Genetic evidence that EBNA-1 is needed for efficient, stable latent infection by Epstein-Barr virus
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Inhibition of Epstein-Barr virus OriP function by tankyrase, a telomere-associated poly-ADP ribose polymerase that binds and modifies EBNA1Human herpesvirus 8 LANA interacts with proteins of the mSin3 corepressor complex and negatively regulates Epstein-Barr virus gene expression in dually infected PEL cellsThermophilic bacteria strictly obey Szybalski's transcription direction rule and politely purine-load RNAs with both adenine and guanineEBNA-1, a bifunctional transcriptional activatorThe EBV nuclear antigen 1 (EBNA1) enhances B cell immortalization several thousandfoldInterplay of Murine Gammaherpesvirus 68 with NF-kappaB Signaling of the HostAntigen-binding properties of monoclonal antibodies reactive with EBNA1 and use in immunoaffinity chromatographyA role for MRE11, NBS1, and recombination junctions in replication and stable maintenance of EBV episomes.EBNA1 regulates cellular gene expression by binding cellular promotersInhibition of antigen presentation by the glycine/alanine repeat domain is not conserved in simian homologues of Epstein-Barr virus nuclear antigen 1.Epstein-Barr virus nuclear antigen 1 does not induce lymphoma in transgenic FVB mice.Cytomegalovirus latency and reactivation: recent insights into an age old problem.The placental specific gene, PLAC1, is induced by the Epstein-Barr virus and is expressed in human tumor cellsKaposi's sarcoma-associated herpesvirus latency-associated nuclear antigen 1 mediates episome persistence through cis-acting terminal repeat (TR) sequence and specifically binds TR DNA.EBNA-LP associates with cellular proteins including DNA-PK and HA95.Close but distinct regions of human herpesvirus 8 latency-associated nuclear antigen 1 are responsible for nuclear targeting and binding to human mitotic chromosomes.Linkage between STAT regulation and Epstein-Barr virus gene expression in tumors.Establishment of latent Epstein-Barr virus infection and stable episomal maintenance in murine B-cell lines.The genetic approach to the Epstein-Barr virus: from basic virology to gene therapy.Epstein-Barr virus nuclear antigen 1 activates transcription from episomal but not integrated DNA and does not alter lymphocyte growthC-terminal region of EBNA-2 determines the superior transforming ability of type 1 Epstein-Barr virus by enhanced gene regulation of LMP-1 and CXCR7.Evolutionary aspects of oncogenic herpesvirusesMeasurement of Epstein-Barr virus DNA load using a novel quantification standard containing two EBV DNA targets and SYBR Green I dyeMolecular virology of Epstein-Barr virus.Epstein-barr virus-induced changes in B-lymphocyte gene expression.Complete genomic sequence of an Epstein-Barr virus-related herpesvirus naturally infecting a new world primate: a defining point in the evolution of oncogenic lymphocryptoviruses.Functional modulation of the metastatic suppressor Nm23-H1 by oncogenic virusesThe latency-associated nuclear antigen of Kaposi's sarcoma-associated herpesvirus manipulates the activity of glycogen synthase kinase-3betaThe Epstein-Barr virus latency BamHI-Q promoter is positively regulated by STATs and Zta interference with JAK/STAT activation leads to loss of BamHI-Q promoter activityLive-cell imaging reveals multiple interactions between Epstein-Barr virus nuclear antigen 1 and cellular chromatin during interphase and mitosisCD8 T cell recognition of endogenously expressed epstein-barr virus nuclear antigen 1Telomere repeat binding factors TRF1, TRF2, and hRAP1 modulate replication of Epstein-Barr virus OriPPhosphorylation sites of Epstein-Barr virus EBNA1 regulate its functionNucleoside diphosphate kinase/Nm23 and Epstein-Barr virus.Prospects of a novel vaccination strategy for human gamma-herpesviruses.The Epstein-Barr Virus EBNA1 Protein.Epstein-Barr virus infection and persistence in nasopharyngeal epithelial cells.Deregulation of the cell cycle machinery by Epstein-Barr virus nuclear antigen 3C.Epstein-Barr virus EBNA-3C is targeted to and regulates expression from the bidirectional LMP-1/2B promoter.Epstein-Barr virus small RNAs potentiate tumorigenicity of Burkitt lymphoma cells independently of an effect on apoptosis.
P2860
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P2860
Genetic evidence that EBNA-1 is needed for efficient, stable latent infection by Epstein-Barr virus
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Genetic evidence that EBNA-1 i ...... nfection by Epstein-Barr virus
@en
Genetic evidence that EBNA-1 i ...... fection by Epstein-Barr virus.
@nl
type
label
Genetic evidence that EBNA-1 i ...... nfection by Epstein-Barr virus
@en
Genetic evidence that EBNA-1 i ...... fection by Epstein-Barr virus.
@nl
prefLabel
Genetic evidence that EBNA-1 i ...... nfection by Epstein-Barr virus
@en
Genetic evidence that EBNA-1 i ...... fection by Epstein-Barr virus.
@nl
P2093
P2860
P1433
P1476
Genetic evidence that EBNA-1 i ...... nfection by Epstein-Barr virus
@en
P2093
P2860
P304
P407
P577
1999-04-01T00:00:00Z