Fresh isolates from children with severe Plasmodium falciparum malaria bind to multiple receptors.
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Improved in vitro culture of Plasmodium falciparum permits establishment of clinical isolates with preserved multiplication, invasion and rosetting phenotypesSurface co-expression of two different PfEMP1 antigens on single plasmodium falciparum-infected erythrocytes facilitates binding to ICAM1 and PECAM1Release of sequestered malaria parasites upon injection of a glycosaminoglycanA Plasmodium gene family encoding Maurer's cleft membrane proteins: structural properties and expression profilingPlacental Sequestration of Plasmodium falciparum Malaria Parasites Is Mediated by the Interaction Between VAR2CSA and Chondroitin Sulfate A on Syndecan-1The role of PfEMP1 as targets of naturally acquired immunity to childhood malaria: prospects for a vaccineThe contribution of Plasmodium chabaudi to our understanding of malariaGeographical and temporal conservation of antibody recognition of Plasmodium falciparum variant surface antigens.High throughput functional assays of the variant antigen PfEMP1 reveal a single domain in the 3D7 Plasmodium falciparum genome that binds ICAM1 with high affinity and is targeted by naturally acquired neutralizing antibodiesContinued cytoadherence of Plasmodium falciparum infected red blood cells after antimalarial treatment.Specific receptor usage in Plasmodium falciparum cytoadherence is associated with disease outcome.CR1 Knops blood group alleles are not associated with severe malaria in the GambiaHigh levels of Plasmodium falciparum rosetting in all clinical forms of severe malaria in African children.Adhesion of Plasmodium falciparum-infected erythrocytes to human cells: molecular mechanisms and therapeutic implications.Amplification of P. falciparum Cytoadherence through induction of a pro-adhesive state in host endothelium.Cytoadherence phenotype of Plasmodium falciparum-infected erythrocytes is associated with specific pfemp-1 expression in parasites from children with cerebral malariaA subset of group A-like var genes encodes the malaria parasite ligands for binding to human brain endothelial cellsPlasmodium falciparum rosetting epitopes converge in the SD3-loop of PfEMP1-DBL1α.Plasmodium falciparum var genes expressed in children with severe malaria encode CIDRα1 domains.The structural basis for CD36 binding by the malaria parasite.Low anticoagulant heparin disrupts Plasmodium falciparum rosettes in fresh clinical isolates.B-cell epitopes in NTS-DBL1α of PfEMP1 recognized by human antibodies in Rosetting Plasmodium falciparumNonimmune immunoglobulin binding and multiple adhesion characterize Plasmodium falciparum-infected erythrocytes of placental origin.Homology blocks of Plasmodium falciparum var genes and clinically distinct forms of severe malaria in a local populationCD36 contributes to malaria parasite-induced pro-inflammatory cytokine production and NK and T cell activation by dendritic cells.Association of the single-nucleotide polymorphism and haplotype of the complement receptor 1 gene with malariaPlasmodium falciparum-infected erythrocytes adhere both in the intervillous space and on the villous surface of human placenta by binding to the low-sulfated chondroitin sulfate proteoglycan receptor.DNA immunization with the cysteine-rich interdomain region 1 of the Plasmodium falciparum variant antigen elicits limited cross-reactive antibody responses.Generation of cross-protective antibodies against Plasmodium falciparum sequestration by immunization with an erythrocyte membrane protein 1-duffy binding-like 1 alpha domain.Expression of merozoite surface protein markers by Plasmodium falciparum-infected erythrocytes in peripheral blood and tissues of children with fatal malaria.Chondroitin sulfate proteoglycan but not hyaluronic acid is the receptor for the adherence of Plasmodium falciparum-infected erythrocytes in human placenta, and infected red blood cell adherence up-regulates the receptor expression.Cytoadhesion to gC1qR through Plasmodium falciparum Erythrocyte Membrane Protein 1 in Severe Malaria.Cytoadherence and severe malaria.Clinical and molecular aspects of severe malaria.Functional analysis of monoclonal antibodies against the Plasmodium falciparum PfEMP1-VarO adhesinThe host genetic diversity in malaria infection.Genetic diversity of Plasmodium falciparum infections in mild and severe malaria of children from Kampala, Uganda.Activated Neutrophils Are Associated with Pediatric Cerebral Malaria Vasculopathy in Malawian ChildrenAnalysis of antibody induction upon immunization with distinct NTS-DBL1α-domains of PfEMP1 from rosetting Plasmodium falciparum parasites.An in vivo and in vitro model of Plasmodium falciparum rosetting and autoagglutination mediated by varO, a group A var gene encoding a frequent serotype.
P2860
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P2860
Fresh isolates from children with severe Plasmodium falciparum malaria bind to multiple receptors.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Fresh isolates from children w ...... ia bind to multiple receptors.
@en
Fresh isolates from children w ...... ia bind to multiple receptors.
@nl
type
label
Fresh isolates from children w ...... ia bind to multiple receptors.
@en
Fresh isolates from children w ...... ia bind to multiple receptors.
@nl
prefLabel
Fresh isolates from children w ...... ia bind to multiple receptors.
@en
Fresh isolates from children w ...... ia bind to multiple receptors.
@nl
P2093
P2860
P1476
Fresh isolates from children w ...... ria bind to multiple receptors
@en
P2093
F Pettersson
M Wahlgren
P2860
P304
P356
10.1128/IAI.69.9.5849-5856.2001
P407
P577
2001-09-01T00:00:00Z