Responsiveness to HY antigen Ir gene complementation and target cell specificity.
about
H-2-linked control of cytotoxic T-cell responsiveness to alphavirus infection. Presence of H-2Dk during differentiation and stimulation converts stem cells of low responder genotype to T cells of responder phenotypeCD40 ligand-mediated interactions are involved in the generation of memory CD8(+) cytotoxic T lymphocytes (CTL) but are not required for the maintenance of CTL memory following virus infection.Evidence for major histocompatibility complex restriction in transplantation immunity.Cytotoxic T-cell responses to H-Y: correlation with the rejection of syngeneic male skin grafts.Cytotoxic T-cell responses to H-Y: mapping of the Ir genes.Cytotoxic T cells recognize male antigen and H-2 as distinct entities.Quantitative differences in the expression of parentally-derived H-2 antigens in F1 hybrid mice affect T-cell responsesH-2-linked genetic control of murine T-cell-mediated lympholysis to autologous cells modified with low concentrations of trinitrobenzene sulfonateRegulation of T-cell-mediated lympholysis by the murine major histocompatibility complex. I. Preferential in vitro responses to trinitrophenyl-modified self K- and D-coded gene products in parental and F1 hybrid mouse strainsCytotoxic T-cell responses in mice infected with influenza and vaccinia viruses vary in magnitude with H-2 genotype.Lethal graft-versus-host disease after bone marrow transplantation across minor histocompatibility barriers in mice. Prevention by removing mature T cells from marrow.Inhibition of H-Y cell-mediated cytolysis by H-2Db antiserum.H-Y antigen. Cell surface mapping and testosterone-induced supramolecular repatterning.Absence of allogeneic restriction in human T-cell-mediated cytotoxicity to Epstein-Barr virus-infected target cells. Demonstration of an HLA-linked control at the effector levelImmune response genes control T killer cell response against Moloney tumor antigen cytolysis regulating reactions against the best available H-2 + viral antigen association.Selective turnover and shedding of H-2K and H-2D antigens is controlled by the major histocompatibility complex. Implications for H-2-restricted recognition.H-2D (Rfv-1) gene influence on recovery from Friend virus leukemia is mediated by nonleukemic cells of the spleen and bone marrow.Ir gene function in an I-A subregion mutant B6.C-H-2bm12.Cytotoxic T cells specific for antigens expressed on surface immunoglobulin-positive cells.Influence of the major histocompatibility complex on the repertoire of allospecific cytolytic T lymphocytes.Murine interstitial nephritis. I. Analysis of disease susceptibility and its relationship of pleiomorphic gene products defining both immune-response genes and a restrictive requirement for cytotoxic T cells at H-2K.Helper T cells for cytotoxic T lymphocytes need not be I region restricted.Cytotoxic T cell response to minor histocompatibility antigens: apparent lack of H-2 restriction in killers stimulated by membrane fragmentsSusceptibility of human male keratinocytes to MHC-restricted H-Y-specific lysis.Successful T cell priming in B cell-deficient mice.Long-lasting CD8 T cell memory in the absence of CD4 T cells or B cells.Complementary role of CD4+ T cells and secondary lymphoid tissues for cross-presentation of tumor antigen to CD8+ T cells.Neonatal tolerance to alloantigens alters major histocompatibility complex-restricted response patternsB cells directly tolerize CD8(+) T cells.H-2 restriction as a consequence of intentional priming: T cells of fully allogeneic chimeric mice as well as of normal mice respond to foreign antigens in the context of H-2 determinants not encountered on thymic epithelial cellsDecay accelerating factor is essential for successful corneal engraftmentMajor histocompatibility complex-linked immune-responsiveness is acquired by lymphocytes of low-responder mice differentiating in thymus of high-responder miceH-2 complementation in anti-H-Y cytotoxic T-cell responses can occur in chimeric mice.Nuclear myxovirus-resistance protein Mx is a minor histocompatibility antigen.Dendritic cells and antigen presentation in the regulation of cytotoxic T lymphocyte responses against viruses and transplantation antigens.Ir-genes in H-2 regulate generation of anti-viral cytotoxic T cells. Mapping to K or D and dominance of unresponsiveness.Peptide HER2(776-788) represents a naturally processed broad MHC class II-restricted T cell epitope.Selection of cytotoxic T-cell precursors specific for minor histocompatibility determinants. I. Negative selection across H-2 barriers induced with disrupted cells but not with glutaraldehyde-treated cells: evidence for antigen processing.Fine specificity of alloimmune cytotoxic T lymphocytes directed against H-2K. A study with Kb mutants.Xenogeneic skin graft rejection is especially dependent on CD4+ T cells.
P2860
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P2860
Responsiveness to HY antigen Ir gene complementation and target cell specificity.
description
1977 nî lūn-bûn
@nan
1977年の論文
@ja
1977年学术文章
@wuu
1977年学术文章
@zh-cn
1977年学术文章
@zh-hans
1977年学术文章
@zh-my
1977年学术文章
@zh-sg
1977年學術文章
@yue
1977年學術文章
@zh
1977年學術文章
@zh-hant
name
Responsiveness to HY antigen Ir gene complementation and target cell specificity.
@en
Responsiveness to HY antigen Ir gene complementation and target cell specificity.
@nl
type
label
Responsiveness to HY antigen Ir gene complementation and target cell specificity.
@en
Responsiveness to HY antigen Ir gene complementation and target cell specificity.
@nl
prefLabel
Responsiveness to HY antigen Ir gene complementation and target cell specificity.
@en
Responsiveness to HY antigen Ir gene complementation and target cell specificity.
@nl
P2860
P1476
Responsiveness to HY antigen Ir gene complementation and target cell specificity.
@en
P2093
P2860
P356
10.1111/J.1600-065X.1977.TB00235.X
P577
1977-01-01T00:00:00Z