Lysine acetyltransferase PCAF is a key regulator of arteriogenesis. - Wikidata - wikimedia - TriplyDB

Lysine acetyltransferase PCAF is a key regulator of arteriogenesis.

Lysine acetyltransferase PCAF is a key regulator of arteriogenesis.

http://www.wikidata.org/entity/Q40428568

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Histone acetyltransferases: challenges in targeting bi-substrate enzymes Adipose tissue-derived mesenchymal stem cells in long-term dialysis patients display downregulation of PCAF expression and poor angiogenesis activation.TLR4 accessory molecule RP105 (CD180) regulates monocyte-driven arteriogenesis in a murine hind limb ischemia model.P2Y2 nucleotide receptor mediates arteriogenesis in a murine model of hind limb ischemia.CXCL1 microspheres: a novel tool to stimulate arteriogenesis.Histone acetyltransferase PCAF regulates inflammatory molecules in the development of renal injury Histone acetyltransferase PCAF accelerates apoptosis by repressing a GLI1/BCL2/BAX axis in hepatocellular carcinoma.Histone acetylation and histone acetyltransferases show significant alterations in human abdominal aortic aneurysm.P300/CBP-associated factor (PCAF) inhibits the growth of hepatocellular carcinoma by promoting cell autophagy.A functional genomics screen identifies PCAF and ADA3 as regulators of human granzyme B-mediated apoptosis and Bid cleavage.Discovery of a PCAF Bromodomain Chemical Probe.Epigenetics in the Vascular Endothelium: Looking From a Different Perspective in the Epigenomics Era.Translational Perspective on Epigenetics in Cardiovascular Disease.Changes in DNA Methylation and Chromatin Structure of Pro-inflammatory Cytokines Stimulated by LPS in Broiler Peripheral Blood Mononuclear Cells.The epigenetic factor PCAF regulates vascular inflammation and is essential for intimal hyperplasia development DNA Methyltransferase 1-Dependent DNA Hypermethylation Constrains Arteriogenesis by Augmenting Shear Stress Set Point.CCDC134 serves a crucial role in embryonic development.Histone acetyltransferase 7 (KAT7)-dependent intragenic histone acetylation regulates endothelial cell gene regulation.Loss of osteoglycin promotes angiogenesis in limb ischaemia mouse models via modulation of vascular endothelial growth factor and vascular endothelial growth factor receptor 2 signalling pathway.A homozygous KAT2B variant modulates the clinical phenotype of ADD3 deficiency in humans and flies.Discovery of a PCAF Bromodomain Chemical Probe

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Lysine acetyltransferase PCAF is a key regulator of arteriogenesis.

http://www.wikidata.org/entity/Q40428568

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2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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2013年學術文章

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2013年學術文章

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name

Lysine acetyltransferase PCAF is a key regulator of arteriogenesis.

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type

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Lysine acetyltransferase PCAF is a key regulator of arteriogenesis.

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prefLabel

Lysine acetyltransferase PCAF is a key regulator of arteriogenesis.

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ATVBAHA.113.301579

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Arteriosclerosis, Thrombosis, and Vascular Biology

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Lysine acetyltransferase PCAF is a key regulator of arteriogenesis

@en

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Antonius J N M Bastiaansen

http://www.w3.org/2001/XMLSchema#string

Erna A B Peters

http://www.w3.org/2001/XMLSchema#string

Hetty C de Boer

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J Wouter Jukema

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Jaap F Hamming

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James E Faber

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Margreet R de Vries

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Mark M Ewing

http://www.w3.org/2001/XMLSchema#string

Ramon Arens

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Sabine M J Welten

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P2860

Sirtuin 1 modulates cellular responses to hypoxia by deacetylating hypoxia-inducible factor 1alpha

p53 sites acetylated in vitro by PCAF and p300 are acetylated in vivo in response to DNA damage

NF-kappa B: a pleiotropic mediator of inducible and tissue-specific gene control

Spatial and temporal coordination of bone marrow-derived cell activity during arteriogenesis: regulation of the endogenous response and therapeutic implications.

Blood monocyte concentration is critical for enhancement of collateral artery growth.

Direct evidence for tumor necrosis factor-alpha signaling in arteriogenesis.

CD8+ T lymphocytes regulate the arteriogenic response to ischemia by infiltrating the site of collateral vessel development and recruiting CD4+ mononuclear cells through the expression of interleukin-16.

Matrix metalloproteinase-9 is essential for ischemia-induced neovascularization by modulating bone marrow-derived endothelial progenitor cells.

Strain-dependent variation in collateral circulatory function in mouse hindlimb.

Variations in surgical procedures for hind limb ischaemia mouse models result in differences in collateral formation.

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1902-1910

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scholarly article

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10.1161/ATVBAHA.113.301579

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8

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33

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Anne Yael Nossent

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2013-06-20T00:00:00Z

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23788761

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4049097

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