BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases.
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Zerumbone induced apoptosis in liver cancer cells via modulation of Bax/Bcl-2 ratioAlternatively spliced products CC3 and TC3 have opposing effects on apoptosisBax is increased in the retina of diabetic subjects and is associated with pericyte apoptosis in vivo and in vitroTAJ, a novel member of the tumor necrosis factor receptor family, activates the c-Jun N-terminal kinase pathway and mediates caspase-independent cell deathBNIP3 and genetic control of necrosis-like cell death through the mitochondrial permeability transition poreThe p53-inducible E3 ubiquitin ligase p53RFP induces p53-dependent apoptosisHSpin1, a transmembrane protein interacting with Bcl-2/Bcl-xL, induces a caspase-independent autophagic cell deathGlutathione peroxidase-1 protects from CD95-induced apoptosisCIDE, a novel family of cell death activators with homology to the 45 kDa subunit of the DNA fragmentation factorThe novel presenilin-1-associated protein is a proapoptotic mitochondrial proteinApoptosis, pyroptosis, and necrosis: mechanistic description of dead and dying eukaryotic cells.Caspases: the executioners of apoptosisBim: a novel member of the Bcl-2 family that promotes apoptosisEnforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosisProapoptotic Bid mediates the Atr-directed DNA damage response to replicative stressCaspases are activated in a branched protease cascade and control distinct downstream processes in Fas-induced apoptosisCell damage-induced conformational changes of the pro-apoptotic protein Bak in vivo precede the onset of apoptosisDeath by a B cell superantigen: In vivo VH-targeted apoptotic supraclonal B cell deletion by a Staphylococcal ToxinDAP kinase and DRP-1 mediate membrane blebbing and the formation of autophagic vesicles during programmed cell deathMitochondrial release of caspase-2 and -9 during the apoptotic processProapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activityEquine estrogens differentially inhibit DNA fragmentation induced by glutamate in neuronal cells by modulation of regulatory proteins involved in programmed cell deathGene therapy for carcinoma of the breast: Pro-apoptotic gene therapyCrystal structure of rat Bcl-xL. Implications for the function of the Bcl-2 protein familyThe proteasomal substrate Stm1 participates in apoptosis-like cell death in yeast.An ARL1 mutation affected autophagic cell death in yeast, causing a defect in central vacuole formation.The ectodermal dysplasia receptor activates the nuclear factor-kappaB, JNK, and cell death pathways and binds to ectodysplasin ACD47 ligation induces caspase-independent cell death in chronic lymphocytic leukemiaStimulation of Kv1.3 potassium channels by death receptors during apoptosis in Jurkat T lymphocytesEssential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cellsMitochondrial hyperpolarization and ATP depletion in patients with systemic lupus erythematosusInduction of caspase-independent programmed cell death by vitamin E natural homologs and synthetic derivativesPhosphorylation of PITSLRE p110 isoforms accompanies their processing by caspases during Fas-mediated cell deathUrsodeoxycholic acid may inhibit deoxycholic acid-induced apoptosis by modulating mitochondrial transmembrane potential and reactive oxygen species production2,2'-diphenyl-3,3'-diindolylmethane: a potent compound induces apoptosis in breast cancer cells by inhibiting EGFR pathwayMtd, a novel Bcl-2 family member activates apoptosis in the absence of heterodimerization with Bcl-2 and Bcl-XLDeacetylation of p53 induces autophagy by suppressing Bmf expressionThe anti-cancer activities of Vernonia amygdalina extract in human breast cancer cell lines are mediated through caspase-dependent and p53-independent pathwaysMonobenzyltin Complex C1 Induces Apoptosis in MCF-7 Breast Cancer Cells through the Intrinsic Signaling Pathway and through the Targeting of MCF-7-Derived Breast Cancer Stem Cells via the Wnt/β-Catenin Signaling PathwayBax and Bcl-2 interaction in a transgenic mouse model of familial amyotrophic lateral sclerosis
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P2860
BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 1996
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
BAX-induced cell death may not ...... verting enzyme-like proteases.
@en
BAX-induced cell death may not ...... verting enzyme-like proteases.
@nl
type
label
BAX-induced cell death may not ...... verting enzyme-like proteases.
@en
BAX-induced cell death may not ...... verting enzyme-like proteases.
@nl
prefLabel
BAX-induced cell death may not ...... verting enzyme-like proteases.
@en
BAX-induced cell death may not ...... verting enzyme-like proteases.
@nl
P2093
P2860
P356
P1476
BAX-induced cell death may not ...... verting enzyme-like proteases.
@en
P2093
P2860
P304
14559-14563
P356
10.1073/PNAS.93.25.14559
P407
P577
1996-12-01T00:00:00Z