Physiological consequences of drug resistance in Leishmania and their relevance for chemotherapy - sprookjes - yvandenbroek - TriplyDB

Physiological consequences of drug resistance in Leishmania and their relevance for chemotherapy

Physiological consequences of drug resistance in Leishmania and their relevance for chemotherapy

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Interventions for Old World cutaneous leishmaniasis Interventions for American cutaneous and mucocutaneous leishmaniasis Andrographolide nanoparticles in leishmaniasis: characterization and in vitro evaluations.Detection of drug resistance gene in cutaneous leishmaniasis by PCR in some endemic areas of iran.Phosphoproteomic analysis of wild-type and antimony-resistant Leishmania braziliensis lines by 2D-DIGE technology.Evaluation of Genetic Polymorphism of Leishmania (V.) braziliensis Isolates Obtained from the Same Patient before and after Therapeutic Failure or Reactivation of Cutaneous Lesions.Interventions for Old World cutaneous leishmaniasis.[Comparative study between oral miltefosine and parenteral N-metil glucamine antimoniate for the treatment of experimental leishmaniasis caused Leishmania (Leishmania) amazonensis].Drug resistance and treatment failure in leishmaniasis: A 21st century challenge.

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Physiological consequences of drug resistance in Leishmania and their relevance for chemotherapy

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2003 nî lūn-bûn

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2003 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2003 թվականի հոտեմբերին հրատարակված գիտական հոդված

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P2860

Trypanothione overproduction and resistance to antimonials and arsenicals in Leishmania

ABC transporters: from microorganisms to man

Drug resistance in leishmaniasis: its implication in systemic chemotherapy of cutaneous and mucocutaneous disease

Isolation of genes mediating resistance to inhibitors of nucleoside and ergosterol metabolism in Leishmania by overexpression/selection.

Multidrug resistance in Leishmania donovani is conferred by amplification of a gene homologous to the mammalian mdr1 gene

Recent advances in identifying and validating drug targets in trypanosomes and leishmanias.

Are ion-exchange processes central to understanding drug-resistance phenomena?

Mechanism of amphotericin B resistance in Leishmania donovani promastigotes.

Multiple recognition of various amphiphilic molecules by the multidrug resistance P-glycoprotein: molecular mechanisms and pharmacological consequences coming from functional interactions between various drugs.

Miltefosine, an oral agent, for the treatment of Indian visceral leishmaniasis.

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