TNF-alpha inhibition attenuates adverse myocardial remodeling in a rat model of volume overload.
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Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophyGender differences in non-ischemic myocardial remodeling: are they due to estrogen modulation of cardiac mast cells and/or membrane type 1 matrix metalloproteinaseRight ventricular failure and pathobiology in patients with congenital heart disease - implications for long-term follow-upIncreased atrial arrhythmia susceptibility induced by intense endurance exercise in mice requires TNFαMembrane-associated matrix proteolysis and heart failure.Estrogen attenuates chronic volume overload induced structural and functional remodeling in male rat hearts.Inhibition of matrix metalloproteinase activity prevents increases in myocardial tumor necrosis factor-alpha.Cardiac mast cells: the centrepiece in adverse myocardial remodelling.CXCR6 deficiency attenuates pressure overload-induced monocytes migration and cardiac fibrosis through downregulating TNF-α-dependent MMP9 pathway.Effects of a myofilament calcium sensitizer on left ventricular systolic and diastolic function in rats with volume overload heart failureTumor necrosis factor-alpha produced in cardiomyocytes mediates a predominant myocardial inflammatory response to stretch in early volume overload.Dynamic molecular and histopathological changes in the extracellular matrix and inflammation in the transition to heart failure in isolated volume overloadSubstance P induces adverse myocardial remodelling via a mechanism involving cardiac mast cells.The emerging prominence of the cardiac mast cell as a potent mediator of adverse myocardial remodelingEstrogenic modulation of inflammation-related genes in male rats following volume overloadIsolation of functional cardiac immune cellsPrevention of adverse cardiac remodeling to volume overload in female rats is the result of an estrogen-altered mast cell phenotype.Differential Effects of Prevention and Reversal Treatment with Lisinopril on Left Ventricular Remodelling in a Rat Model of Heart Failure.Estrogen modulates the influence of cardiac inflammatory cells on function of cardiac fibroblasts.Myofilament dysfunction as an emerging mechanism of volume overload heart failure.Adiponectin downregulation is associated with volume overload-induced myocyte dysfunction in rats.Estrogen receptor antagonism exacerbates cardiac structural and functional remodeling in female rats.Ang II-salt hypertension depends on neuronal activity in the hypothalamic paraventricular nucleus but not on local actions of tumor necrosis factor-α.miRNA208/Mef2 and TNF-α in right ventricular dysfunction: the transition from hypertrophy to failure.Protective Roles of Interferon-γ in Cardiac Hypertrophy Induced by Sustained Pressure Overload.Vitamin D attenuates pro-inflammatory TNF-α cytokine expression by inhibiting NF-кB/p65 signaling in hypertrophied rat hearts.Tumor necrosis factor-α and its role as a mediator in myocardial infarction: A brief review.
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TNF-alpha inhibition attenuates adverse myocardial remodeling in a rat model of volume overload.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on 07 August 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
TNF-alpha inhibition attenuate ...... rat model of volume overload.
@en
TNF-alpha inhibition attenuate ...... rat model of volume overload.
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type
label
TNF-alpha inhibition attenuate ...... rat model of volume overload.
@en
TNF-alpha inhibition attenuate ...... rat model of volume overload.
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prefLabel
TNF-alpha inhibition attenuate ...... rat model of volume overload.
@en
TNF-alpha inhibition attenuate ...... rat model of volume overload.
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P2093
P2860
P1476
TNF-alpha inhibition attenuate ...... rat model of volume overload.
@en
P2093
Giselle C Meléndez
Gregory L Brower
Joseph S Janicki
Lynetta J Jobe
P2860
P304
P356
10.1152/AJPHEART.00442.2009
P50
P577
2009-08-07T00:00:00Z