FOG2 protein down-regulation by transforming growth factor-β1-induced microRNA-200b/c leads to Akt kinase activation and glomerular mesangial hypertrophy related to diabetic nephropathy. - Wikidata - wikimedia - TriplyDB

FOG2 protein down-regulation by transforming growth factor-β1-induced microRNA-200b/c leads to Akt kinase activation and glomerular mesangial hypertrophy related to diabetic nephropathy.

FOG2 protein down-regulation by transforming growth factor-β1-induced microRNA-200b/c leads to Akt kinase activation and glomerular mesangial hypertrophy related to diabetic nephropathy.

http://www.wikidata.org/entity/Q37311890

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MicroRNAs in Diabetic Kidney Disease The Role of ERK1/2 in the Development of Diabetic Cardiomyopathy Mini-review: emerging roles of microRNAs in the pathophysiology of renal diseases miR-200b inhibits CD133+ glioma cells by targeting the AKT pathway EGFR inhibition attenuates diabetic nephropathy through decreasing ROS and endoplasmic reticulum stress.The role of microRNAs in diabetic nephropathy.Transforming growth factor β1 (TGF-β1) enhances expression of profibrotic genes through a novel signaling cascade and microRNAs in renal mesangial cells Repression of let-7 by transforming growth factor-β1-induced Lin28 upregulates collagen expression in glomerular mesangial cells under diabetic conditions.The role of miR-200a in mammalian epithelial cell transformation.Threonine 32 (Thr32) of FoxO3 is critical for TGF-β-induced apoptosis via Bim in hepatocarcinoma cells.High glucose enhances microRNA-26a to activate mTORC1 for mesangial cell hypertrophy and matrix protein expression.MicroRNAs in diabetic nephropathy: functions, biomarkers, and therapeutic targets.MicroRNA-200 promotes lung cancer cell growth through FOG2-independent AKT activation.Histone Lysine Methylation in TGF-β1 Mediated p21 Gene Expression in Rat Mesangial Cells.Cross talk between miR-214 and PTEN attenuates glomerular hypertrophy under diabetic conditions.MicroRNAs as novel therapeutic targets to treat kidney injury and fibrosis.MicroRNA in diabetic nephropathy: renin angiotensin, aGE/RAGE, and oxidative stress pathway.Inhibition of the processing of miR-25 by HIPK2-Phosphorylated-MeCP2 induces NOX4 in early diabetic nephropathy.Diabetic nephropathy--emerging epigenetic mechanisms.MiR-196a Regulates High Glucose-Induced Mesangial Cell Hypertrophy by Targeting p27kip1.microRNAs in Diabetic Kidney Disease.miR-200c Accelerates Hepatic Stellate Cell-Induced Liver Fibrosis via Targeting the FOG2/PI3K Pathway.miR-200c: a versatile watchdog in cancer progression, EMT, and drug resistance.MiR-135a promotes renal fibrosis in diabetic nephropathy by regulating TRPC1.MicroRNAs as Master Regulators of Glomerular Function in Health and Disease.Epigenetics of kidney disease.Effects of xanthine oxidase inhibition with febuxostat on the development of nephropathy in experimental type 2 diabetes.Circulating miRNA Profiles Associated With Hyperglycemia in Patients With Type 1 Diabetes Mellitus.Therapeutic potential of microRNAs for the treatment of renal fibrosis and CKD.Therapeutic miR-21 Silencing Ameliorates Diabetic Kidney Disease in Mice.Reduced Autophagy by a microRNA-mediated Signaling Cascade in Diabetes-induced Renal Glomerular Hypertrophy.MicroRNA and Microvascular Complications of Diabetes.Noncoding RNAs as therapeutic targets in early stage diabetic kidney disease

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FOG2 protein down-regulation by transforming growth factor-β1-induced microRNA-200b/c leads to Akt kinase activation and glomerular mesangial hypertrophy related to diabetic nephropathy.

http://www.wikidata.org/entity/Q37311890

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 20 June 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

FOG2 protein down-regulation b ...... lated to diabetic nephropathy.

@en

FOG2 protein down-regulation b ...... lated to diabetic nephropathy.

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type

label

FOG2 protein down-regulation b ...... lated to diabetic nephropathy.

@en

FOG2 protein down-regulation b ...... lated to diabetic nephropathy.

@nl

prefLabel

FOG2 protein down-regulation b ...... lated to diabetic nephropathy.

@en

FOG2 protein down-regulation b ...... lated to diabetic nephropathy.

@nl

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JBC.M113.453043

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Journal of Biological Chemistry

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FOG2 protein down-regulation b ...... elated to diabetic nephropathy

@en

P2093

Hang Yuan

http://www.w3.org/2001/XMLSchema#string

Linda Lanting

http://www.w3.org/2001/XMLSchema#string

Mei Wang

http://www.w3.org/2001/XMLSchema#string

Mitsuo Kato

http://www.w3.org/2001/XMLSchema#string

Nancy Castro

http://www.w3.org/2001/XMLSchema#string

Rama Natarajan

http://www.w3.org/2001/XMLSchema#string

P2860

MicroRNAs: target recognition and regulatory functions

Conserved MicroRNA miR-8/miR-200 and its target USH/FOG2 control growth by regulating PI3K

Mesangial cell hypertrophy by high glucose is mediated by downregulation of the tumor suppressor PTEN

Molecular mechanisms of diabetic renal hypertrophy.

miR-192 mediates TGF-beta/Smad3-driven renal fibrosis.

Post-transcriptional up-regulation of Tsc-22 by Ybx1, a target of miR-216a, mediates TGF-{beta}-induced collagen expression in kidney cells.

Conserved cardiogenic functions of the multitype zinc-finger proteins: U-shaped and FOG-2.

Phosphoinositide 3-kinase controls early and late events in mammalian cell division

miR-200a Prevents renal fibrogenesis through repression of TGF-β2 expression.

MicroRNA-29c is a signature microRNA under high glucose conditions that targets Sprouty homolog 1, and its in vivo knockdown prevents progression of diabetic nephropathy.

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22469-22480

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10.1074/JBC.M113.453043

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23788640

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3829336

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