Elevated metals compromise repair of oxidative DNA damage via the base excision repair pathway: implications of pathologic iron overload in the brain on integrity of neuronal DNA.
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New perspectives on oxidized genome damage and repair inhibition by pro-oxidant metals in neurological diseasesIron chelation and multiple sclerosisThe presence and role of iron in mild traumatic brain injury: an imaging perspective.Iron in multiple sclerosis: roles in neurodegeneration and repair.Accumulation of oxidative DNA damage in brain mitochondria in mouse model of hereditary ferritinopathy.Pathogenic implications of iron accumulation in multiple sclerosis.Specific Inhibition of NEIL-initiated repair of oxidized base damage in human genome by copper and iron: potential etiological linkage to neurodegenerative diseases.Transgenic overexpression of neuroglobin attenuates formation of smoke-inhalation-induced oxidative DNA damage, in vivo, in the mouse brain.Oxidative genome damage and its repair: implications in aging and neurodegenerative diseases.Nuclear depletion of apurinic/apyrimidinic endonuclease 1 (Ape1/Ref-1) is an indicator of energy disruption in neuronsAntioxidant and astroprotective effects of a Pulicaria incisa infusion.Overexpressed neuroglobin raises threshold for nitric oxide-induced impairment of mitochondrial respiratory activities and stress signaling in primary cortical neurons.Oxidative genome damage and its repair in neurodegenerative diseases: function of transition metals as a double-edged sword.Oxidized base damage and single-strand break repair in mammalian genomes: role of disordered regions and posttranslational modifications in early enzymes.Molecular, Cellular and Clinical Aspects of Intracerebral Hemorrhage: Are the Enemies Within?Genotoxicity and DNA repair processes of zinc oxide nanoparticles.Chronic oxidative damage together with genome repair deficiency in the neurons is a double whammy for neurodegeneration: Is damage response signaling a potential therapeutic target?Cisplatin Toxicity in Dorsal Root Ganglion Neurons Is Relieved by Meclizine via Diminution of Mitochondrial Compromise and Improved Clearance of DNA Damage.Brain iron overload following intracranial haemorrhage.Polystyrene microsphere-ferritin conjugates: a robust phantom for correlation of relaxivity and size distribution.Neurotoxicity of cytarabine (Ara-C) in dorsal root ganglion neurons originates from impediment of mtDNA synthesis and compromise of mitochondrial function.Hemoglobin metabolism by-products are associated with an inflammatory response in patients with hemorrhagic stroke.Colocalization of Aluminum and Iron in Nuclei of Nerve Cells in Brains of Patients with Alzheimer's Disease
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P2860
Elevated metals compromise repair of oxidative DNA damage via the base excision repair pathway: implications of pathologic iron overload in the brain on integrity of neuronal DNA.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 08 July 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Elevated metals compromise rep ...... on integrity of neuronal DNA.
@en
Elevated metals compromise rep ...... on integrity of neuronal DNA.
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type
label
Elevated metals compromise rep ...... on integrity of neuronal DNA.
@en
Elevated metals compromise rep ...... on integrity of neuronal DNA.
@nl
prefLabel
Elevated metals compromise rep ...... on integrity of neuronal DNA.
@en
Elevated metals compromise rep ...... on integrity of neuronal DNA.
@nl
P2093
P2860
P1476
Elevated metals compromise rep ...... on integrity of neuronal DNA.
@en
P2093
Ella W Englander
Rafal Swiercz
P2860
P304
P356
10.1111/J.1471-4159.2009.06271.X
P407
P577
2009-07-08T00:00:00Z