Mitochondrial superoxide dismutase and glutathione peroxidase in idiosyncratic drug-induced liver injury. - Wikidata - wikimedia - TriplyDB

Mitochondrial superoxide dismutase and glutathione peroxidase in idiosyncratic drug-induced liver injury.

Mitochondrial superoxide dismutase and glutathione peroxidase in idiosyncratic drug-induced liver injury.

http://www.wikidata.org/entity/Q43005717

about

Case Characterization, Clinical Features and Risk Factors in Drug-Induced Liver Injury Drug Induced Liver Injury: Review with a Focus on Genetic Factors, Tissue Diagnosis, and Treatment Options Genetic variations in drug-induced liver injury (DILI): resolving the puzzle.Mechanisms of drug-induced liver injury: from bedside to bench.Polymorphism in glutamate cysteine ligase catalytic subunit (GCLC) is associated with sulfamethoxazole-induced hypersensitivity in HIV/AIDS patients Superoxide Dismutase Gene (SOD1, SOD2, and SOD3) Polymorphisms and Antituberculosis Drug-induced Hepatitis.Trends in qualifying biomarkers in drug safety. Consensus of the 2011 meeting of the spanish society of clinical pharmacology FCERI and Histamine Metabolism Gene Variability in Selective Responders to NSAIDS Expression and polymorphism (rs4880) of mitochondrial superoxide dismutase (SOD2) and asparaginase induced hepatotoxicity in adult patients with acute lymphoblastic leukemia.Idiosyncratic Drug-Induced Liver Injury (IDILI): Potential Mechanisms and Predictive Assays.Oxidant stress, mitochondria, and cell death mechanisms in drug-induced liver injury: lessons learned from acetaminophen hepatotoxicity Drug-induced autoimmune-like hepatitis.Assessment of nonsteroidal anti-inflammatory drug-induced hepatotoxicity.Central role of mitochondria in drug-induced liver injury.Genetic association studies in drug-induced liver injury.The primary role of hepatic metabolism in idiosyncratic drug-induced liver injury.Drug-induced liver injury: the role of drug metabolism and transport.Molecular mechanisms of hepatic apoptosis.Transitioning from Idiopathic to Explainable Autoimmune Hepatitis.Gamma-Glutamylcysteine Ethyl Ester Protects against Cyclophosphamide-Induced Liver Injury and Hematologic Alterations via Upregulation of PPARγ and Attenuation of Oxidative Stress, Inflammation, and Apoptosis.Serum markers for mitochondrial dysfunction and cell death are possible predictive indicators for drug-induced liver injury by direct acting antivirals."Autoimmune(-Like)" Drug and Herb Induced Liver Injury: New Insights into Molecular Pathogenesis.Role of chemical structures and the 1331T>C bile salt export pump polymorphism in idiosyncratic drug-induced liver injury.Quercetin ameliorates liver injury induced with Tripterygium glycosides by reducing oxidative stress and inflammation.Hepatitis E Virus Induces Hepatocyte Apoptosis via Mitochondrial Pathway in Mongolian Gerbils.

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P2860

Mitochondrial superoxide dismutase and glutathione peroxidase in idiosyncratic drug-induced liver injury.

http://www.wikidata.org/entity/Q43005717

description

2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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2010年學術文章

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name

Mitochondrial superoxide dismu ...... tic drug-induced liver injury.

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Mitochondrial superoxide dismu ...... tic drug-induced liver injury.

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type

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Mitochondrial superoxide dismu ...... tic drug-induced liver injury.

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Mitochondrial superoxide dismu ...... tic drug-induced liver injury.

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Mitochondrial superoxide dismu ...... atic drug-induced liver injury

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Ana Melcón De Dios

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Augustin Castiella

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Camilla Stephens

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Carmen Martínez

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Eugenia Ulzurrun

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Jhon D Ruiz

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José Antonio Durán

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M Carmen Fernandez

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M Isabel Lucena

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Manuel Romero-Gomez

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P2860

Mechanisms of hepatotoxicity

Idiosyncratic drug reactions: past, present, and future

Criteria of drug-induced liver disorders. Report of an international consensus meeting.

Candidate gene case-control association studies: advantages and potential pitfalls.

Underlying mitochondrial dysfunction triggers flutamide-induced oxidative liver injury in a mouse model of idiosyncratic drug toxicity.

Genetic polymorphisms of CYP2C9 and CYP2C19 are not related to drug-induced idiosyncratic liver injury (DILI)

Mechanism of the tumor suppressive effect of MnSOD overexpression.

Idiosyncratic drug hepatotoxicity.

A comprehensive listing of bioactivation pathways of organic functional groups.

Role of metabolism in drug-induced idiosyncratic hepatotoxicity.

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303-312

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scholarly article

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10.1002/HEP.23668

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1

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52

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Elena García-Martín

Raul J. Andrade

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2010-07-01T00:00:00Z

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44800155

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