Neutrophils in development of multiple organ failure in sepsis.
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Paradoxical Roles of the Neutrophil in Sepsis: Protective and DeleteriousNeutrophils in type 1 diabetesBalancing Innate Immunity and Inflammatory State via Modulation of Neutrophil Function: A Novel Strategy to Fight SepsisLiver dysfunction and phosphatidylinositol-3-kinase signalling in early sepsis: experimental studies in rodent models of peritonitisA Neutrophil Phenotype Model for Extracorporeal Treatment of SepsisCapillary plexuses are vulnerable to neutrophil extracellular trapsInflection points in sepsis biology: from local defense to systemic organ injuryNociceptin system as a target in sepsis?From neutrophil extracellular traps release to thrombosis: an overshooting host-defense mechanism?A novel inflammatory pathway involved in leukocyte recruitment: role for the kinin B1 receptor and the chemokine CXCL5Modulation of antigen processing by haem-oxygenase 1. Implications on inflammation and toleranceAerosol exposure to Rift Valley fever virus causes earlier and more severe neuropathology in the murine model, which has important implications for therapeutic developmentA biomimetic membrane device that modulates the excessive inflammatory response to sepsisAcute loss of renal function attenuates slow leukocyte rolling and transmigration by interfering with intracellular signalingAffinity flow fractionation of cells via transient interactions with asymmetric molecular patterns.The small GTPase Rap1b negatively regulates neutrophil chemotaxis and transcellular diapedesis by inhibiting Akt activation.ATP-gated P2X1 ion channels protect against endotoxemia by dampening neutrophil activation.Septic shock and chemotherapy-induced cytopenia: effects on microcirculation.Contribution of protein Z and protein Z-dependent protease inhibitor in generalized Shwartzman reaction.Endogenous morphine levels are increased in sepsis: a partial implication of neutrophils.Blockade of Neutrophil's Chemokine Receptors CXCR1/2 Abrogate Liver Damage in Acute-on-Chronic Liver Failure.Ectodomain Shedding by ADAM17: Its Role in Neutrophil Recruitment and the Impairment of This Process during SepsisBlockade of ICAM-1 improves the outcome of polymicrobial sepsis via modulating neutrophil migration and reversing immunosuppression.IVIG regulates the survival of human but not mouse neutrophils.Neutrophils from critically ill septic patients mediate profound loss of endothelial barrier integrityCirculating neutrophil counts and mortality in septic shock.Extracorporeal immune therapy with immobilized agonistic anti-Fas antibodies leads to transient reduction of circulating neutrophil numbers and limits tissue damage after hemorrhagic shock/resuscitation in a porcine modelThe SaeR/S gene regulatory system induces a pro-inflammatory cytokine response during Staphylococcus aureus infection.Ensemble models of neutrophil trafficking in severe sepsis.Baicalin improves survival in a murine model of polymicrobial sepsis via suppressing inflammatory response and lymphocyte apoptosis.Thaliporphine preserves cardiac function of endotoxemic rabbits by both directly and indirectly attenuating NFκB signaling pathway.MMP-8 deficiency increases TLR/RAGE ligands S100A8 and S100A9 and exacerbates lung inflammation during endotoxemia.The effects of a selective inhibitor of c-Fos/activator protein-1 on endotoxin-induced acute kidney injury in mice.Modulation of chemokine gradients by apheresis redirects leukocyte trafficking to different compartments during sepsis, studies in a rat model.Neutrophil IL-10 suppresses peritoneal inflammatory monocytes during polymicrobial sepsis.Sepsis lethality via exacerbated tissue infiltration and TLR-induced cytokine production by neutrophils is integrin α3β1-dependent.Neutrophils with myeloid derived suppressor function deplete arginine and constrain T cell function in septic shock patients.Citronellal, a monoterpene present in Java citronella oil, attenuates mechanical nociception response in mice.Sepsis induced by Staphylococcus aureus: participation of biomarkers in a murine model.Excessive neutrophils and neutrophil extracellular traps contribute to acute lung injury of influenza pneumonitis.
P2860
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P2860
Neutrophils in development of multiple organ failure in sepsis.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Neutrophils in development of multiple organ failure in sepsis.
@ast
Neutrophils in development of multiple organ failure in sepsis.
@en
type
label
Neutrophils in development of multiple organ failure in sepsis.
@ast
Neutrophils in development of multiple organ failure in sepsis.
@en
prefLabel
Neutrophils in development of multiple organ failure in sepsis.
@ast
Neutrophils in development of multiple organ failure in sepsis.
@en
P2093
P1433
P1476
Neutrophils in development of multiple organ failure in sepsis.
@en
P2093
D F Treacher
J D Edgeworth
J D Pearson
P304
P356
10.1016/S0140-6736(06)69005-3
P407
P577
2006-07-01T00:00:00Z