Programmed elimination of cells by caspase-independent cell extrusion in C. elegans. - Wikidata - wikimedia - TriplyDB

Programmed elimination of cells by caspase-independent cell extrusion in C. elegans.

Programmed elimination of cells by caspase-independent cell extrusion in C. elegans.

http://www.wikidata.org/entity/Q36157242

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STRAD pseudokinases regulate axogenesis and LKB1 stability Xk-related protein 8 and CED-8 promote phosphatidylserine exposure in apoptotic cells Epithelial homeostasis Both the caspase CSP-1 and a caspase-independent pathway promote programmed cell death in parallel to the canonical pathway for apoptosis in Caenorhabditis elegans Cell turnover and detritus production in marine sponges from tropical and temperate benthic ecosystems Matrine induces caspase-independent program cell death in hepatocellular carcinoma through bid-mediated nuclear translocation of apoptosis inducing factor The Par3-like polarity protein Par3L is essential for mammary stem cell maintenance.Noncanonical cell death in the nematode Caenorhabditis elegans.Asymmetric neuroblast divisions producing apoptotic cells require the cytohesin GRP-1 in Caenorhabditis elegans Programmed cell death: a new way worms get rid of unwanted cells Bringing balance by force: live cell extrusion controls epithelial cell numbers The insulin/IGF signaling regulators cytohesin/GRP-1 and PIP5K/PPK-1 modulate susceptibility to excitotoxicity in C. elegans.MELK-a conserved kinase: functions, signaling, cancer, and controversy.A genome-wide RNAi screen for enhancers of par mutants reveals new contributors to early embryonic polarity in Caenorhabditis elegans.Caenorhabditis elegans PIG-1/MELK acts in a conserved PAR-4/LKB1 polarity pathway to promote asymmetric neuroblast divisions The Caenorhabditis elegans Excretory System: A Model for Tubulogenesis, Cell Fate Specification, and Plasticity Negative Selection and Chromosome Instability Induced by Mad2 Overexpression Delay Breast Cancer but Facilitate Oncogene-Independent Outgrowth.An Sp1 transcription factor coordinates caspase-dependent and -independent apoptotic pathways Both the anti- and pro-apoptotic functions of villin regulate cell turnover and intestinal homeostasis.Cell Death in C. elegans Development.Comparative genetic, proteomic and phosphoproteomic analysis of C. elegans embryos with a focus on ham-1/STOX and pig-1/MELK in dopaminergic neuron development.Epithelial cell extrusion: Pathways and pathologies.Size Matters: How C. elegans Asymmetric Divisions Regulate Apoptosis.Caenorhabditis elegans CES-1 Snail Represses pig-1 MELK Expression To Control Asymmetric Cell Division.Programmed Cell Death During Caenorhabditis elegans Development Lkb1 is indispensable for skeletal muscle development, regeneration, and satellite cell homeostasis Mechanisms of apoptotic phosphatidylserine exposure.Spontaneous presence of FOXO3-specific T cells in cancer patients.If you don't want them shed them.Transcriptional control of non-apoptotic developmental cell death in C. elegans.High expression of partitioning defective 3-like protein is associated with malignancy in colorectal cancer.Extracellular cleavage of E-cadherin promotes epithelial cell extrusion.Programmed cell removal by calreticulin in tissue homeostasis and cancer

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Programmed elimination of cells by caspase-independent cell extrusion in C. elegans.

http://www.wikidata.org/entity/Q36157242

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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name

Programmed elimination of cells by caspase-independent cell extrusion in C. elegans.

@ast

Programmed elimination of cells by caspase-independent cell extrusion in C. elegans.

@en

type

label

Programmed elimination of cells by caspase-independent cell extrusion in C. elegans.

@ast

Programmed elimination of cells by caspase-independent cell extrusion in C. elegans.

@en

prefLabel

Programmed elimination of cells by caspase-independent cell extrusion in C. elegans.

@ast

Programmed elimination of cells by caspase-independent cell extrusion in C. elegans.

@en

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Programmed elimination of cells by caspase-independent cell extrusion in C. elegans.

@en

P2093

Daniel P Denning

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Victoria Hatch

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P2860

MO25alpha/beta interact with STRADalpha/beta enhancing their ability to bind, activate and localize LKB1 in the cytoplasm

LKB1 is a master kinase that activates 13 kinases of the AMPK subfamily, including MARK/PAR-1

Structure of the LKB1-STRAD-MO25 complex reveals an allosteric mechanism of kinase activation

The genetics of Caenorhabditis elegans

A serine/threonine kinase gene defective in Peutz-Jeghers syndrome

The embryonic cell lineage of the nematode Caenorhabditis elegans

Toward improving Caenorhabditis elegans phenome mapping with an ORFeome-based RNAi library

Post-embryonic cell lineages of the nematode, Caenorhabditis elegans

Functional genomic analysis of C. elegans chromosome I by systematic RNA interference

The LKB1-AMPK pathway: metabolism and growth control in tumour suppression

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226-230

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scholarly article

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10.1038/NATURE11240

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7410

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488

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H. Robert Horvitz

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2012-08-01T00:00:00Z

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229155674

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1047562121

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22801495

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positive regulation of apoptotic process involved in development

regulation of apoptotic process involved in development

negative regulation of apoptotic process involved in development

Caenorhabditis elegans

P932

3416925

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