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MicroRNA Regulation of Human Breast Cancer Stem CellsMiR-200, a new star miRNA in human cancerTGF-β1/Smads and miR-21 in Renal Fibrosis and InflammationmiR-200c Regulation of Metastases in Ovarian Cancer: Potential Role in Epithelial and Mesenchymal TransitionmiRNA-200c inhibits invasion and metastasis of human non-small cell lung cancer by directly targeting ubiquitin specific peptidase 25Four amino acids within a tandem QxVx repeat in a predicted extended α-helix of the Smad-binding domain of Sip1 are necessary for binding to activated Smad proteinsTGF-β/Smad2/3 signaling directly regulates several miRNAs in mouse ES cells and early embryosmiR-376c promotes carcinogenesis and serves as a plasma marker for gastric carcinomaMicroRNA applications for prostate, ovarian and breast cancer in the era of precision medicine.Members of the microRNA-200 family are promising therapeutic targets in cancer.MiR-489 regulates chemoresistance in breast cancer via epithelial mesenchymal transition pathway.Crosstalk between TGF-β/Smad3 and BMP/BMPR2 signaling pathways via miR-17-92 cluster in carotid artery restenosisMicroRNA-200 (miR-200) cluster regulation by achaete scute-like 2 (Ascl2): impact on the epithelial-mesenchymal transition in colon cancer cells.Sonic hedgehog increases the skin wound-healing ability of mouse embryonic stem cells through the microRNA 200 family.Smad-mediated regulation of microRNA biosynthesis.Loss expression of micro ribonucleic acid (miRNA)-200c induces adverse post-surgical prognosis of advanced stage non-small cell lung carcinoma and its potential relationship with ETAR messenger RNAUpregulation of gingival tissue miR-200b in obese periodontitis subjectsKaempferol Suppresses Transforming Growth Factor-β1-Induced Epithelial-to-Mesenchymal Transition and Migration of A549 Lung Cancer Cells by Inhibiting Akt1-Mediated Phosphorylation of Smad3 at Threonine-179.Reversal of transforming growth factor-β induced epithelial-to-mesenchymal transition and the ZEB proteinsThe role of microRNAs in Helicobacter pylori pathogenesis and gastric carcinogenesis.Histone demethylase RBP2 promotes malignant progression of gastric cancer through TGF-β1-(p-Smad3)-RBP2-E-cadherin-Smad3 feedback circuit.Opposing Effects of Cyclooxygenase-2 (COX-2) on Estrogen Receptor β (ERβ) Response to 5α-Reductase Inhibition in Prostate Epithelial Cells.miR-489 inhibits silica-induced pulmonary fibrosis by targeting MyD88 and Smad3 and is negatively regulated by lncRNA CHRF.Regulation of the microRNA 200b (miRNA-200b) by transcriptional regulators PEA3 and ELK-1 protein affects expression of Pin1 protein to control anoikisFOG2 protein down-regulation by transforming growth factor-β1-induced microRNA-200b/c leads to Akt kinase activation and glomerular mesangial hypertrophy related to diabetic nephropathy.A double-negative feedback loop between E2F3b and miR- 200b regulates docetaxel chemosensitivity of human lung adenocarcinoma cellsThe role of microRNAs in cancers of the upper gastrointestinal tract.miR-200b and cancer/testis antigen CAGE form a feedback loop to regulate the invasion and tumorigenic and angiogenic responses of a cancer cell line to microtubule-targeting drugs.KLF5 activates microRNA 200 transcription to maintain epithelial characteristics and prevent induced epithelial-mesenchymal transition in epithelial cells.MicroRNA 135a suppresses lymph node metastasis through down-regulation of ROCK1 in early gastric cancer.Advances in molecular biomarkers for gastric cancer: miRNAs as emerging novel cancer markers.Emerging role of mucins in epithelial to mesenchymal transition.Post-transcriptional regulatory network of epithelial-to-mesenchymal and mesenchymal-to-epithelial transitions.Epithelial-mesenchymal transition, the tumor microenvironment, and metastatic behavior of epithelial malignancies.Gastric cancer: basic aspects.Molecular pathogenesis of gastric cancer.The conserved miR-8/miR-200 microRNA family and their role in invertebrate and vertebrate neurogenesis.Non-small-cell lung cancer and miRNAs: novel biomarkers and promising tools for treatment.Current perspectives toward the identification of key players in gastric cancer microRNA dysregulation.miR‑203 inhibits the expression of collagen‑related genes and the proliferation of hepatic stellate cells through a SMAD3‑dependent mechanism.
P2860
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P2860
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh
2011年學術文章
@zh-hant
name
Smad3 regulates E-cadherin via miRNA-200 pathway.
@en
Smad3 regulates E-cadherin via miRNA-200 pathway.
@nl
type
label
Smad3 regulates E-cadherin via miRNA-200 pathway.
@en
Smad3 regulates E-cadherin via miRNA-200 pathway.
@nl
prefLabel
Smad3 regulates E-cadherin via miRNA-200 pathway.
@en
Smad3 regulates E-cadherin via miRNA-200 pathway.
@nl
P2093
P356
P1433
P1476
Smad3 regulates E-cadherin via miRNA-200 pathway.
@en
P2093
H T H Trang
P2888
P304
P356
10.1038/ONC.2011.484
P407
P577
2011-10-24T00:00:00Z
P5875
P6179
1010147130