ADAMTS13 deficiency exacerbates VWF-dependent acute myocardial ischemia/reperfusion injury in mice
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Animal models for thrombotic thrombocytopenic purpuraPlasma ADAMTS-13 levels and the risk of myocardial infarction: an individual patient data meta-analysis.Multiple major morbidities and increased mortality during long-term follow-up after recovery from thrombotic thrombocytopenic purpura.Regulation of VWF expression, and secretion in health and disease.ADAMTS13 controls vascular remodeling by modifying VWF reactivity during stroke recovery.Protective vascular and cardiac effects of inducible nitric oxide synthase in mice with hyperhomocysteinemia.The Fifth Domain of Beta 2 Glycoprotein I Protects from Natural IgM Mediated Cardiac Ischaemia Reperfusion InjuryEmerging Roles of ADAMTSs in Angiogenesis and Cancer.Structure-function and regulation of ADAMTS-13 protease.Gene silencing of endothelial von Willebrand Factor attenuates angiotensin II-induced endothelin-1 expression in porcine aortic endothelial cells.The role of ADAMTS13 in acute myocardial infarction: cause or consequence?ADAMTS13 predicts renal and cardiovascular events in type 2 diabetic patients and response to therapy.von Willebrand factor contributes to poor outcome in a mouse model of intracerebral haemorrhage.VWF-mediated leukocyte recruitment with chromatin decondensation by PAD4 increases myocardial ischemia/reperfusion injury in mice.Evaluation of US 2016/0115161 A1: isoindoline compounds and methods of their use.The Role of von Willebrand Factor in Vascular Inflammation: From Pathogenesis to Targeted Therapy.von Willebrand factor and inflammation.The ADAMTS13-von Willebrand factor axis is involved in the pathophysiology of kidney ischemia-reperfusion injury.Low ADAMTS-13 activity and the risk of coronary heart disease - a prospective cohort study: the Rotterdam Study.Endothelial Cell-Derived von Willebrand Factor Is the Major Determinant That Mediates von Willebrand Factor-Dependent Acute Ischemic Stroke by Promoting Postischemic Thrombo-Inflammation.DNase, ADAMTS13, and iPAD4: good for the heart.Absence of mutations in cereblon (CRBN) and DNA damage-binding protein 1 (DDB1) genes and significance for IMiD therapyEndothelial Cell-Derived Von Willebrand Factor, But Not Platelet-Derived, Promotes Atherosclerosis in Apolipoprotein E-Deficient Mice.New insights into the non-hemostatic role of von Willebrand factor in endothelial protection.Recombinant Human ADAMTS13 Treatment Improves Myocardial Remodeling and Functionality After Pressure Overload Injury in Mice.ADAMTS-13 and von Willebrand factor: a dynamic duo.ADAMTS13 modulates atherosclerotic plaque progression in mice via a VWF-dependent mechanism.ADAMTS13 Retards Progression of Diabetic Nephropathy by Inhibiting Intrarenal Thrombosis in Mice.TTP-like syndrome: novel concept and molecular pathogenesis of endotheliopathy-associated vascular microthrombotic disease
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P2860
ADAMTS13 deficiency exacerbates VWF-dependent acute myocardial ischemia/reperfusion injury in mice
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2012 nî lūn-bûn
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2012年の論文
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2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
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2012年论文
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name
ADAMTS13 deficiency exacerbate ...... mia/reperfusion injury in mice
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ADAMTS13 deficiency exacerbate ...... mia/reperfusion injury in mice
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type
label
ADAMTS13 deficiency exacerbate ...... mia/reperfusion injury in mice
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ADAMTS13 deficiency exacerbate ...... mia/reperfusion injury in mice
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prefLabel
ADAMTS13 deficiency exacerbate ...... mia/reperfusion injury in mice
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ADAMTS13 deficiency exacerbate ...... mia/reperfusion injury in mice
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P2093
P2860
P1433
P1476
ADAMTS13 deficiency exacerbate ...... mia/reperfusion injury in mice
@en
P2093
Anil K Chauhan
Chintan Gandhi
David G Motto
Melissa Jensen
Steven R Lentz
P2860
P304
P356
10.1182/BLOOD-2012-06-440255
P407
P577
2012-09-14T00:00:00Z