Deregulated Myc requires MondoA/Mlx for metabolic reprogramming and tumorigenesis - sprookjes - yvandenbroek - TriplyDB

Deregulated Myc requires MondoA/Mlx for metabolic reprogramming and tumorigenesis

Deregulated Myc requires MondoA/Mlx for metabolic reprogramming and tumorigenesis

http://www.wikidata.org/entity/Q35079601

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Metabolic vulnerabilities of MYC-induced cancer Interactions between Myc and MondoA transcription factors in metabolism and tumourigenesis MYC, Metabolism, and Cancer Oncogenes strike a balance between cellular growth and homeostasis MYC and metabolism on the path to cancer Latent KSHV Infected Endothelial Cells Are Glutamine Addicted and Require Glutaminolysis for Survival Menin enhances c-Myc-mediated transcription to promote cancer progression Globally Optimized Targeted Mass Spectrometry: Reliable Metabolomics Analysis with Broad Coverage Combining NMR and LC/MS Using Backward Variable Elimination: Metabolomics Analysis of Colorectal Cancer, Polyps, and Healthy Controls.Mechanisms and functions of Nrf2 signaling in Drosophila.Metabolomics method to comprehensively analyze amino acids in different domains Metabolic reprogramming in triple-negative breast cancer through Myc suppression of TXNIP MYC oncogene overexpression drives renal cell carcinoma in a mouse model through glutamine metabolism Targeted plasma metabolome response to variations in dietary glycemic load in a randomized, controlled, crossover feeding trial in healthy adults Strategies to Inhibit Myc and Their Clinical Applicability The glucose-sensing transcription factor MLX promotes myogenesis via myokine signaling Myc Depletion Induces a Pluripotent Dormant State Mimicking Diapause Phototransduction Influences Metabolic Flux and Nucleotide Metabolism in Mouse Retina Driver or passenger effects of augmented c-Myc and Cdc20 in gliomagenesis.Multi-omics analyses reveal metabolic alterations regulated by hepatitis B virus core protein in hepatocellular carcinoma cells.Deregulated KLF4 Expression in Myeloid Leukemias Alters Cell Proliferation and Differentiation through MicroRNA and Gene Targets.How does metabolism affect cell death in cancer?Integration of ChREBP-Mediated Glucose Sensing into Whole Body Metabolism.The MYC mRNA 3'-UTR couples RNA polymerase II function to glutamine and ribonucleotide levels.Pharmacometabonomic Prediction of Busulfan Clearance in Hematopoetic Cell Transplant Recipients.Lactate-An Integrative Mirror of Cancer Metabolism.MYC, Metabolic Synthetic Lethality, and Cancer.Emerging tale of UPR and cancer: an essentiality for malignancy.MNT and Emerging Concepts of MNT-MYC Antagonism Resetting cancer stem cell regulatory nodes upon MYC inhibition.The MYCN Protein in Health and Disease.Candidate serum metabolite biomarkers for differentiating gastroesophageal reflux disease, Barrett's esophagus, and high-grade dysplasia/esophageal adenocarcinoma.A novel role for the extended MYC network in cancer cell survival.MYC is a positive regulator of choline metabolism and impedes mitophagy-dependent necroptosis in diffuse large B-cell lymphoma.Genome-wide association study identifies novel type II diabetes risk loci in Jordan subpopulations.Allele-Specific Reprogramming of Cancer Metabolism by the Long Non-coding RNA CCAT2.Merkel cell polyomavirus recruits MYCL to the EP400 complex to promote oncogenesis.Oncogene KRAS activates fatty acid synthase, resulting in specific ERK and lipid signatures associated with lung adenocarcinoma.MYC and tumor metabolism: chicken and egg.Quantitative Method to Investigate the Balance between Metabolism and Proteome Biomass: Starting from Glycine.

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P2860

Deregulated Myc requires MondoA/Mlx for metabolic reprogramming and tumorigenesis

http://www.wikidata.org/entity/Q35079601

description

2015 nî lūn-bûn

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2015 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի հունվարին հրատարակված գիտական հոդված

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2015年の論文

@ja

2015年学术文章

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2015年学术文章

@zh-cn

2015年学术文章

@zh-hans

2015年学术文章

@zh-my

2015年学术文章

@zh-sg

2015年學術文章

@yue

name

Deregulated Myc requires MondoA/Mlx for metabolic reprogramming and tumorigenesis

@ast

Deregulated Myc requires MondoA/Mlx for metabolic reprogramming and tumorigenesis

@en

type

label

Deregulated Myc requires MondoA/Mlx for metabolic reprogramming and tumorigenesis

@ast

Deregulated Myc requires MondoA/Mlx for metabolic reprogramming and tumorigenesis

@en

prefLabel

Deregulated Myc requires MondoA/Mlx for metabolic reprogramming and tumorigenesis

@ast

Deregulated Myc requires MondoA/Mlx for metabolic reprogramming and tumorigenesis

@en

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J.CCELL.2014.11.024

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P1476

Deregulated Myc requires MondoA/Mlx for metabolic reprogramming and tumorigenesis

@en

P2093

Daniel Diolaiti

http://www.w3.org/2001/XMLSchema#string

Daniel Raftery

http://www.w3.org/2001/XMLSchema#string

Danijel Djukovic

http://www.w3.org/2001/XMLSchema#string

Haiwei Gu

http://www.w3.org/2001/XMLSchema#string

James B Hurley

http://www.w3.org/2001/XMLSchema#string

Jianhai Du

http://www.w3.org/2001/XMLSchema#string

Lisa McFerrin

http://www.w3.org/2001/XMLSchema#string

Michelle Ulrich

http://www.w3.org/2001/XMLSchema#string

Pei Feng Cheng

http://www.w3.org/2001/XMLSchema#string

Robert N Eisenman

http://www.w3.org/2001/XMLSchema#string

P2860

Mlx, a novel Max-like BHLHZip protein that interacts with the Max network of transcription factors

Mlx, a new Max-like bHLHZip family member: the center stage of a novel transcription factors regulatory pathway?

MondoA-Mlx heterodimers are candidate sensors of cellular energy status: mitochondrial localization and direct regulation of glycolysis

The glucose-responsive transcription factor ChREBP contributes to glucose-dependent anabolic synthesis and cell proliferation

Understanding the Warburg effect: the metabolic requirements of cell proliferation

MetaboAnalyst 2.0--a comprehensive server for metabolomic data analysis

Cluster analysis and display of genome-wide expression patterns

Coordination of nutrient availability and utilization by MAX- and MLX-centered transcription networks

Functional interactions among members of the MAX and MLX transcriptional network during oncogenesis

Lipogenesis and lipolysis: the pathways exploited by the cancer cells to acquire fatty acids

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271-285

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scholarly article

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10.1016/J.CCELL.2014.11.024

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2

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27

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Patrick Carroll

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2015-01-29T00:00:00Z

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271709085

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25640402

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4326605

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