The role of reactive oxygen species in capsaicin-induced mechanical hyperalgesia and in the activities of dorsal horn neurons
about
Effects of tempol and redox-cycling nitroxides in models of oxidative stress.Capsaicin: Current Understanding of Its Mechanisms and Therapy of Pain and Other Pre-Clinical and Clinical UsesReactive Oxygen Species: Physiological and Physiopathological Effects on Synaptic PlasticityInvolvement of reactive oxygen species in long-term potentiation in the spinal cord dorsal horn.Molecular hydrogen attenuates neuropathic pain in mice.Neuronal P2X7 receptor-induced reactive oxygen species production contributes to nociceptive behavior in miceSupraspinal inactivation of mitochondrial superoxide dismutase is a source of peroxynitrite in the development of morphine antinociceptive tolerance.Reactive oxygen species are involved in group I mGluR-mediated facilitation of nociceptive processing in amygdala neurons.Global inhibition of reactive oxygen species (ROS) inhibits paclitaxel-induced painful peripheral neuropathy.Spinal NADPH oxidase is a source of superoxide in the development of morphine-induced hyperalgesia and antinociceptive toleranceAdditive antinociceptive effects of a combination of vitamin C and vitamin E after peripheral nerve injury.Involvement of spinal orexin A in the electroacupuncture analgesia in a rat model of post-laparotomy pain.Mitochondrial reactive oxygen species are activated by mGluR5 through IP3 and activate ERK and PKA to increase excitability of amygdala neurons and pain behavior.Mitochondrial Ca(2+) uptake is essential for synaptic plasticity in pain.Reactive oxygen species mediate visceral pain-related amygdala plasticity and behaviors.The effect of phosphodiesterase-4-specific inhibitor in the rat model of spinal nerve ligation.Tempol modulates changes in xenobiotic permeability and occludin oligomeric assemblies at the blood-brain barrier during inflammatory painAlcoholic neuropathy: possible mechanisms and future treatment possibilitiesD-Amino acid oxidase-mediated increase in spinal hydrogen peroxide is mainly responsible for formalin-induced tonic pain.Reactive Oxygen Species Donors Increase the Responsiveness of Dorsal Horn Neurons and Induce Mechanical Hyperalgesia in Rats.Mechanisms of G protein-coupled estrogen receptor-mediated spinal nociception.A Novel Nitronyl Nitroxide with Salicylic Acid Framework Attenuates Pain Hypersensitivity and Ectopic Neuronal Discharges in Radicular Low Back Pain.Unravelling the mystery of capsaicin: a tool to understand and treat painAntinociceptive effects of intraperitoneal and intrathecal vitamin e in the rat formalin testAntinociceptive drug interaction between intrathecal vitamin E and gabapentin in the rat formalin testProoxidant-induced c-Src/nuclear factor kappa B-coupled signalling in sensory ganglia mediates cutaneous hyperalgesia.HSV vector-mediated GAD67 suppresses neuropathic pain induced by perineural HIV gp120 in rats through inhibition of ROS and Wnt5aWingless-type mammary tumor virus integration site family, member 5A (Wnt5a) regulates human immunodeficiency virus type 1 (HIV-1) envelope glycoprotein 120 (gp120)-induced expression of pro-inflammatory cytokines via the Ca2+/calmodulin-dependent pOxidative stress in the spinal cord is an important contributor in capsaicin-induced mechanical secondary hyperalgesia in mice.Lipophilicity is a critical parameter that dominates the efficacy of metalloporphyrins in blocking the development of morphine antinociceptive tolerance through peroxynitrite-mediated pathways.Effect of antioxidant treatment on spinal GABA neurons in a neuropathic pain model in the mouseIncreased production of mitochondrial superoxide in the spinal cord induces pain behaviors in mice: the effect of mitochondrial electron transport complex inhibitorsPeroxynitrite and opiate antinociceptive tolerance: a painful reality.Activation of the nuclear factor E2-related factor 2/anitioxidant response element alleviates the nitroglycerin-induced hyperalgesia in rats.Persistent pain is dependent on spinal mitochondrial antioxidant levelsElectroacupuncture suppresses capsaicin-induced secondary hyperalgesia through an endogenous spinal opioid mechanismReactive nitroxidative species and nociceptive processing: determining the roles for nitric oxide, superoxide, and peroxynitrite in pain.Roles of reactive oxygen and nitrogen species in painSustained TRPA1 activation in vivo.The role of mitochondrial dysfunctions due to oxidative and nitrosative stress in the chronic pain or chronic fatigue syndromes and fibromyalgia patients: peripheral and central mechanisms as therapeutic targets?
P2860
Q24595411-DF483746-ED98-46EA-90BD-906D05E80D9DQ26745591-D210DB69-D6F1-413A-9855-D970FF1D8C18Q28070007-99381563-2A76-4BD3-B6B8-8CB22AFC04A2Q33593550-F34D84C0-9FA4-40CE-8523-22EDD32964A4Q33774014-10C048E6-1492-4A9C-915F-9E99A6FF4739Q33799452-1CA0D55E-0CF5-4096-A0AB-865F66218636Q33813707-FD6386D7-4F2E-4917-9EC0-A20DAC4E28CBQ33995347-7884A1F7-6E62-4CDB-88E1-64799C67EE1DQ34038073-62AAB1A5-0549-491C-8BE3-218805BE2270Q34103705-6CCB7F31-5706-4CC9-B50A-0D17A164E732Q34110860-F4ABBBA1-13E9-4DCD-9BDF-6D7533E35D42Q34485538-A75AD1D8-00D0-4298-B440-20A1A5B9DC6FQ34776360-A5017E09-33A2-4821-82E7-2BDE96FD6229Q35229769-63680E56-5CE7-4B2F-8841-0BF36F3BA0C9Q35472078-4DA412F1-EE37-4E7F-AA76-0D183451ACE8Q35502836-C9F8C34D-B34A-4150-9057-7C72AD2BE24CQ35964989-11B46945-1530-48DB-AEF1-4DDB6CCA2DEFQ36017359-EF0844F3-9427-4B2A-848E-2DACF15E36C7Q36025408-47C1CEA8-0BEB-4FE9-800F-D9238D8AF203Q36118999-C30CD4C6-AC13-4E44-B2F8-5151B4746143Q36140716-04C04AFE-DE07-415A-993A-BFDF92CE760DQ36279182-E1741385-BBEA-465E-9C15-53E1120C97EEQ36293653-D8E67EE9-6A98-4425-811F-5B73776B0FFCQ36310253-853C8DEC-D561-40FD-AFCE-E7985137554CQ36424572-9B9D81AD-41AD-4A5A-9230-A0DEC5C64E23Q36751546-0E070CF1-F966-4B79-96DB-56BFF0CA63B9Q36777541-68BFFCA7-4014-47AB-AB3A-00CBA168FAB4Q36832652-F18DB548-6C60-400B-A35C-058AF67C5D68Q36968217-0B740189-6050-4FC1-AB16-4B874EA8703CQ37131042-CDDA0E74-46C9-4275-9631-3C90CC3717E4Q37276780-83F1D0CD-1CB0-4C31-A043-618F3DE5F708Q37292473-D23B3B6B-7F2B-4AFF-81BA-840145842834Q37328701-3CC68092-3542-43D3-A678-726C21F2C5A5Q37362988-99D48442-A7D3-4780-B83E-530CA07D5D5CQ37375145-0BCC0E54-2F8F-4481-8997-30BBE1F24E9BQ37388123-D0E7EAC3-4221-4C19-8288-AB37C6CFF98DQ37765512-B6AACFC4-F25D-4590-88BA-D6DC143A784AQ37833285-2907F131-2F31-4068-A44C-5CC7135FE053Q37868764-A764BABC-274C-469D-B66D-D04BBD6A9B94Q38120080-F90062B0-B520-4C12-A2F8-3DDF073EE763
P2860
The role of reactive oxygen species in capsaicin-induced mechanical hyperalgesia and in the activities of dorsal horn neurons
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
The role of reactive oxygen sp ...... ivities of dorsal horn neurons
@ast
The role of reactive oxygen sp ...... ivities of dorsal horn neurons
@en
type
label
The role of reactive oxygen sp ...... ivities of dorsal horn neurons
@ast
The role of reactive oxygen sp ...... ivities of dorsal horn neurons
@en
prefLabel
The role of reactive oxygen sp ...... ivities of dorsal horn neurons
@ast
The role of reactive oxygen sp ...... ivities of dorsal horn neurons
@en
P2093
P2860
P1433
P1476
The role of reactive oxygen sp ...... ivities of dorsal horn neurons
@en
P2093
Hee Kee Kim
Inhyung Lee
Jae Hyo Kim
Jin Mo Chung
Kyungsoon Chung
P2860
P356
10.1016/J.PAIN.2007.01.035
P407
P577
2007-03-26T00:00:00Z