Bifunctional major histocompatibility-linked genetic regulation of cell-mediated lympholysis to trinitrophenyl-modified autologous lymphocytes.
about
Lymphocytes. 1. Function. Genetic restrictions in the immune responseCytotoxic T cells distinguish between trinitrophenyl- and dinitrophenyl-modified syngeneic cells.H-2-restricted cytotoxic effectors generated in vitro by the addition of trinitrophenyl-conjugated soluble proteinsH-2-linked genetic control of murine T-cell-mediated lympholysis to autologous cells modified with low concentrations of trinitrobenzene sulfonateRegulation of T-cell-mediated lympholysis by the murine major histocompatibility complex. I. Preferential in vitro responses to trinitrophenyl-modified self K- and D-coded gene products in parental and F1 hybrid mouse strainsCytotoxic T-cell responses in mice infected with influenza and vaccinia viruses vary in magnitude with H-2 genotype.HLA restriction of human cytotoxic T lymphocytes specific for influenza virus. Poor recognition of virus associated with HLA A2.Absence of allogeneic restriction in human T-cell-mediated cytotoxicity to Epstein-Barr virus-infected target cells. Demonstration of an HLA-linked control at the effector levelRegulation of T-cell-mediated lympholysis by the murine major histocompatibility complex. II. Control of cytotoxic responses to trinitrophenyl-K and -D self products by H-2K- and H-2D-Region genes.Role of self carriers in the immune response and tolerance. V. Reversal of trinitrophenyl-modified self suppression of the B-cell response by blocking of H-2 antigens.Selective turnover and shedding of H-2K and H-2D antigens is controlled by the major histocompatibility complex. Implications for H-2-restricted recognition.Major histocompatibility complex restriction of soluble helper molecules in T cell responses to altered self.Trinitrophenyl modification of H-2k and H-2b spleen cells results in enhanced serological detection of Kk-like determinants.Immunization with SV40-transformed cells yields mainly MHC-restricted monoclonal antibodiesCell-mediated lympholysis of trinitrophenyl-modified autologous lymphocytes. Confirmation of genetic control of response to trinitrophenyl-modified H-2 antigens by the use of anti-H-2 and anti-Ia antibodiesCell-mediated lympholysis of N-(3-nitro-4-hydroxy-5-iodophenylacetyl)-beta-anaylglycylglycyl-modified autologous lymphocytes. Effector cell specificity to modified cell surface components controlled by the H-2K and H-2D serological regions of the muT-cell-deficient mice produce more antihapten antibodies against syngeneic than against allogeneic erythrocyte conjugatesMultiple H-2 linked immune response gene control of H-2 D-associated T-cell-mediated lympholysis to trinitrophenyl-modified autologous cells: Ir-like genes mapping to the left of I-A and within the I region.Virus specificity of cytotoxic T lymphocytes generated during acute lymphocytic choriomeningitis virus infection: role of the H-2 region in determining cross-reactivity for different lymphocytic choriomeningitis virus strains.Role of the H-2s haplotype in survival of mice after infection with Trypanosoma cruzi.Possible evolution of acquired immunity from self-recognition structures.Thymus and lymphohemopoietic cells: their role in T cell maturation in selection of T cells' H-2-restriction-specificity and in H-2 linked Ir gene control.Ir-genes in H-2 regulate generation of anti-viral cytotoxic T cells. Mapping to K or D and dominance of unresponsiveness.Rosette formation between murine lymphocytes and erythrocytes. A new locus in the H-2 regionAllorestricted cytotoxic T cells. Large numbers of allo-H-2Kb-restricted antihapten and antiviral cytotoxic T cell populations clonally develop in vitro from murine splenic precursor T cells.Comparison between the specificity of primary and secondary killer cells against alloantigens and hapten-modified syngeneic lymphoid cells.H–2 antigen restriction of viral infection (reply)
P2860
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P2860
Bifunctional major histocompatibility-linked genetic regulation of cell-mediated lympholysis to trinitrophenyl-modified autologous lymphocytes.
description
1975 nî lūn-bûn
@nan
1975年の論文
@ja
1975年学术文章
@wuu
1975年学术文章
@zh-cn
1975年学术文章
@zh-hans
1975年学术文章
@zh-my
1975年学术文章
@zh-sg
1975年學術文章
@yue
1975年學術文章
@zh
1975年學術文章
@zh-hant
name
Bifunctional major histocompat ...... dified autologous lymphocytes.
@ast
Bifunctional major histocompat ...... dified autologous lymphocytes.
@en
type
label
Bifunctional major histocompat ...... dified autologous lymphocytes.
@ast
Bifunctional major histocompat ...... dified autologous lymphocytes.
@en
prefLabel
Bifunctional major histocompat ...... dified autologous lymphocytes.
@ast
Bifunctional major histocompat ...... dified autologous lymphocytes.
@en
P2860
P356
P1476
Bifunctional major histocompat ...... dified autologous lymphocytes.
@en
P2093
Schmitt-Verhulst AM
Shearer GM
P2860
P304
P356
10.1084/JEM.142.4.914
P407
P577
1975-10-01T00:00:00Z