Reactive oxygen species amplify glucose signalling in renal cells cultured under high glucose and in diabetic kidney.
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Drug Targets for Oxidative Podocyte Injury in Diabetic NephropathyTargeting Mitochondria and Reactive Oxygen Species-Driven Pathogenesis in Diabetic NephropathyEpithelial-mesenchymal transition: An emerging target in tissue fibrosisOxidative stress and glutathione in TGF-beta-mediated fibrogenesisImprovement of renal oxidative stress markers after ozone administration in diabetic nephropathy in ratsHigh glucose stimulates TNFα and MCP-1 expression in rat microglia via ROS and NF-κB pathways.Accumulation of long-chain glycosphingolipids during aging is prevented by caloric restriction.Decrease of Klotho in the kidney of streptozotocin-induced diabetic ratsTransgenic overexpression of GLUT1 in mouse glomeruli produces renal disease resembling diabetic glomerulosclerosis.Urinary excretion of neutrophil gelatinase-associated lipocalin in diabetic rats.p66Shc mediates high-glucose and angiotensin II-induced oxidative stress renal tubular injury via mitochondrial-dependent apoptotic pathway.Astragaloside IV attenuates glycated albumin-induced epithelial-to-mesenchymal transition by inhibiting oxidative stress in renal proximal tubular cells.Hyperlipidemia-associated renal damage decreases Klotho expression in kidneys from ApoE knockout miceRole of Bone Morphogenetic Proteins-7 (BMP-7) in the Renal Improvement Effect of DangGui (Angelica sinensis) in Type-1 Diabetic RatsMultipotent mesenchymal stromal cells increase tPA expression and concomitantly decrease PAI-1 expression in astrocytes through the sonic hedgehog signaling pathway after stroke (in vitro study).Renal protective effect of xiao-chai-hu-tang on diabetic nephropathy of type 1-diabetic mice.Inflammatory cytokines as predictive markers for early detection and progression of diabetic nephropathyCell signaling through protein kinase C oxidation and activation.Sodium-glucose cotransporter inhibition prevents oxidative stress in the kidney of diabetic rats.Protein kinase C activation and its role in kidney disease.Lack of p47(phox) in Akita Diabetic Mice Is Associated with Interstitial Pneumonia, Fibrosis, and Oral Inflammation.Antioxidant Enzymes and Lipid Peroxidation in Type 2 Diabetes Mellitus Patients with and without Nephropathy.The Protective Effect of Beraprost Sodium on Diabetic Nephropathy by Inhibiting Inflammation and p38 MAPK Signaling Pathway in High-Fat Diet/Streptozotocin-Induced Diabetic Rats.A glimpse of various pathogenetic mechanisms of diabetic nephropathy.High glucose-induced oxidative stress increases the copy number of mitochondrial DNA in human mesangial cells.Nicotinamide adenine dinucleotide phosphate oxidase and diabetes: vascular implications.Identification of urinary proteins potentially associated with diabetic kidney disease.Mitochondrial dysfunction and mitophagy: the beginning and end to diabetic nephropathy?Over-production of nitric oxide by oxidative stress-induced activation of the TGF-β1/PI3K/Akt pathway in mesangial cells cultured in high glucoseCell signaling and receptors in toxicity of advanced glycation end products (AGEs): α-dicarbonyls, radicals, oxidative stress and antioxidants.High glucose enhances MMP-2 production in adventitial fibroblasts via Akt1-dependent NF-kappaB pathway.New developments concerning the proximal tubule in diabetic nephropathy: in vitro models and mechanisms.High Glucose-Induced Hypomethylation Promotes Binding of Sp-1 to Myo-Inositol Oxygenase: Implication in the Pathobiology of Diabetic Tubulopathy.Berberine ameliorates renal interstitial fibrosis induced by unilateral ureteral obstruction in rats.Effect of Ziziphus jujube Fruit Infusion on Lipid Profiles, Glycaemic Index and Antioxidant Status in Type 2 Diabetic Patients: A Randomized Controlled Clinical Trial.Myofibroblasts and their resistance to apoptosis: a possible mechanism of osteoradionecrosis.Chronic intermittent hypoxia induces local inflammation of the rat carotid body via functional upregulation of proinflammatory cytokine pathwaysScavenging of reactive oxygen species by astaxanthin inhibits epithelial-mesenchymal transition in high glucose-stimulated mesothelial cells.Gymnemasylvestre derived compounds inhibit GSH depletion and increase cGMP and nitric oxide to attenuate advanced glycation end products induced hypertrophic growth in renal tubular epithelial cells.Body weight control by a high-carbohydrate/low-fat diet slows the progression of diabetic kidney damage in an obese, hypertensive, type 2 diabetic rat model.
P2860
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P2860
Reactive oxygen species amplify glucose signalling in renal cells cultured under high glucose and in diabetic kidney.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Reactive oxygen species amplif ...... lucose and in diabetic kidney.
@ast
Reactive oxygen species amplif ...... lucose and in diabetic kidney.
@en
type
label
Reactive oxygen species amplif ...... lucose and in diabetic kidney.
@ast
Reactive oxygen species amplif ...... lucose and in diabetic kidney.
@en
prefLabel
Reactive oxygen species amplif ...... lucose and in diabetic kidney.
@ast
Reactive oxygen species amplif ...... lucose and in diabetic kidney.
@en
P2860
P1433
P1476
Reactive oxygen species amplif ...... glucose and in diabetic kidney
@en
P2093
Hi Bahl Lee
P2860
P356
10.1111/J.1440-1797.2005.00448.X
P478
P50
P577
2005-10-01T00:00:00Z