The development of emphysema in cigarette smoke-exposed mice is strain dependent. - Wikidata - wikimedia - TriplyDB

The development of emphysema in cigarette smoke-exposed mice is strain dependent.

The development of emphysema in cigarette smoke-exposed mice is strain dependent.

http://www.wikidata.org/entity/Q47949544

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Mammalian nicotinic acetylcholine receptors: from structure to function Effect of short-term cigarette smoke exposure on body weight, appetite and brain neuropeptide Y in mice Animal models of chronic obstructive pulmonary disease Rtp801, a suppressor of mTOR signaling, is an essential mediator of cigarette smoke-induced pulmonary injury and emphysema Thioredoxin-1 protects against neutrophilic inflammation and emphysema progression in a mouse model of chronic obstructive pulmonary disease exacerbation Rat and hamster species differences in susceptibility to elastase-induced pulmonary emphysema relate to differences in elastase inhibitory capacity Protein carbonylation and muscle function in COPD and other conditions.Integrative analysis of DNA methylation and gene expression data identifies EPAS1 as a key regulator of COPD.p38 mitogen-activated protein kinase determines the susceptibility to cigarette smoke-induced emphysema in mice.Targeted disruption of NF-{kappa}B1 (p50) augments cigarette smoke-induced lung inflammation and emphysema in mice: a critical role of p50 in chromatin remodeling A murine model of elastase- and cigarette smoke-induced emphysema.Leptin receptor polymorphisms and lung function decline in COPD Inflammatory changes in the airways of mice caused by cigarette smoke exposure are only partially reversed after smoking cessation.Novosphingobium and its potential role in chronic obstructive pulmonary diseases: insights from microbiome studies.Haploinsufficiency of Hedgehog interacting protein causes increased emphysema induced by cigarette smoke through network rewiring.An aberrant leukotriene A4 hydrolase-proline-glycine-proline pathway in the pathogenesis of chronic obstructive pulmonary disease Chronic cigarette smoking causes hypertension, increased oxidative stress, impaired NO bioavailability, endothelial dysfunction, and cardiac remodeling in mice Adenosine metabolism and murine strain-specific IL-4-induced inflammation, emphysema, and fibrosis.Adipose stem cell treatment in mice attenuates lung and systemic injury induced by cigarette smoking.Cigarette smoke-induced lung emphysema in mice is associated with prolyl endopeptidase, an enzyme involved in collagen breakdown.Proteomic differences with and without ozone-exposure in a smoking-induced emphysema lung model A model of chronic inflammation and pulmonary emphysema after multiple ozone exposures in mice.Role of recently migrated monocytes in cigarette smoke-induced lung inflammation in different strain of mice Mmp1a and Mmp1b are not functional orthologs to human MMP1 in cigarette smoke induced lung disease The lung inflammation and skeletal muscle wasting induced by subchronic cigarette smoke exposure are not altered by a high-fat diet in mice.FOXO3 deficiency leads to increased susceptibility to cigarette smoke-induced inflammation, airspace enlargement, and chronic obstructive pulmonary disease The genetics of chronic obstructive pulmonary disease.Automated measurement of pulmonary emphysema and small airway remodeling in cigarette smoke-exposed mice.Respiratory mechanics do not always mirror pulmonary histological changes in emphysema.Telomere length is a determinant of emphysema susceptibility.Angiotensin receptor blockade attenuates cigarette smoke-induced lung injury and rescues lung architecture in mice.Transcriptional profiling and targeted proteomics reveals common molecular changes associated with cigarette smoke-induced lung emphysema development in five susceptible mouse strains.Alveolar fractal box dimension inversely correlates with mean linear intercept in mice with elastase-induced emphysema.Airway epithelial cell PPARγ modulates cigarette smoke-induced chemokine expression and emphysema susceptibility in mice.Use of a soluble epoxide hydrolase inhibitor in smoke-induced chronic obstructive pulmonary disease.Monitoring in vivo changes in lung microstructure with ³He MRI in Sendai virus-infected mice.SIRT1 protects against emphysema via FOXO3-mediated reduction of premature senescence in mice.Allergic rhinitis, asthma, airway biology, and chronic obstructive pulmonary disease in AJRCCM in 2004.Experimental progressive emphysema in BALB/cJ mice as a model for chronic alveolar destruction in humans.Sex-Related Differences in Pulmonary Function following 6 Months of Cigarette Exposure: Implications for Sexual Dimorphism in Mild COPD.

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P2860

The development of emphysema in cigarette smoke-exposed mice is strain dependent.

http://www.wikidata.org/entity/Q47949544

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

@zh-hant

name

The development of emphysema in cigarette smoke-exposed mice is strain dependent.

@en

The development of emphysema in cigarette smoke-exposed mice is strain dependent.

@nl

type

label

The development of emphysema in cigarette smoke-exposed mice is strain dependent.

@en

The development of emphysema in cigarette smoke-exposed mice is strain dependent.

@nl

prefLabel

The development of emphysema in cigarette smoke-exposed mice is strain dependent.

@en

The development of emphysema in cigarette smoke-exposed mice is strain dependent.

@nl

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RCCM.200309-1270OC

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American Journal of Respiratory and Critical Care Medicine

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The development of emphysema in cigarette smoke-exposed mice is strain dependent.

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Alexandra Triantafillopoulos

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Alexei Guerassimov

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Antony Turcotte

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Heberto Ghezzo

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John R Hoidal

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Kevin Whittaker

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Manuel G Cosio

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Midori Yamamoto

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Yasutaka Takubo

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Yuma Hoshino

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974-980

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scholarly article

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10.1164/RCCM.200309-1270OC

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9

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2004-07-28T00:00:00Z

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