Xanthine oxidase inhibitors improve energetics and function after infarction in failing mouse hearts.
about
Therapeutic effects of xanthine oxidase inhibitors: renaissance half a century after the discovery of allopurinolAntioxidants in Cardiovascular Therapy: Panacea or False Hope?Abnormal Ca(2+) cycling in failing ventricular myocytes: role of NOS1-mediated nitroso-redox balanceAre either or both hyperuricemia and xanthine oxidase directly toxic to the vasculature? A critical appraisalAllopurinol acutely increases adenosine triphospate energy delivery in failing human hearts.Xanthine oxidase inhibitors in heart failure: where do we go from here?Cardiac magnetic resonance imaging in small rodents using clinical 1.5 T and 3.0 T scanners.Role of xanthine oxidoreductase in cardiac nitroso-redox imbalanceThe emerging role of neuronal nitric oxide synthase in the regulation of myocardial function.Magnetic resonance imaging and spectroscopy of the murine cardiovascular systemXanthine oxidase inhibition with febuxostat attenuates systolic overload-induced left ventricular hypertrophy and dysfunction in miceRole of urate, xanthine oxidase and the effects of allopurinol in vascular oxidative stress.Oxidative stress and hyperuricaemia: pathophysiology, clinical relevance, and therapeutic implications in chronic heart failure.Redox signaling in cardiovascular health and disease.Xanthine oxidase inhibition for the treatment of cardiovascular disease: an updated systematic review and meta-analysis.Reoxygenation-derived toxic reactive oxygen/nitrogen species modulate the contribution of bone marrow progenitor cells to remodeling after myocardial infarction.Effect of acute xanthine oxidase inhibition on myocardial energetics during basal and very high cardiac workstatesAllopurinol, uric acid, and oxidative stress in cardiorenal disease.MRS: a noninvasive window into cardiac metabolism.The role of urate and xanthine oxidase in vascular oxidative stress: future directions.Biochemical and mechanical dysfunction in a mouse model of desmin-related myopathy.Delayed treatment effects of xanthine oxidase inhibition on systolic overload-induced left ventricular hypertrophy and dysfunctionInhibition of xanthine oxidase reduces hyperglycemia-induced oxidative stress and improves mitochondrial alterations in skeletal muscle of diabetic mice.Effect of allopurinol on cardiomyocyte apoptosis in rats after myocardial infarction.Inhibition of xanthine oxidase reduces wasting and improves outcome in a rat model of cancer cachexia.Allopurinol attenuates oxidative stress and cardiac fibrosis in angiotensin II-induced cardiac diastolic dysfunction.
P2860
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P2860
Xanthine oxidase inhibitors improve energetics and function after infarction in failing mouse hearts.
description
2005 nî lūn-bûn
@nan
2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Xanthine oxidase inhibitors im ...... ction in failing mouse hearts.
@ast
Xanthine oxidase inhibitors im ...... ction in failing mouse hearts.
@en
type
label
Xanthine oxidase inhibitors im ...... ction in failing mouse hearts.
@ast
Xanthine oxidase inhibitors im ...... ction in failing mouse hearts.
@en
prefLabel
Xanthine oxidase inhibitors im ...... ction in failing mouse hearts.
@ast
Xanthine oxidase inhibitors im ...... ction in failing mouse hearts.
@en
P2093
P2860
P1476
Xanthine oxidase inhibitors im ...... ction in failing mouse hearts.
@en
P2093
Anna V Naumova
Eduardo Marbán
Linda Stull
Robert G Weiss
Ronald Ouwerkerk
Vadappuram P Chacko
P2860
P304
P356
10.1152/AJPHEART.00831.2005
P577
2005-09-23T00:00:00Z