CD14 is an essential mediator of LPS-induced airway disease. - Wikidata - wikimedia - TriplyDB

CD14 is an essential mediator of LPS-induced airway disease.

CD14 is an essential mediator of LPS-induced airway disease.

http://www.wikidata.org/entity/Q53574724

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Role of CD14 in a mouse model of acute lung inflammation induced by different lipopolysaccharide chemotypes.Modulation of SCF beta-TrCP-dependent I kappaB alpha ubiquitination by hydrogen peroxide.Human resistin promotes neutrophil proinflammatory activation and neutrophil extracellular trap formation and increases severity of acute lung injury Reducing LPS content in cockroach allergens increases pulmonary cytokine production without increasing inflammation: a randomized laboratory study.Exposure to hydrogen peroxide induces oxidation and activation of AMP-activated protein kinase.Inhibition of neutrophil apoptosis by PAI-1.Toll-like receptor 4 engagement inhibits adenosine 5'-monophosphate-activated protein kinase activation through a high mobility group box 1 protein-dependent mechanism Monocytes control second-phase neutrophil emigration in established lipopolysaccharide-induced murine lung injury.Vitronectin inhibits efferocytosis through interactions with apoptotic cells as well as with macrophages Endotoxin receptor CD14 in PiZ alpha-1-antitrypsin deficiency individuals.HMGB1 promotes neutrophil extracellular trap formation through interactions with Toll-like receptor 4 Racial differences in the association of CD14 polymorphisms with serum total IgE levels and allergen skin test reactivity Unfractionated heparin and new heparin analogues from ascidians (chordate-tunicate) ameliorate colitis in rats Signal transduction pathways linking the activation of alveolar macrophages with the recruitment of neutrophils to lungs in chronic obstructive pulmonary disease.Role of CD14 in lung inflammation and infection.siRNA targeting mCD14 inhibits TNF-α, MIP-2, and IL-6 secretion and NO production from LPS-induced RAW264.7 cells.CD14 gene silencing alters the microRNA expression profile of RAW264.7 cells stimulated by Brucella melitensis infection.A potential role for macrophages in maintaining lipopolysaccharide-induced subacute airway inflammation in rats.Nuclear factor-kappaB activation in airway epithelium induces inflammation and hyperresponsiveness.Prolyl hydroxylase 3 (PHD3) is essential for hypoxic regulation of neutrophilic inflammation in humans and mice Changes in Expression of the Membrane Receptors CD14, MHC-II, SR-A, and TLR4 in Tissue-Specific Monocytes/Macrophages Following Porphyromonas gingivalis-LPS Stimulation.

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P2860

CD14 is an essential mediator of LPS-induced airway disease.

http://www.wikidata.org/entity/Q53574724

description

2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

@zh-my

2007年学术文章

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2007年學術文章

@yue

2007年學術文章

@zh-hant

name

CD14 is an essential mediator of LPS-induced airway disease.

@en

CD14 is an essential mediator of LPS-induced airway disease.

@nl

type

label

CD14 is an essential mediator of LPS-induced airway disease.

@en

CD14 is an essential mediator of LPS-induced airway disease.

@nl

prefLabel

CD14 is an essential mediator of LPS-induced airway disease.

@en

CD14 is an essential mediator of LPS-induced airway disease.

@nl

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AJPLUNG.00282.2006

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American Journal of Physiology - Lung Cellular and Molecular Physiology

P1476

CD14 is an essential mediator of LPS-induced airway disease.

@en

P2093

David A Schwartz

http://www.w3.org/2001/XMLSchema#string

David M Brass

http://www.w3.org/2001/XMLSchema#string

Erin McElvania-Tekippe

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Imtaz Hossain

http://www.w3.org/2001/XMLSchema#string

John W Hollingsworth

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Stavros Garantziotis

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P2860

Resistance to endotoxin shock and reduced dissemination of gram-negative bacteria in CD14-deficient mice

CD14, a receptor for complexes of lipopolysaccharide (LPS) and LPS binding protein

Distinct roles of pattern recognition receptors CD14 and Toll-like receptor 4 in acute lung injury

Regulation of experimental lung inflammation.

Interleukin-8 gene expression by a pulmonary epithelial cell line. A model for cytokine networks in the lung.

Transgenic mice expressing human CD14 are hypersensitive to lipopolysaccharide.

Lipopolysaccharide activation of human endothelial and epithelial cells is mediated by lipopolysaccharide-binding protein and soluble CD14.

Lung inflammatory responses.

Cell-free pool of CD14 mediates activation of transcription factor NF-kappa B by lipopolysaccharide in human endothelial cells

Respiratory epithelial cells regulate lung inflammation in response to inhaled endotoxin.

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10.1152/AJPLUNG.00282.2006

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1

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17384086

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