Abeta(31-35) and Abeta(25-35) fragments of amyloid beta-protein induce cellular death through apoptotic signals: Role of the redox state of methionine-35. - Wikidata - wikimedia - TriplyDB

Abeta(31-35) and Abeta(25-35) fragments of amyloid beta-protein induce cellular death through apoptotic signals: Role of the redox state of methionine-35.

Abeta(31-35) and Abeta(25-35) fragments of amyloid beta-protein induce cellular death through apoptotic signals: Role of the redox state of methionine-35.

http://www.wikidata.org/entity/Q46487407

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Mitochondrial Dysfunction Contributes to the Pathogenesis of Alzheimer's Disease Novel NSAID-Derived Drugs for the Potential Treatment of Alzheimer's Disease Selenomethionine incorporation into amyloid sequences regulates fibrillogenesis and toxicity Neuroprotective effects of genistein and folic acid on apoptosis of rat cultured cortical neurons induced by beta-amyloid 31-35.Genistein and folic acid prevent oxidative injury induced by β-amyloid peptide.Interactions between amyloid-β and Tau fragments promote aberrant aggregates: implications for amyloid toxicity Mitochondrial protection by the mixed muscarinic/σ1 ligand ANAVEX2-73, a tetrahydrofuran derivative, in Aβ25-35 peptide-injected mice, a nontransgenic Alzheimer's disease model.Effects of solvent on the structure of the Alzheimer amyloid-beta(25-35) peptide.Mass spectrometry as an efficient tool for the characterization of amyloid β peptide 25-35 self-assembly species in aggregation and inhibition studies.Interaction of Aβ(25-35) fibrillation products with mitochondria: Effect of small-molecule natural products.Accumulation of exogenous amyloid-beta peptide in hippocampal mitochondria causes their dysfunction: a protective role for melatonin.Role of β-hairpin formation in aggregation: the self-assembly of the amyloid-β(25-35) peptide.Centella asiatica extract protects against amyloid β1-40-induced neurotoxicity in neuronal cells by activating the antioxidative defence system.Exendin-4 antagonizes Aβ1-42-induced attenuation of spatial learning and memory ability Investigating the Neuroprotective Effects of Turmeric Extract: Structural Interactions of β-Amyloid Peptide with Single Curcuminoids.Deep Phosphoproteomic Measurements Pinpointing Drug Induced Protective Mechanisms in Neuronal Cells.Amyloid-β-Induced Changes in Molecular Clock Properties and Cellular Bioenergetics.Protective role of melatonin in mitochondrial dysfunction and related disorders.Cholinesterase inhibitory, anti-amyloidogenic and neuroprotective effect of the medicinal plant Grewia tiliaefolia - An in vitro and in silico study.Icariside II, a Broad-Spectrum Anti-cancer Agent, Reverses Beta-Amyloid-Induced Cognitive Impairment through Reducing Inflammation and Apoptosis in Rats.Selenofuranoside Ameliorates Memory Loss in Alzheimer-Like Sporadic Dementia: AChE Activity, Oxidative Stress, and Inflammation Involvement.From the dual function lead AP2238 to AP2469, a multi-target-directed ligand for the treatment of Alzheimer's disease.Specific reaction of Met 35 in amyloid beta peptide with hypochlorous acid.Long-term Aβ exposure augments mCa2+-independent mROS-mediated depletion of cardiolipin for the shift of a lethal transient mitochondrial permeability transition to its permanent mode in NARP cybrids: a protective targeting of melatonin.Structure and membrane interactions of the β-amyloid fragment 25-35 as viewed using spectroscopic approaches.Effect of methionine-35 oxidation on the aggregation of amyloid-β peptide.Amyloid β-protein suppressed nicotinic acetylcholine receptor-mediated currents in acutely isolated rat hippocampal CA1 pyramidal neurons.Hydrophobic tail length plays a pivotal role in amyloid beta (25-35) fibril-surfactant interactions.

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P2860

Abeta(31-35) and Abeta(25-35) fragments of amyloid beta-protein induce cellular death through apoptotic signals: Role of the redox state of methionine-35.

http://www.wikidata.org/entity/Q46487407

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

@zh-my

2005年学术文章

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2005年學術文章

@yue

2005年學術文章

@zh-hant

name

Abeta(31-35) and Abeta(25-35) ...... redox state of methionine-35.

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type

label

Abeta(31-35) and Abeta(25-35) ...... redox state of methionine-35.

@en

prefLabel

Abeta(31-35) and Abeta(25-35) ...... redox state of methionine-35.

@en

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P356

J.FEBSLET.2005.04.041

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P1476

Abeta(31-35) and Abeta(25-35) ...... e redox state of methionine-35

@en

P2093

Bruno Giardina

http://www.w3.org/2001/XMLSchema#string

Federica Orsini

http://www.w3.org/2001/XMLSchema#string

Massimo Coletta

http://www.w3.org/2001/XMLSchema#string

Stefano Marini

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P2860

Neurotrophic and neurotoxic effects of amyloid beta protein: reversal by tachykinin neuropeptides

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

Caspases: killer proteases

Neurotoxic, redox-competent Alzheimer's beta-amyloid is released from lipid membrane by methionine oxidation.

Amyloid beta protein-(1-42) forms calcium-permeable, Zn2+-sensitive channel.

Methionine residue 35 is critical for the oxidative stress and neurotoxic properties of Alzheimer's amyloid beta-peptide 1-42.

X-ray diffraction from intraneuronal paired helical filaments and extraneuronal amyloid fibers in Alzheimer disease indicates cross-beta conformation.

Beta-amyloid neurotoxicity requires fibril formation and is inhibited by congo red.

Apoptosis is induced by beta-amyloid in cultured central nervous system neurons

Oxidation of methionyl residues in proteins: tools, targets, and reversal.

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2913-2918

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10.1016/J.FEBSLET.2005.04.041

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13

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Maria Elisabetta Clementi

Francesco Misiti

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7851655

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15890345

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