about
Calpain-1 deletion impairs mGluR-dependent LTD and fear memory extinctionA novel form of synaptic plasticity in field CA3 of hippocampus requires GPER1 activation and BDNF releaseDistinct roles for μ-calpain and m-calpain in synaptic NMDAR-mediated neuroprotection and extrasynaptic NMDAR-mediated neurodegeneration.Estrogen Regulates Protein Synthesis and Actin Polymerization in Hippocampal Neurons through Different Molecular Mechanisms.Multiple cellular cascades participate in long-term potentiation and in hippocampus-dependent learning.Calpains: Master Regulators of Synaptic Plasticity.The non-coding RNA BC1 regulates experience-dependent structural plasticity and learning.A molecular brake controls the magnitude of long-term potentiation.Differential estrogenic effects of the persistent organochlorine pesticides dieldrin, endosulfan, and lindane in primary neuronal cultures.Reduction of glutamatergic neurotransmission by prolonged exposure to dieldrin involves NMDA receptor internalization and metabotropic glutamate receptor 5 downregulation.Allopregnanolone prevents dieldrin-induced NMDA receptor internalization and neurotoxicity by preserving GABA(A) receptor function.Homeostatic regulation of glutamate neurotransmission in primary neuronal cultures.Alpha2-Containing Glycine Receptors Promote Neonatal Spontaneous Activity of Striatal Medium Spiny Neurons and Support Maturation of Glutamatergic Inputs
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P50
description
researcher, ORCID id # 0000-0001-6936-0918
@en
wetenschapper
@nl
name
Victor Briz
@ast
Victor Briz
@en
Victor Briz
@es
Victor Briz
@nl
type
label
Victor Briz
@ast
Victor Briz
@en
Victor Briz
@es
Victor Briz
@nl
prefLabel
Victor Briz
@ast
Victor Briz
@en
Victor Briz
@es
Victor Briz
@nl
P108
P106
P108
P1153
35757431100
P21
P31
P496
0000-0001-6936-0918