Cellular entry of human papillomavirus type 16 involves activation of the phosphatidylinositol 3-kinase/Akt/mTOR pathway and inhibition of autophagy. - sprookjes - yvandenbroek - TriplyDB

Cellular entry of human papillomavirus type 16 involves activation of the phosphatidylinositol 3-kinase/Akt/mTOR pathway and inhibition of autophagy.

Cellular entry of human papillomavirus type 16 involves activation of the phosphatidylinositol 3-kinase/Akt/mTOR pathway and inhibition of autophagy.

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The signaling involving in autophagy machinery in keratinocytes and therapeutic approaches for skin diseases Autophagy knocked down by high-risk HPV infection and uterine cervical carcinogenesis Concepts of papillomavirus entry into host cells Large scale RNAi reveals the requirement of nuclear envelope breakdown for nuclear import of human papillomaviruses Decoding protein networks during virus entry by quantitative proteomics BECN1-dependent CASP2 incomplete autophagy induction by binding to rabies virus phosphoprotein.Reverse-phase protein array profiling of oropharyngeal cancer and significance of PIK3CA mutations in HPV-associated head and neck cancer.NOD1 deficiency impairs CD44a/Lck as well as PI3K/Akt pathway.Reduced expression of autophagy markers correlates with high-risk human papillomavirus infection in human cervical squamous cell carcinoma Recent Insights into the Control of Human Papillomavirus (HPV) Genome Stability, Loss, and Degradation.Coxsackievirus A16 elicits incomplete autophagy involving the mTOR and ERK pathways.Restricted protein phosphatase 2A targeting by Merkel cell polyomavirus small T antigen.Interaction of human tumor viruses with host cell surface receptors and cell entry Kallikrein-8 Proteolytically Processes Human Papillomaviruses in the Extracellular Space To Facilitate Entry into Host Cells Role of autophagy in the ω-3 long chain polyunsaturated fatty acid-induced death of lung cancer A549 cells.The role of the PI3K/Akt/mTOR signalling pathway in human cancers induced by infection with human papillomaviruses.A Dual Role for the Nonreceptor Tyrosine Kinase Pyk2 during the Intracellular Trafficking of Human Papillomavirus 16.Suppression of Virulent Porcine Epidemic Diarrhea Virus Proliferation by the PI3K/Akt/GSK-3α/β Pathway.Autophagy regulates UBC9 levels during viral-mediated tumorigenesis.Expression of cyclin-dependent kinase inhibitor 2A 16, tumour protein 53 and epidermal growth factor receptor in salivary gland carcinomas is not associated with oncogenic virus infection HPV virions hitchhike a ride on retromer complexes.The evolving field of human papillomavirus receptor research: a review of binding and entry.Nip the HPV encoded evil in the cancer bud: HPV reshapes TRAILs and signaling landscapes Annexin A2 and S100A10 regulate human papillomavirus type 16 entry and intracellular trafficking in human keratinocytes.The papillomavirus major capsid protein L1.Antimicrobial autophagy: a conserved innate immune response in Drosophila.Multiple heparan sulfate binding site engagements are required for the infectious entry of human papillomavirus type 16.DEPTOR promotes survival of cervical squamous cell carcinoma cells and its silencing induces apoptosis through downregulating PI3K/AKT and by up-regulating p38 MAP kinase Identification of TRAPPC8 as a host factor required for human papillomavirus cell entry MARCH2 regulates autophagy by promoting CFTR ubiquitination and degradation and PIK3CA-AKT-MTOR signaling mTOR downstream effectors, 4EBP1 and eIF4E, are overexpressed and associated with HPV status in precancerous lesions and carcinomas of the uterine cervix.The tetraspanin CD151 in papillomavirus infection.The Fusion Protein of CTP-HBcAg18-27-Tapasin Mediates the Apoptosis of CD8(+)T Cells and CD8(+) T Cell Response in HLA-A2 Transgenic Mice.HPV disease transmission protection and control.Nutrient sensing and metabolic stress pathways in innate immunity.The role and therapeutic potential of autophagy modulation in controlling virus-induced cell death.Make yourself at home: viral hijacking of the PI3K/Akt signaling pathway.Role of autophagy in head and neck cancer and therapeutic resistance.Viruses exploit the function of epidermal growth factor receptor.Dynamics of virus-receptor interactions in virus binding, signaling, and endocytosis

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P2860

Cellular entry of human papillomavirus type 16 involves activation of the phosphatidylinositol 3-kinase/Akt/mTOR pathway and inhibition of autophagy.

http://www.wikidata.org/entity/Q36607050

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2012 nî lūn-bûn

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Cellular entry of human papill ...... y and inhibition of autophagy.

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Cellular entry of human papill ...... y and inhibition of autophagy.

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type

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Cellular entry of human papill ...... y and inhibition of autophagy.

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Cellular entry of human papill ...... y and inhibition of autophagy.

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Cellular entry of human papill ...... y and inhibition of autophagy.

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Cellular entry of human papill ...... y and inhibition of autophagy.

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Cellular entry of human papill ...... ay and inhibition of autophagy

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Rosa T Sterk

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Sergio A DeHaro

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Zurab Surviladze

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P2860

Two highly conserved cysteine residues in HPV16 L2 form an intramolecular disulfide bond and are critical for infectivity in human keratinocytes

Phosphorylation of the PTEN tail regulates protein stability and function

Phosphorylation and regulation of Akt/PKB by the rictor-mTOR complex

Mammalian TOR complex 2 controls the actin cytoskeleton and is rapamycin insensitive

Human papillomavirus infection is inhibited by host autophagy in primary human keratinocytes

SREBP activity is regulated by mTORC1 and contributes to Akt-dependent cell growth

Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2A

LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing

Regulation of 4E-BP1 phosphorylation: a novel two-step mechanism

The mTOR signalling pathway in human cancer

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2508-2517

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10.1128/JVI.02319-12

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87

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Michelle A Ozbun

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2012-12-19T00:00:00Z

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23255786

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