Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion. - sprookjes - yvandenbroek - TriplyDB

Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion.

Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion.

http://www.wikidata.org/entity/Q30888193

about

Ca2+ Sparks and Ca2+ waves are the subcellular events underlying Ca2+ overload during ischemia and reperfusion in perfused intact hearts.Cardiac electrophysiological imaging systems scalable for high-throughput drug testing CaMKII-dependent phosphorylation of cardiac ryanodine receptors regulates cell death in cardiac ischemia/reperfusion injury.High-fat, low-carbohydrate diet promotes arrhythmic death and increases myocardial ischemia-reperfusion injury in rats.IKs protects from ventricular arrhythmia during cardiac ischemia and reperfusion in rabbits by preserving the repolarization reserve.A technical review of optical mapping of intracellular calcium within myocardial tissue Characterization of Cardiac Anoctamin1 Ca²⁺-Activated Chloride Channels and Functional Role in Ischemia-Induced Arrhythmias Sarcoplasmic phospholamban protein is involved in the mechanisms of postresuscitation myocardial dysfunction and the cardioprotective effect of nitrite during resuscitation.Calcium-calmodulin dependent protein kinase II (CaMKII): a main signal responsible for early reperfusion arrhythmias.Chasing cardiac physiology and pathology down the CaMKII cascade Intracellular Ca2+ release underlies the development of phase 2 in mouse ventricular action potentials.Ablation of junctin or triadin is associated with increased cardiac injury following ischaemia/reperfusion The continuing evolution of the Langendorff and ejecting murine heart: new advances in cardiac phenotyping.Mesencephalic astrocyte-derived neurotrophic factor protects the heart from ischemic damage and is selectively secreted upon sarco/endoplasmic reticulum calcium depletion Local Field Fluorescence Microscopy: Imaging Cellular Signals in Intact Hearts.Modification of STIM1 by O-linked N-acetylglucosamine (O-GlcNAc) attenuates store-operated calcium entry in neonatal cardiomyocytes New concepts of endoplasmic reticulum function in the heart: programmed to conserve.Confocal imaging of transmembrane voltage by SEER of di-8-ANEPPS Gene therapy for ventricular tachyarrhythmias.Calsequestrin 2 deletion shortens the refractoriness of Ca²⁺ release and reduces rate-dependent Ca²⁺-alternans in intact mouse hearts.STIM1/Orai1-mediated SOCE: current perspectives and potential roles in cardiac function and pathology The role of CaMKII regulation of phospholamban activity in heart disease.Optical mapping of sarcoplasmic reticulum Ca2+ in the intact heart: ryanodine receptor refractoriness during alternans and fibrillation.Cardioprotection in ischaemia-reperfusion injury: novel mechanisms and clinical translation.In Vivo Post-Cardiac Arrest Myocardial Dysfunction Is Supported by Ca2+/Calmodulin-Dependent Protein Kinase II-Mediated Calcium Long-Term Potentiation and Mitigated by Alda-1, an Agonist of Aldehyde Dehydrogenase Type 2 Reversible redox modifications of ryanodine receptor ameliorate ventricular arrhythmias in the ischemic-reperfused heart.N-n-butyl haloperidol iodide preserves cardiomyocyte calcium homeostasis during hypoxia/ischemia.β-Adrenergic Inhibition Prevents Action Potential and Calcium Handling Changes during Regional Myocardial Ischemia.Mitochondrial Bioenergetics During Ischemia and Reperfusion.Phospholamban ablation rescues the enhanced propensity to arrhythmias of mice with CaMKII-constitutive phosphorylation of RyR2 at site S2814.Energetic study of cardioplegic hearts under ischaemia/reperfusion and [Ca(2+)] changes in cardiomyocytes of guinea-pig: mitochondrial role

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P2860

Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion.

http://www.wikidata.org/entity/Q30888193

description

2009 nî lūn-bûn

@nan

2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年学术文章

@wuu

2009年学术文章

@zh-cn

2009年学术文章

@zh-hans

2009年学术文章

@zh-my

2009年学术文章

@zh-sg

2009年學術文章

@yue

name

Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion.

@ast

Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion.

@en

type

label

Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion.

@ast

Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion.

@en

prefLabel

Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion.

@ast

Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion.

@en

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Cardiovascular Research

P1476

Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion.

@en

P2093

Alicia Mattiazzi

http://www.w3.org/2001/XMLSchema#string

Ariel L Escobar

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Azadé D Petrosky

http://www.w3.org/2001/XMLSchema#string

Carlos A Valverde

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Dmytro Kornyeyev

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Marcela Ferreiro

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P2860

Functional adult myocardium in the absence of Na+-Ca2+ exchange: cardiac-specific knockout of NCX1

Mitofusin 2 tethers endoplasmic reticulum to mitochondria

Calcium measurements in perfused mouse heart: quantitating fluorescence and absorbance of Rhod-2 by application of photon migration theory.

Pulsed local-field fluorescence microscopy: a new approach for measuring cellular signals in the beating heart.

Physical coupling supports the local Ca2+ transfer between sarcoplasmic reticulum subdomains and the mitochondria in heart muscle.

Molecular and cellular mechanisms of myocardial stunning.

The Brugada syndrome: ionic basis and arrhythmia mechanisms.

Transient outward current prominent in canine ventricular epicardium but not endocardium.

Pathophysiology and pathogenesis of stunned myocardium. Depressed Ca2+ activation of contraction as a consequence of reperfusion-induced cellular calcium overload in ferret hearts.

Blebbistatin specifically inhibits actin-myosin interaction in mouse cardiac muscle.

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671-680

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scholarly article

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10.1093/CVR/CVP371

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4

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85

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2009-11-17T00:00:00Z

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38095944

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19920131

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2819836

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