Variation in chronic nicotinamide treatment after traumatic brain injury can alter components of functional recovery independent of histological damage.
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Sensory cortex underpinnings of traumatic brain injury deficitsComparison of the effect of minocycline and simvastatin on functional recovery and gene expression in a rat traumatic brain injury model.Diabetes mellitus: channeling care through cellular discoveryCOG1410, an apolipoprotein E-based peptide, improves cognitive performance and reduces cortical loss following moderate fluid percussion injury in the rat.Pyridoxine administration improves behavioral and anatomical outcome after unilateral contusion injury in the rat.Novel avenues of drug discovery and biomarkers for diabetes mellitus.Preclinical efficacy testing in middle-aged rats: nicotinamide, a novel neuroprotectant, demonstrates diminished preclinical efficacy after controlled cortical impact.The effects of a high-fat sucrose diet on functional outcome following cortical contusion injury in the rat.A Combination Therapy of Nicotinamide and Progesterone Improves Functional Recovery following Traumatic Brain Injury.A behavioral and histological comparison of fluid percussion injury and controlled cortical impact injury to the rat sensorimotor cortexA comparison of the effects of nicotinamide and progesterone on functional recovery of cognitive behavior following cortical contusion injury in the rat.The effect of nicotinamide on gene expression in a traumatic brain injury modelVitamins and nutrients as primary treatments in experimental brain injury: Clinical implications for nutraceutical therapies.Comparison of the effects of erythropoietin and anakinra on functional recovery and gene expression in a traumatic brain injury model.The vitamin nicotinamide: translating nutrition into clinical care.The effects of hypertonic saline and nicotinamide on sensorimotor and cognitive function following cortical contusion injury in the rat.Nicotinamide pre-treatment ameliorates NAD(H) hyperoxidation and improves neuronal function after severe hypoxia.Continuous nicotinamide administration improves behavioral recovery and reduces lesion size following bilateral frontal controlled cortical impact injurySustained delivery of nicotinamide limits cortical injury and improves functional recovery following traumatic brain injury.
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P2860
Variation in chronic nicotinamide treatment after traumatic brain injury can alter components of functional recovery independent of histological damage.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on October 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Variation in chronic nicotinam ...... endent of histological damage.
@en
Variation in chronic nicotinam ...... endent of histological damage.
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type
label
Variation in chronic nicotinam ...... endent of histological damage.
@en
Variation in chronic nicotinam ...... endent of histological damage.
@nl
prefLabel
Variation in chronic nicotinam ...... endent of histological damage.
@en
Variation in chronic nicotinam ...... endent of histological damage.
@nl
P2093
P2860
P356
P1476
Variation in chronic nicotinam ...... endent of histological damage.
@en
P2093
Jason E Beare
Jeremy L Pierce
Michael R Hoane
Nicholas A Kaufman
P2860
P356
10.4161/OXIM.1.1.6694
P577
2008-10-01T00:00:00Z