectal cancer.Increased mucosal expression of miR-215 precedes the development of neoplasia in patients with long-standing ulcerative colitis.The Dual Role of MicroRNAs in Colorectal Cancer Progression

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MicroRNA214 Is Associated With Progression of Ulcerative Colitis, and Inhibition Reduces Development of Colitis and Colitis-Associated Cancer in Mice

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Gastroenterology

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Andrea E van der Meulen-de Jong

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Angelos Oikonomopoulos

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Charalabos Pothoulakis

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Christina Vorvis

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Christos Polytarchou

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Daniel W Hommes

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Dimitrios Iliopoulos

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Eleni Birli

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Hein W Verspaget

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Jennifer Choi

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P2860

Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease

The kinase Akt1 controls macrophage response to lipopolysaccharide by regulating microRNAs

An epigenetic switch involving NF-kappaB, Lin28, Let-7 MicroRNA, and IL6 links inflammation to cell transformation

The risk of colorectal cancer in ulcerative colitis: a meta-analysis

Mammalian microRNAs predominantly act to decrease target mRNA levels

Genome-wide association identifies multiple ulcerative colitis susceptibility loci

Inflammation and cancer: back to Virchow?

NF-kappaB is a target of AKT in anti-apoptotic PDGF signalling

Therapeutic silencing of microRNA-122 in primates with chronic hepatitis C virus infection

Akt-dependent regulation of NF-{kappa}B is controlled by mTOR and Raptor in association with IKK.

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Maria Hatziapostolou

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