Dendritic cell and NK cell reciprocal cross talk promotes gamma interferon-dependent immunity to blood-stage Plasmodium chabaudi AS infection in mice.
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Does EBV alter the pathogenesis of malaria?In vivo approaches reveal a key role for DCs in CD4+ T cell activation and parasite clearance during the acute phase of experimental blood-stage malariaThe contribution of Plasmodium chabaudi to our understanding of malariaThe role of the spleen in malaria.Listeria monocytogenes infection affects a subset of Ly49-expressing NK cells in the ratComparative analysis of activation phenotype, proliferation, and IFN-gamma production by spleen NK1.1(+) and NK1.1(-) T cells during Plasmodium chabaudi AS malaria.The spleen CD4+ T cell response to blood-stage Plasmodium chabaudi malaria develops in two phases characterized by different propertiesIL-18, but not IL-15, contributes to the IL-12-dependent induction of NK-cell effector functions by Leishmania infantum in vivo.Activation of naive NK cells in response to Listeria monocytogenes requires IL-18 and contact with infected dendritic cellsGammadelta T cells but not NK cells are essential for cell-mediated immunity against Plasmodium chabaudi malaria.Gene-gene interaction and functional impact of polymorphisms on innate immune genes in controlling Plasmodium falciparum blood infection level.The contribution of natural killer complex loci to the development of experimental cerebral malaria.Regulating the adaptive immune response to blood-stage malaria: role of dendritic cells and CD4⁺Foxp3⁺ regulatory T cells.Co-expression of Interleukin-15 Enhances the Protective Immune Responses Induced by Immunization with a Murine Malaria MVA-Based Vaccine Encoding the Circumsporozoite Protein.Colpodella spp.-like parasite infection in woman, China.Plasmodium yoelii blood-stage primes macrophage-mediated innate immune response through modulation of toll-like receptor signalling.Interferon regulatory factor modulation underlies the bystander suppression of malaria antigen-driven IL-12 and IFN-γ in filaria-malaria co-infection.Allicin enhances host pro-inflammatory immune responses and protects against acute murine malaria infection.Roles of IFN-γ and γδ T Cells in Protective Immunity Against Blood-Stage MalariaIL-18-induced expression of high-affinity IL-2R on murine NK cells is essential for NK-cell IFN-γ production during murine Plasmodium yoelii infection.Pre-existing Schistosoma japonicum infection alters the immune response to Plasmodium berghei infection in C57BL/6 mice.NK Cells: Uncertain Allies against Malaria.Flow cytometric detection of p38 MAPK phosphorylation and intracellular cytokine expression in peripheral blood subpopulations from patients with autoimmune rheumatic diseases.NK cells modulate the lung dendritic cell-mediated Th1/Th17 immunity during intracellular bacterial infection.Natural killer cell activation by dendritic cells: balancing inhibitory and activating signals.Activation of natural killer cells during microbial infectionsNK-DC Crosstalk in Immunity to Microbial InfectionSmall molecule-based inhibition of MEK1/2 proteins dampens inflammatory responses to malaria, reduces parasite load, and mitigates pathogenic outcomes.CD36 receptor regulates malaria-induced immune responses primarily at early blood stage infection contributing to parasitemia control and resistance to mortality.Escherichia coli Nissle 1917 protects gnotobiotic pigs against human rotavirus by modulating pDC and NK-cell responses.Bacillus Calmette-Guérin-inoculation at different time points influences the outcome of C57BL/6 mice infected with Plasmodium chabaudi chabaudi AS.Mesenchymal Stromal Cells Prevent Allostimulation In Vivo and Control Checkpoints of Th1 Priming: Migration of Human DC to Lymph Nodes and NK Cell Activation.Activated NKT cells imprint NK-cell differentiation, functionality and education.Plasmodium attenuation: connecting the dots between early immune responses and malaria disease severityCD3⁻NK1.1⁺ cells aid in the early induction of a Th1 response to an attaching and effacing enteric pathogen.Mapping of Char10, a novel malaria susceptibility locus on mouse chromosome 9.Transient attenuated Foxp3 expression on CD4⁺ T cells treated with 7D4 mAb contributes to the control of parasite burden in DBA / 2 mice infected with lethal Plasmodium chabaudi chabaudi AS.Supplement of L-Arg improves protective immunity during early-stage Plasmodium yoelii 17XL infection.Vaccinating for natural killer cell effector functions.A malaria protein factor induces IL-4 production by dendritic cells via PI3K-Akt-NF-κB signaling independent of MyD88/TRIF and promotes Th2 response.
P2860
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P2860
Dendritic cell and NK cell reciprocal cross talk promotes gamma interferon-dependent immunity to blood-stage Plasmodium chabaudi AS infection in mice.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Dendritic cell and NK cell rec ...... chabaudi AS infection in mice.
@en
Dendritic cell and NK cell rec ...... chabaudi AS infection in mice.
@nl
type
label
Dendritic cell and NK cell rec ...... chabaudi AS infection in mice.
@en
Dendritic cell and NK cell rec ...... chabaudi AS infection in mice.
@nl
prefLabel
Dendritic cell and NK cell rec ...... chabaudi AS infection in mice.
@en
Dendritic cell and NK cell rec ...... chabaudi AS infection in mice.
@nl
P2860
P356
P1476
Dendritic cell and NK cell rec ...... chabaudi AS infection in mice.
@en
P2093
Rebecca Ing
P2860
P304
P356
10.1128/IAI.00994-08
P407
P577
2008-11-17T00:00:00Z