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Contribution of direct and indirect recognition pathways to T cell alloreactivity.

Contribution of direct and indirect recognition pathways to T cell alloreactivity.

http://www.wikidata.org/entity/Q36361819

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Antigenicity and immunogenicity of allogeneic retinal transplants NK cell patrolling and elimination of donor-derived dendritic cells favor indirect alloreactivity Measurement of T Cell Alloreactivity Using Imaging Flow Cytometry Allospecific rejection of MHC class I-deficient bone marrow by CD8 T cells.Donor MHC and adhesion molecules in transplant arteriosclerosis.Restricted and conserved T-cell repertoires involved in allorecognition of class II major histocompatibility complex.Chronic allograft failure in human renal transplantation: a multivariate risk factor analysis.Immunosuppression for lung transplantation.Indirect recognition of allopeptides promotes the development of cardiac allograft vasculopathy Immunology and immunomodulation of corneal transplantation.Alloreactivity across HLA barriers is mediated by both naïve and antigen-experienced T cells.The potential of antibody-based immunosuppressive agents for corneal transplantation.CD4 T cell-mediated cardiac allograft rejection requires donor but not host MHC class II.A review of research endeavors to optimize peripheral nerve reconstruction.Exosomal miR-142-3p is increased during cardiac allograft rejection and augments vascular permeability through down-regulation of endothelial RAB11FIP2 expression.Phagocytosis of antigens by Langerhans cells in vitro.Hybrid antibody mediated veto of cytotoxic T lymphocyte responses.From current immunosuppressive strategies to clinical tolerance of allografts.Alloreactive CD8 T cell tolerance requires recipient B cells, dendritic cells, and MHC class II.In vitro assays of allosensitization.T cell receptor biases and clonal proliferations among lung transplant recipients with obliterative bronchiolitis.Indirect recognition of donor HLA-DR peptides in organ allograft rejection.T cells reactive to a single immunodominant self-restricted allopeptide induce skin graft rejection in mice.CD4(+) T cells tolerized ex vivo to host alloantigen by anti-CD40 ligand (CD40L:CD154) antibody lose their graft-versus-host disease lethality capacity but retain nominal antigen responses.Dendritic cells stimulated with Actinobacillus actinomycetemcomitans elicit rapid gamma interferon responses by natural killer cells.Immune recognition and rejection of allogeneic skin grafts.Indirect recognition of MHC class I allopeptides accelerates lung allograft rejection in miniature swine.A Paradigm Shift on the Question of B Cells in Transplantation? Recent Insights on Regulating the Alloresponse.The role of MHC genes in contagious cancer: the story of Tasmanian devils.Blood transfusion, blood storage and immunomodulation.Induction of immunologic tolerance for transplantation.Overexpression of human homologs of the bacterial DnaJ chaperone in the synovial tissue of patients with rheumatoid arthritis.Mechanisms of oral tolerance by MHC peptides.New approaches to specific immunomodulation in transplantation.Role of indirect allorecognition in allograft rejection.Indirect T cell recognition in allograft rejection.The role of endogenous peptides in the direct pathway of alloreactivity to human MHC class II molecules expressed on CHO cells.Suppression of the indirect pathway of T cell reactivity by high doses of allopeptide.Cross-tolerance of recipient-derived transforming growth factor-beta dendritic cells.The differences between late graft loss group and long-term graft survival group in renal transplantation.

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P2860

Contribution of direct and indirect recognition pathways to T cell alloreactivity.

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1993 nî lūn-bûn

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1993年學術文章

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Contribution of direct and indirect recognition pathways to T cell alloreactivity.

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Contribution of direct and indirect recognition pathways to T cell alloreactivity.

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Contribution of direct and indirect recognition pathways to T cell alloreactivity.

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Contribution of direct and indirect recognition pathways to T cell alloreactivity.

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Contribution of direct and indirect recognition pathways to T cell alloreactivity.

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Contribution of direct and indirect recognition pathways to T cell alloreactivity.

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Contribution of direct and indirect recognition pathways to T cell alloreactivity.

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P2860

Allelic variation in the DR subregion of the human major histocompatibility complex

Allorecognition of DR1 by T cells from a DR4/DRw13 responder mimics self-restricted recognition of endogenous peptides.

Processed MHC class I alloantigen as the stimulus for CD4+ T-cell dependent antibody-mediated graft rejection.

Donor major histocompatibility complex (MHC) peptides are presented by recipient MHC molecules during graft rejection.

T cell recognition of self-human histocompatibility leukocyte antigens (HLA)-DR peptides in context of syngeneic HLA-DR molecules

Phenotype, specificity, and function of T cell subsets and T cell interactions involved in skin allograft rejection.

Cytotoxic T cell recognition of an endogenous class I HLA peptide presented by a class II HLA molecule

Immunogenicity and tolerogenicity of self-major histocompatibility complex peptides.

Effector cells in allelic H-2 class I-incompatible skin graft rejection

Cellular basis of skin allograft rejection across a class I major histocompatibility barrier in mice depleted of CD8+ T cells in vivo

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