Organoselenium (Sel-Plex diet) decreases amyloid burden and RNA and DNA oxidative damage in APP/PS1 mice.
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Selenoproteins in nervous system development and functionRegulation and function of selenoproteins in human diseaseGene expression profiling reveals differential effects of sodium selenite, selenomethionine, and yeast-derived selenium in the mouse.Dietary Supplementation with Organoselenium Accelerates Recovery of Bladder Expression, but Does Not Improve Locomotor Function, following Spinal Cord Injury.Does a role for selenium in DNA damage repair explain apparent controversies in its use in chemoprevention?Sex-specific transcriptional responses of the zebrafish (Danio rerio) brain selenoproteome to acute sodium selenite supplementation.Nucleic acid oxidation: an early feature of Alzheimer's disease.Oxidant/Antioxidant imbalance and the risk of Alzheimer's diseaseSelenium interactions and toxicity: a review. Selenium interactions and toxicity.Oxidative damage to RNA in aging and neurodegenerative disorders.Metal dyshomeostasis and inflammation in Alzheimer's and Parkinson's diseases: possible impact of environmental exposures.Which form is that? The importance of selenium speciation and metabolism in the prevention and treatment of disease.Selenium and selenoprotein function in brain disorders.Strategy to reduce free radical species in Alzheimer's disease: an update of selected antioxidants.Selenoprotein S Reduces Endoplasmic Reticulum Stress-Induced Phosphorylation of Tau: Potential Role in Selenate Mitigation of Tau Pathology.Association of Antioxidant Supplement Use and Dementia in the Prevention of Alzheimer's Disease by Vitamin E and Selenium Trial (PREADViSE).Dimebon inhibits calcium-induced swelling of rat brain mitochondria but does not alter calcium retention or cytochrome C release.Supplementation with selenium-enriched yeast attenuates brain metastatic growth.Intravenous Administration of Human Umbilical Cord Mesenchymal Stem Cells Improves Cognitive Impairments and Reduces Amyloid-Beta Deposition in an AβPP/PS1 Transgenic Mouse Model.Aerobic exercise combined with antioxidative treatment does not counteract moderate- or mid-stage Alzheimer-like pathophysiology of APP/PS1 mice.Elevated amyloid-β plaque deposition in dietary selenium-deficient Tg2576 transgenic mice.Overexpression of selenoprotein H prevents mitochondrial dynamic imbalance induced by glutamate exposure.Reversal of memory and neuropsychiatric symptoms and reduced tau pathology by selenium in 3xTg-AD mice.Design, synthesis and evaluation of seleno-dihydropyrimidinones as potential multi-targeted therapeutics for Alzheimer's disease.
P2860
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P2860
Organoselenium (Sel-Plex diet) decreases amyloid burden and RNA and DNA oxidative damage in APP/PS1 mice.
description
2009 nî lūn-bûn
@nan
2009年の論文
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2009年学术文章
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2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
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2009年學術文章
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2009年學術文章
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name
Organoselenium (Sel-Plex diet) ...... dative damage in APP/PS1 mice.
@en
Organoselenium
@nl
type
label
Organoselenium (Sel-Plex diet) ...... dative damage in APP/PS1 mice.
@en
Organoselenium
@nl
prefLabel
Organoselenium (Sel-Plex diet) ...... dative damage in APP/PS1 mice.
@en
Organoselenium
@nl
P2093
P2860
P1476
Organoselenium (Sel-Plex diet) ...... dative damage in APP/PS1 mice.
@en
P2093
Ganna Lyubartseva
Mark A Lovell
Shuling Xiong
William R Markesbery
P2860
P304
P356
10.1016/J.FREERADBIOMED.2009.03.008
P577
2009-03-19T00:00:00Z