Airway smooth muscle growth in asthma: proliferation, hypertrophy, and migration. - sprookjes - yvandenbroek - TriplyDB

Airway smooth muscle growth in asthma: proliferation, hypertrophy, and migration.

Airway smooth muscle growth in asthma: proliferation, hypertrophy, and migration.

http://www.wikidata.org/entity/Q37038075

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The metalloprotease-disintegrin ADAM8 is essential for the development of experimental asthma Regulator of G-protein signaling-5 inhibits bronchial smooth muscle contraction in severe asthma.Metabolomic biomarkers in a model of asthma exacerbation: urine nuclear magnetic resonance.Alterations of the lung methylome in allergic airway hyper-responsiveness.Beta-agonist-associated reduction in RGS5 expression promotes airway smooth muscle hyper-responsiveness.PPARγ agonist rosiglitazone prevents perinatal nicotine exposure-induced asthma in rat offspring Bronchial smooth muscle cells of asthmatics promote angiogenesis through elevated secretion of CXC-chemokines (ENA-78, GRO-α, and IL-8).The role of inflammation resolution speed in airway smooth muscle mass accumulation in asthma: insight from a theoretical model.IgE regulates the expression of smMLCK in human airway smooth muscle cells.Mechanical stimuli and IL-13 interact at integrin adhesion complexes to regulate expression of smooth muscle myosin heavy chain in airway smooth muscle tissue.Expression profiling identifies Klf15 as a glucocorticoid target that regulates airway hyperresponsiveness.Airway remodeling in murine asthma correlates with a defect in PGE2 synthesis by lung fibroblasts.A Critical Role of the mTOR/eIF2α Pathway in Hypoxia-Induced Pulmonary Hypertension.β2-adrenergic receptor agonists modulate human airway smooth muscle cell migration via vasodilator-stimulated phosphoprotein Cox4i2, Ifit2, and Prdm11 Mutant Mice: Effective Selection of Genes Predisposing to an Altered Airway Inflammatory Response from a Large Compendium of Mutant Mouse Lines.cAMP-mediated secretion of brain-derived neurotrophic factor in developing airway smooth muscle.Eosinophils enhance WNT-5a and TGF-β1 genes expression in airway smooth muscle cells and promote their proliferation by increased extracellular matrix proteins production in asthma.Airway smooth muscle STIM1 and Orai1 are upregulated in asthmatic mice and mediate PDGF-activated SOCE, CRAC currents, proliferation, and migration Sex-specific perinatal nicotine-induced asthma in rat offspring.Synergistic interaction between PPAR ligands and salbutamol on human bronchial smooth muscle cell proliferation Competitive metabolism of L-arginine: arginase as a therapeutic target in asthma.Aberrant DNA Methylation of Phosphodiesterase [corrected] 4D Alters Airway Smooth Muscle Cell Phenotypes.Integrin-linked kinase regulates smooth muscle differentiation marker gene expression in airway tissue.Peripheral Airway Smooth Muscle, but Not the Trachealis, Is Hypercontractile in an Equine Model of Asthma.Epigenetic alterations by DNA methylation in house dust mite-induced airway hyperresponsiveness.Airway smooth muscle in airway reactivity and remodeling: what have we learned?Link between vitamin D and airway remodeling.What's new in asthma pathophysiology and immunopathology?The pivotal role of airway smooth muscle in asthma pathophysiology Airway remodelling and inflammation in asthma are dependent on the extracellular matrix protein fibulin-1c.Interaction between allergic asthma and atherosclerosis.Emerging concepts in smooth muscle contributions to airway structure and function: implications for health and disease.An Official American Thoracic Society Research Statement: Current Challenges Facing Research and Therapeutic Advances in Airway Remodeling.Recombinant rat CC10 protein inhibits PDGF-induced airway smooth muscle cells proliferation and migration.Azithromycin reduces the viability of human bronchial smooth muscle cells.Human Rhinovirus Infection of Epithelial Cells Modulates Airway Smooth Muscle Migration.Thymic stromal lymphopoietin induces migration in human airway smooth muscle cells Vitamin D - a new treatment for airway remodelling in asthma?Airway epithelium stimulates smooth muscle proliferation.MicroRNA Regulation of Smooth Muscle Phenotype.

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Airway smooth muscle growth in asthma: proliferation, hypertrophy, and migration.

http://www.wikidata.org/entity/Q37038075

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on January 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Airway smooth muscle growth in asthma: proliferation, hypertrophy, and migration.

@en

Airway smooth muscle growth in asthma: proliferation, hypertrophy, and migration.

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type

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Airway smooth muscle growth in asthma: proliferation, hypertrophy, and migration.

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Airway smooth muscle growth in asthma: proliferation, hypertrophy, and migration.

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prefLabel

Airway smooth muscle growth in asthma: proliferation, hypertrophy, and migration.

@en

Airway smooth muscle growth in asthma: proliferation, hypertrophy, and migration.

@nl

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PATS.200705-063VS

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Proceedings of the American Thoracic Society

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Airway smooth muscle growth in asthma: proliferation, hypertrophy, and migration.

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J Kelley Bentley

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Marc B Hershenson

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P2860

Mammalian TOR complex 2 controls the actin cytoskeleton and is rapamycin insensitive

Tuberous sclerosis complex-1 and -2 gene products function together to inhibit mammalian target of rapamycin (mTOR)-mediated downstream signaling

TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling

Rheb is a direct target of the tuberous sclerosis tumour suppressor proteins

Regulation of eukaryotic initiation factor eIF2B: glycogen synthase kinase-3 phosphorylates a conserved serine which undergoes dephosphorylation in response to insulin

Rapamycin blocks the phosphorylation of 4E-BP1 and inhibits cap-dependent initiation of translation

Phosphorylation of the translational repressor PHAS-I by the mammalian target of rapamycin

Human bronchial fibroblasts exhibit a mesenchymal stem cell phenotype and multilineage differentiating potentialities.

Cell cycle versus density dependence of smooth muscle alpha actin expression in cultured rat aortic smooth muscle cells.

Hyperplasia of smooth muscle in mild to moderate asthma without changes in cell size or gene expression.

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10.1513/PATS.200705-063VS

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