Immunotherapy Gone Viral: Bortezomib and oHSV Enhance Antitumor NK-Cell Activity

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Immunotherapy Gone Viral: Bortezomib and oHSV Enhance Antitumor NK-Cell Activity

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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2016年學術文章

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Immunotherapy Gone Viral: Bortezomib and oHSV Enhance Antitumor NK-Cell Activity

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Immunotherapy Gone Viral: Bortezomib and oHSV Enhance Antitumor NK-Cell Activity

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Immunotherapy Gone Viral: Bortezomib and oHSV Enhance Antitumor NK-Cell Activity

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P1476

Immunotherapy Gone Viral: Bortezomib and oHSV Enhance Antitumor NK-Cell Activity

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P2093

Carter M Suryadevara

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Katherine A Riccione

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P2860

Oncolytic herpes simplex virus-based strategies: toward a breakthrough in glioblastoma therapy

Molecular mechanisms of necroptosis: an ordered cellular explosion

Bortezomib-induced unfolded protein response increases oncolytic HSV-1 replication resulting in synergistic antitumor effects.

Immunologic escape after prolonged progression-free survival with epidermal growth factor receptor variant III peptide vaccination in patients with newly diagnosed glioblastoma.

The proteasome: a suitable antineoplastic target.

NK cells impede glioblastoma virotherapy through NKp30 and NKp46 natural cytotoxicity receptors.

Potentiating oncolytic viral therapy through an understanding of the initial immune responses to oncolytic viral infection.

Bortezomib Treatment Sensitizes Oncolytic HSV-1-Treated Tumors to NK Cell Immunotherapy.

Increased regulatory T-cell fraction amidst a diminished CD4 compartment explains cellular immune defects in patients with malignant glioma.

P304

5164-5166

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P31

scholarly article

P356

10.1158/1078-0432.CCR-16-1666

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P407

English

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P478

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P50

J H Sampson

P577

2016-08-12T00:00:00Z

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P698

27521450

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P932

5093093

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