Modulation of DNA damage tolerance in Escherichia coli recG and ruv strains by mutations affecting PriB, the ribosome and RNA polymerase.
about
Replication Termination: Containing Fork Fusion-Mediated Pathologies in Escherichia coliStringent response processes suppress DNA damage sensitivity caused by deficiency in full-length translation initiation factor 2 or PriA helicaseEffects of conserved residues and naturally occurring mutations on Mycobacterium tuberculosis RecG helicase activity.Regulation of Transcript Elongation.Inhibiting translation elongation can aid genome duplication in Escherichia coli.Cellular location and activity of Escherichia coli RecG proteins shed light on the function of its structurally unresolved C-terminus.25 years on and no end in sight: a perspective on the role of RecG proteinAvoiding chromosome pathology when replication forks collide.Mechanisms of bacterial DNA replication restart.
P2860
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P2860
Modulation of DNA damage tolerance in Escherichia coli recG and ruv strains by mutations affecting PriB, the ribosome and RNA polymerase.
description
2012 nî lūn-bûn
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
@zh-hk
2012年論文
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2012年論文
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2012年论文
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2012年论文
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2012年论文
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name
Modulation of DNA damage toler ...... e ribosome and RNA polymerase.
@en
type
label
Modulation of DNA damage toler ...... e ribosome and RNA polymerase.
@en
prefLabel
Modulation of DNA damage toler ...... e ribosome and RNA polymerase.
@en
P2860
P356
P1476
Modulation of DNA damage toler ...... e ribosome and RNA polymerase.
@en
P2093
Akeel A Mahdi
Geoffrey S Briggs
P2860
P304
P356
10.1111/MMI.12010
P407
P577
2012-09-07T00:00:00Z